Tumor necrosis factor alpha produces insulin resistance in skeletal muscle by activation of inhibitor kappaB kinase in a p38 MAPK-dependent manner
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Regulation of PKD by the MAPK p38delta in insulin secretion and glucose homeostasisMicroRNAs as regulators of metabolic disease: pathophysiologic significance and emerging role as biomarkers and therapeuticsNitric oxide, oxidative stress, and p66Shc interplay in diabetic endothelial dysfunctionInconsistencies and controversies surrounding the amyloid hypothesis of Alzheimer's diseaseCellular and molecular mechanisms of muscle atrophyA Potential Role for Pro-Inflammatory Cytokines in the Development of Insulin Resistance in HorsesDirect involvement of tumor necrosis factor-α in the regulation of glucose uptake in rainbow trout muscle cellsCaffeine modulates phosphorylation of insulin receptor substrate-1 and impairs insulin signal transduction in rat skeletal musclePalmitate-activated macrophages confer insulin resistance to muscle cells by a mechanism involving protein kinase C θ and εLeucine signaling in the pathogenesis of type 2 diabetes and obesitymicroRNA in the development of diabetic complications.Insulin resistance in non-obese subjects is associated with activation of the JNK pathway and impaired insulin signaling in skeletal muscle.Deficiency of oncostatin M receptor β (OSMRβ) exacerbates high-fat diet-induced obesity and related metabolic disorders in mice.Exercise and Glycemic Control: Focus on Redox Homeostasis and Redox-Sensitive Protein Signaling.Environmental tobacco use and indicators of metabolic syndrome in Chinese adults.Insulin resistance, lipotoxicity, type 2 diabetes and atherosclerosis: the missing links. The Claude Bernard Lecture 2009Endothelial dysfunction in diabetes mellitus: molecular mechanisms and clinical implications.Lipid-induced insulin resistance is prevented in lean and obese myotubes by AICAR treatment.Palmitoleic acid prevents palmitic acid-induced macrophage activation and consequent p38 MAPK-mediated skeletal muscle insulin resistance.Intracellular molecular effects of insulin resistance in patients with metabolic syndrome.Disassociation of muscle insulin signaling and insulin-stimulated glucose uptake during endotoxemiaSkeletal muscle changes after hemiparetic stroke and potential beneficial effects of exercise intervention strategies.Protein phosphatase 4 interacts with and down-regulates insulin receptor substrate 4 following tumor necrosis factor-alpha stimulation.Task-oriented treadmill exercise training in chronic hemiparetic stroke.Biomedical consequences of alcohol use disorders in the HIV-infected hostAPPL1 mediates adiponectin-stimulated p38 MAPK activation by scaffolding the TAK1-MKK3-p38 MAPK pathway.p38 mitogen-activated protein kinase-dependent transactivation of ErbB receptor family: a novel common mechanism for stress-induced IRS-1 serine phosphorylation and insulin resistance.Branched motifs enable long-range interactions in signaling networks through retrograde propagationIdentification of nuclear hormone receptor pathways causing insulin resistance by transcriptional and epigenomic analysis.Curcumin prevents lipopolysaccharide-induced atrogin-1/MAFbx upregulation and muscle mass loss.Elevated muscle TLR4 expression and metabolic endotoxemia in human aging.MicroRNA-494, upregulated by tumor necrosis factor-α, desensitizes insulin effect in C2C12 muscle cellsDietary exercise as a novel strategy for the prevention and treatment of metabolic syndrome: effects on skeletal muscle function.Disrupted anabolic and catabolic processes may contribute to alcohol-accentuated SAIDS-associated wastingLiving in a box or call of the wild? Revisiting lifetime inactivity and sarcopeniaRelationships between serum adiponectin and soluble TNF-α receptors and glucose and lipid oxidation in lean and obese subjects.Wnt7a-Fzd7 signalling directly activates the Akt/mTOR anabolic growth pathway in skeletal muscle.G protein-coupled receptor kinase 2 plays a relevant role in insulin resistance and obesityImproved insulin sensitivity with calorie restriction does not require reduced JNK1/2, p38, or ERK1/2 phosphorylation in skeletal muscle of 9-month-old rats.The brown adipose cell: a model for understanding the molecular mechanisms of insulin resistance.
P2860
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P2860
Tumor necrosis factor alpha produces insulin resistance in skeletal muscle by activation of inhibitor kappaB kinase in a p38 MAPK-dependent manner
description
2004 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
article publié dans la revue scientifique Journal of Biological Chemistry
@fr
artículu científicu espublizáu en 2004
@ast
im April 2004 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2004/04/23)
@sk
vědecký článek publikovaný v roce 2004
@cs
wetenschappelijk artikel (gepubliceerd op 2004/04/23)
@nl
наукова стаття, опублікована у квітні 2004
@uk
name
Tumor necrosis factor alpha pr ...... in a p38 MAPK-dependent manner
@ast
Tumor necrosis factor alpha pr ...... in a p38 MAPK-dependent manner
@en
Tumor necrosis factor alpha pr ...... in a p38 MAPK-dependent manner
@nl
type
label
Tumor necrosis factor alpha pr ...... in a p38 MAPK-dependent manner
@ast
Tumor necrosis factor alpha pr ...... in a p38 MAPK-dependent manner
@en
Tumor necrosis factor alpha pr ...... in a p38 MAPK-dependent manner
@nl
prefLabel
Tumor necrosis factor alpha pr ...... in a p38 MAPK-dependent manner
@ast
Tumor necrosis factor alpha pr ...... in a p38 MAPK-dependent manner
@en
Tumor necrosis factor alpha pr ...... in a p38 MAPK-dependent manner
@nl
P2093
P3181
P356
P1476
Tumor necrosis factor alpha pr ...... in a p38 MAPK-dependent manner
@en
P2093
Cristina de Alvaro
Margarita Lorenzo
Rosario Hernandez
Teresa Teruel
P304
17070–17078
P3181
P356
10.1074/JBC.M312021200
P407
P577
2004-04-23T00:00:00Z