Internalization of eNOS and NO delivery to subcellular targets determine agonist-induced hyperpermeability. - Wikidata - awgldk - TriplyDB

Internalization of eNOS and NO delivery to subcellular targets determine agonist-induced hyperpermeability.

Internalization of eNOS and NO delivery to subcellular targets determine agonist-induced hyperpermeability.

http://www.wikidata.org/entity/Q37167720

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Rnd3 as a Novel Target to Ameliorate Microvascular Leakage Dynamin activates NO production in rat renal inner medullary collecting ducts via protein-protein interaction with NOS1 Vascular permeability to plasma, plasma proteins, and cells: an update.The NO cascade, eNOS location, and microvascular permeability.Rapamycin inhibits VEGF-induced microvascular hyperpermeability in vivo.Nitroglycerin tolerance in caveolin-1 deficient mice.Novel peptide for attenuation of hyperoxia-induced disruption of lung endothelial barrier and pulmonary edema via modulating peroxynitrite formation Functional significance of cytosolic endothelial nitric-oxide synthase (eNOS): regulation of hyperpermeability Disruption of caveolae blocks ischemic preconditioning-mediated S-nitrosylation of mitochondrial proteins.Ischaemic preconditioning preferentially increases protein S-nitrosylation in subsarcolemmal mitochondria Role of reactive oxygen and nitrogen species in the vascular responses to inflammation.Pre-conditioned mesenchymal stem cells ameliorate renal ischemic injury in rats by augmented survival and engraftment.S-nitrosylation regulates VE-cadherin phosphorylation and internalization in microvascular permeability.New Member of Endothelial Arsenal Against Inflammation.S-Nitrosation of β-catenin and p120 catenin: a novel regulatory mechanism in endothelial hyperpermeability.Nitric oxide, S-nitrosation, and endothelial permeability.S-nitrosation of proteins: An emergent regulatory mechanism in microvascular permeability and vascular function.Endothelial cAMP deactivates ischemia-reperfusion-induced microvascular hyperpermeability via Rap1-mediated mechanisms.Angiotensin II impairs endothelial nitric-oxide synthase bioavailability under free cholesterol-enriched conditions via intracellular free cholesterol-rich membrane microdomains.Nuclear localization of platelet-activating factor receptor controls retinal neovascularization.Remodelling of membrane rafts expression in lung cells as an early sign of mechanotransduction-signalling in pulmonary edema.The Janus-faced regulation of endothelial permeability by cyclic GMP.S-nitrosylation of VASP at cysteine 64 mediates the inflammation-stimulated increase in microvascular permeability.

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P2860

Internalization of eNOS and NO delivery to subcellular targets determine agonist-induced hyperpermeability.

http://www.wikidata.org/entity/Q37167720

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 02 April 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Internalization of eNOS and NO ...... ist-induced hyperpermeability.

@en

Internalization of eNOS and NO ...... ist-induced hyperpermeability.

@nl

type

label

Internalization of eNOS and NO ...... ist-induced hyperpermeability.

@en

Internalization of eNOS and NO ...... ist-induced hyperpermeability.

@nl

prefLabel

Internalization of eNOS and NO ...... ist-induced hyperpermeability.

@en

Internalization of eNOS and NO ...... ist-induced hyperpermeability.

@nl

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PNAS.0812694106

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

Internalization of eNOS and NO ...... ist-induced hyperpermeability.

@en

P2093

Brajesh K Lal

http://www.w3.org/2001/XMLSchema#string

Cynthia J Meininger

http://www.w3.org/2001/XMLSchema#string

David D Kim

http://www.w3.org/2001/XMLSchema#string

Donna M Gordon

http://www.w3.org/2001/XMLSchema#string

Fabiola A Sánchez

http://www.w3.org/2001/XMLSchema#string

Roshniben Rana

http://www.w3.org/2001/XMLSchema#string

Ruifang Zheng

http://www.w3.org/2001/XMLSchema#string

Toru Iwahashi

http://www.w3.org/2001/XMLSchema#string

Walter N Durán

http://www.w3.org/2001/XMLSchema#string

P2860

Regulation of endothelium-derived nitric oxide production by the protein kinase Akt

VE-cadherin and beta-catenin binding dynamics during histamine-induced endothelial hyperpermeability

Regulation of beta-adrenergic receptor signaling by S-nitrosylation of G-protein-coupled receptor kinase 2

Targeting of nitric oxide synthase to endothelial cell caveolae via palmitoylation: implications for nitric oxide signaling

Dissecting the interaction between nitric oxide synthase (NOS) and caveolin. Functional significance of the nos caveolin binding domain in vivo

Phospho-caveolin-1 mediates integrin-regulated membrane domain internalization.

Direct interaction between endothelial nitric-oxide synthase and dynamin-2. Implications for nitric-oxide synthase function.

Signal transduction pathways in enhanced microvascular permeability.

Predominant role of endothelial nitric oxide synthase in vascular endothelial growth factor-induced angiogenesis and vascular permeability.

Anaphylactic shock depends on PI3K and eNOS-derived NO.

P304

6849-6853

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scholarly article

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10.1073/PNAS.0812694106

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P407

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16

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106

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2009-04-02T00:00:00Z

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24254048

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19342481

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2672508

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