Leupeptin-based inhibitors do not improve the mdx phenotype.
about
Rescue of dystrophic skeletal muscle by PGC-1α involves a fast to slow fiber type shift in the mdx mouseLong-Term Quercetin Dietary Enrichment Partially Protects Dystrophic Skeletal MusclePharmacologic management of Duchenne muscular dystrophy: target identification and preclinical trialsA proteasome inhibitor fails to attenuate dystrophic pathology in mdx mice.The role of proteases in excitation-contraction coupling failure in muscular dystrophy.Regulation of the calpain and ubiquitin-proteasome systems in a canine model of muscular dystrophy.Chronic administration of a leupeptin-derived calpain inhibitor fails to ameliorate severe muscle pathology in a canine model of duchenne muscular dystrophyPorcine models of muscular dystrophyAbsence of Dystrophin Disrupts Skeletal Muscle Signaling: Roles of Ca2+, Reactive Oxygen Species, and Nitric Oxide in the Development of Muscular Dystrophy.Long-term wheel running compromises diaphragm function but improves cardiac and plantarflexor function in the mdx mouseCanine models of Duchenne muscular dystrophy and their use in therapeutic strategies.Calpain-mediated proteolysis of tropomodulin isoforms leads to thin filament elongation in dystrophic skeletal muscle.Recessive and dominant mutations in COL12A1 cause a novel EDS/myopathy overlap syndrome in humans and mice.The dystrophin-glycoprotein complex in the prevention of muscle damage.The physiological response of protease inhibition in dystrophic muscle.Calpain research for drug discovery: challenges and potential.Oral quercetin administration transiently protects respiratory function in dystrophin-deficient mice.PGC-1α gene transfer improves muscle function in dystrophic muscle following prolonged disease progress.Increased catalase expression improves muscle function in mdx miceImmunoproteasome in animal models of Duchenne muscular dystrophy.Proteomic assessment of the acute phase of dystrophin deficiency in mdx mice.Rescue of dystrophic skeletal muscle by PGC-1α involves restored expression of dystrophin-associated protein complex components and satellite cell signaling.Ventilator-induced diaphragmatic dysfunction in mdx mice.
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P2860
Leupeptin-based inhibitors do not improve the mdx phenotype.
description
article científic
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article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on 15 September 2010
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Leupeptin-based inhibitors do not improve the mdx phenotype.
@en
Leupeptin-based inhibitors do not improve the mdx phenotype.
@nl
type
label
Leupeptin-based inhibitors do not improve the mdx phenotype.
@en
Leupeptin-based inhibitors do not improve the mdx phenotype.
@nl
prefLabel
Leupeptin-based inhibitors do not improve the mdx phenotype.
@en
Leupeptin-based inhibitors do not improve the mdx phenotype.
@nl
P2093
P2860
P1476
Leupeptin-based inhibitors do not improve the mdx phenotype
@en
P2093
Elisabeth Barton
H Lee Sweeney
Jennifer Pham
Klara Pendrak
Monica Zadel
Pedro Acosta
Ted Carver
Zuozhen Tian
P2860
P304
P356
10.1152/AJPREGU.00586.2009
P577
2010-09-15T00:00:00Z