Immobilization rapidly induces muscle insulin resistance together with the activation of MAPKs (JNK and p38) and impairment of AS160 phosphorylation

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Inhibition of the JNK/MAPK signaling pathway by myogenesis-associated miRNAs is required for skeletal muscle development.

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Immobilization rapidly induces muscle insulin resistance together with the activation of MAPKs (JNK and p38) and impairment of AS160 phosphorylation

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http://www.wikidata.org/entity/Q37176715

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on August 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Immobilization rapidly induces ...... rment of AS160 phosphorylation

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Immobilization rapidly induces ...... r with the activation of MAPKs

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Immobilization rapidly induces ...... rment of AS160 phosphorylation

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Immobilization rapidly induces ...... r with the activation of MAPKs

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Immobilization rapidly induces ...... rment of AS160 phosphorylation

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Immobilization rapidly induces ...... r with the activation of MAPKs

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Immobilization rapidly induces ...... rment of AS160 phosphorylation

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Emi Kawamoto

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Hiroyuki Masuda

http://www.w3.org/2001/XMLSchema#string

Keiichi Koshinaka

http://www.w3.org/2001/XMLSchema#string

Kentaro Kawanaka

http://www.w3.org/2001/XMLSchema#string

Tatsuhiko Yoshimura

http://www.w3.org/2001/XMLSchema#string

P2860

Coupling of stress in the ER to activation of JNK protein kinases by transmembrane protein kinase IRE1

Reversal of obesity- and diet-induced insulin resistance with salicylates or targeted disruption of Ikkbeta

Prevention of fat-induced insulin resistance by salicylate

Contraction regulates site-specific phosphorylation of TBC1D1 in skeletal muscle

Tumor necrosis factor alpha produces insulin resistance in skeletal muscle by activation of inhibitor kappaB kinase in a p38 MAPK-dependent manner

Genetic disruption of AMPK signaling abolishes both contraction- and insulin-stimulated TBC1D1 phosphorylation and 14-3-3 binding in mouse skeletal muscle

Tyrosine kinase activity is essential for the association of phospholipase C-gamma with the epidermal growth factor receptor

Inactivation of acetyl-CoA carboxylase and activation of AMP-activated protein kinase in muscle during exercise

A central role for JNK in obesity and insulin resistance

p38 mitogen-activated protein kinase: a critical node linking insulin resistance and cardiovascular diseases in type 2 diabetes mellitus.

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scholarly article

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10.14814/PHY2.12876

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2016-08-01T00:00:00Z

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27482072

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phosphorylation

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4985544

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Immobilization rapidly induces muscle insulin resistance together with the activation of MAPKs (JNK and p38) and impairment of AS160 phosphorylation

about

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P2860

Immobilization rapidly induces muscle insulin resistance together with the activation of MAPKs (JNK and p38) and impairment of AS160 phosphorylation

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