Interferon-gamma prevents death of bystander neurons during CD8 T cell responses in the brain. - Wikidata - awgldk - TriplyDB

Interferon-gamma prevents death of bystander neurons during CD8 T cell responses in the brain.

Interferon-gamma prevents death of bystander neurons during CD8 T cell responses in the brain.

http://www.wikidata.org/entity/Q37164219

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The pathogenesis of bornaviral diseases in mammals.Neuroprotection by IFN-γ via astrocyte-secreted IL-6 in acute neuroinflammation.T cell-, interleukin-12-, and gamma interferon-driven viral clearance in measles virus-infected brain tissue Granzyme B-inhibitor serpina3n induces neuroprotection in vitro and in vivo.Interferon gamma protects neonatal neural stem/progenitor cells during measles virus infection of the brain Acute in vivo exposure to interferon-gamma enables resident brain dendritic cells to become effective antigen presenting cells.Gliovascular and cytokine interactions modulate brain endothelial barrier in vitro.Absence of IFNγ expression induces neuronal degeneration in the spinal cord of adult mice.

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Interferon-gamma prevents death of bystander neurons during CD8 T cell responses in the brain.

http://www.wikidata.org/entity/Q37164219

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 09 April 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Interferon-gamma prevents deat ...... T cell responses in the brain.

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Interferon-gamma prevents deat ...... T cell responses in the brain.

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type

label

Interferon-gamma prevents deat ...... T cell responses in the brain.

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Interferon-gamma prevents deat ...... T cell responses in the brain.

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prefLabel

Interferon-gamma prevents deat ...... T cell responses in the brain.

@en

Interferon-gamma prevents deat ...... T cell responses in the brain.

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AJPATH.2009.080897

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The American Journal of Pathology

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Interferon-gamma prevents deat ...... T cell responses in the brain.

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Jürgen Hausmann

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Kirsten Richter

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Peter Staeheli

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CD8(+) T lymphocytes mediate Borna disease virus-induced immunopathology independently of perforin

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Selective dysfunction of hippocampal CA1 astrocytes contributes to delayed neuronal damage after transient forebrain ischemia.

Targeted deletion of a high-affinity GATA-binding site in the GATA-1 promoter leads to selective loss of the eosinophil lineage in vivo

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1799-1807

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10.2353/AJPATH.2009.080897

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