Glial-cytokine-neuronal interactions underlying the mechanisms of persistent pain
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Differential involvement of trigeminal transition zone and laminated subnucleus caudalis in orofacial deep and cutaneous hyperalgesia: the effects of interleukin-10 and glial inhibitorsFrequency-dependent effects of vibration on peripheral nerves and sensory nerve function in a rat model of hand-arm vibration syndrome. (Erratum: Characterization of frequency-dependent responses of the vascular systemThe effects of repetitive vibration on sensorineural function: biomarkers of sensorineural injury in an animal model of metabolic syndromeCalcitonin gene-related peptide (CGRP) receptor antagonists in the treatment of migraineThe "toll" of opioid-induced glial activation: improving the clinical efficacy of opioids by targeting gliaCalcitonin gene-related peptide stimulation of nitric oxide synthesis and release from trigeminal ganglion glial cellsInteractions of Opioids and HIV Infection in the Pathogenesis of Chronic PainBiological Roles of Resolvins and Related Substances in the Resolution of PainSpinal interleukin-10 therapy to treat peripheral neuropathic painMechanisms of acupuncture-electroacupuncture on persistent painMicroglia and spinal cord synaptic plasticity in persistent painEmerging targets in neuroinflammation-driven chronic painEmerging roles of resolvins in the resolution of inflammation and painTramadol and propentofylline coadministration exerted synergistic effects on rat spinal nerve ligation-induced neuropathic painInterleukin-1β increased the expression of protease-activated receptor 4 mRNA and protein in dorsal root ganglion neuronsThe Role of Glia in the Peripheral and Central Auditory System Following Noise Overexposure: Contribution of TNF-α and IL-1β to the Pathogenesis of Hearing LossActivation of the neuronal extracellular signal-regulated kinase 2 in the spinal cord dorsal horn is required for complete Freund's adjuvant-induced pain hypersensitivityDifferences in neural-immune gene expression response in rat spinal dorsal horn correlates with variations in electroacupuncture analgesiaPersistent changes in spinal cord gene expression after recovery from inflammatory hyperalgesia: a preliminary study on pain memory.Crosstalk between spinal astrocytes and neurons in nerve injury-induced neuropathic pain.Suppression of spinal connexin 43 expression attenuates mechanical hypersensitivity in rats after an L5 spinal nerve injury.Astroglia in medullary dorsal horn (trigeminal spinal subnucleus caudalis) are involved in trigeminal neuropathic pain mechanisms.The c-Jun N-terminal kinase 1 (JNK1) in spinal astrocytes is required for the maintenance of bilateral mechanical allodynia under a persistent inflammatory pain conditionN-Methyl-D-aspartate receptor (NMDAR) independent maintenance of inflammatory pain.Microinjection of IL-1β into the trigeminal transition zone produces bilateral NMDA receptor-dependent orofacial hyperalgesia involving descending circuitry.Interleukin-1 receptor type 1 is overexpressed in neurons but not in glial cells within the rat superficial spinal dorsal horn in complete Freund adjuvant-induced inflammatory pain.Epigenetic modification of DRG neuronal gene expression subsequent to nerve injury: etiological contribution to complex regional pain syndromes (Part I).Increased neuronal expression of neurokinin-1 receptor and stimulus-evoked internalization of the receptor in the rostral ventromedial medulla of the rat after peripheral inflammatory injury.Connexin-43 induces chemokine release from spinal cord astrocytes to maintain late-phase neuropathic pain in mice.The astrocyte-targeted therapy by Bushi for the neuropathic pain in mice.Long lasting pain hypersensitivity following ligation of the tendon of the masseter muscle in rats: a model of myogenic orofacial painSpinal astrocytic activation is involved in a virally-induced rat model of neuropathic painCombining ketamine with astrocytic inhibitor as a potential analgesic strategy for neuropathic pain ketamine, astrocytic inhibitor and pain.Intrathecal injection of spironolactone attenuates radicular pain by inhibition of spinal microglia activation in a rat modelFocal Inflammation Causes Carbenoxolone-Sensitive Tactile Hypersensitivity in MiceContribution of Primary Afferent Input to Trigeminal Astroglial Hyperactivity, Cytokine Induction and NMDA Receptor Phosphorylation.Dietary grape seed polyphenols repress neuron and glia activation in trigeminal ganglion and trigeminal nucleus caudalisResolvin D1 reverses chronic pancreatitis-induced mechanical allodynia, phosphorylation of NMDA receptors, and cytokines expression in the thoracic spinal dorsal hornEvidence for brain glial activation in chronic pain patients.Cannabinoid receptor type 1 antagonist, AM251, attenuates mechanical allodynia and thermal hyperalgesia after burn injury
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Glial-cytokine-neuronal interactions underlying the mechanisms of persistent pain
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on May 2007
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vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
@cs
name
Glial-cytokine-neuronal interactions underlying the mechanisms of persistent pain
@en
Glial-cytokine-neuronal interactions underlying the mechanisms of persistent pain.
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type
label
Glial-cytokine-neuronal interactions underlying the mechanisms of persistent pain
@en
Glial-cytokine-neuronal interactions underlying the mechanisms of persistent pain.
@nl
prefLabel
Glial-cytokine-neuronal interactions underlying the mechanisms of persistent pain
@en
Glial-cytokine-neuronal interactions underlying the mechanisms of persistent pain.
@nl
P2093
P2860
P1476
Glial-cytokine-neuronal interactions underlying the mechanisms of persistent pain
@en
P2093
Kohei Shimizu
Mineo Watanabe
Ronald Dubner
Shiping Zou
Stacey C LaGraize
P2860
P304
P356
10.1523/JNEUROSCI.0176-07.2007
P407
P577
2007-05-01T00:00:00Z