N-Methyl-D-aspartate receptor (NMDAR) independent maintenance of inflammatory pain. - Wikidata - awgldk - TriplyDB

N-Methyl-D-aspartate receptor (NMDAR) independent maintenance of inflammatory pain.

N-Methyl-D-aspartate receptor (NMDAR) independent maintenance of inflammatory pain.

http://www.wikidata.org/entity/Q33701859

about

Interactions of Opioids and HIV Infection in the Pathogenesis of Chronic Pain Differences in neural-immune gene expression response in rat spinal dorsal horn correlates with variations in electroacupuncture analgesia Interleukin-1 receptor type 1 is overexpressed in neurons but not in glial cells within the rat superficial spinal dorsal horn in complete Freund adjuvant-induced inflammatory pain.Peripheral N-methyl-d-aspartate receptors contribute to mechanical hypersensitivity in a rat model of inflammatory temporomandibular joint pain.Involvement of EphB1 receptors signalling in models of inflammatory and neuropathic pain.Interactions between the immune and nervous systems in pain.The role of trigeminal interpolaris-caudalis transition zone in persistent orofacial pain.Dynamic weight bearing is an efficient and predictable method for evaluation of arthritic nociception and its pathophysiological mechanisms in mice.Glia and pain: is chronic pain a gliopathy?NFκB-mediated CXCL1 production in spinal cord astrocytes contributes to the maintenance of bone cancer pain in mice.Targeting astrocyte signaling for chronic pain.Calcium-permeable ion channels in pain signaling.Endogenous analgesia, dependence, and latent pain sensitization Constitutive μ-opioid receptor activity leads to long-term endogenous analgesia and dependence Light touch induces ERK activation in superficial dorsal horn neurons after inflammation: involvement of spinal astrocytes and JNK signaling in touch-evoked central sensitization and mechanical allodynia.Minocycline and fluorocitrate suppress spinal nociceptive signaling in intrathecal IL-1β-induced thermal hyperalgesic rats.AMPK activation by peri-sciatic nerve administration of ozone attenuates CCI-induced neuropathic pain in rats.

P2860

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P2860

N-Methyl-D-aspartate receptor (NMDAR) independent maintenance of inflammatory pain.

http://www.wikidata.org/entity/Q33701859

description

2009 nî lūn-bûn

@nan

2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

N-Methyl-D-aspartate receptor (NMDAR) independent maintenance of inflammatory pain.

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N-Methyl-D-aspartate receptor (NMDAR) independent maintenance of inflammatory pain.

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type

label

N-Methyl-D-aspartate receptor (NMDAR) independent maintenance of inflammatory pain.

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N-Methyl-D-aspartate receptor (NMDAR) independent maintenance of inflammatory pain.

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prefLabel

N-Methyl-D-aspartate receptor (NMDAR) independent maintenance of inflammatory pain.

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N-Methyl-D-aspartate receptor (NMDAR) independent maintenance of inflammatory pain.

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J.PAIN.2009.11.003

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P1476

N-Methyl-D-aspartate receptor (NMDAR) independent maintenance of inflammatory pain

@en

P2093

Charles E Inturrisi

http://www.w3.org/2001/XMLSchema#string

Cristina Tamasdan

http://www.w3.org/2001/XMLSchema#string

Qinghao Xu

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Sarah J Shin

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P2860

Pathological and protective roles of glia in chronic pain

A conditional deletion of the NR1 subunit of the NMDA receptor in adult spinal cord dorsal horn reduces NMDA currents and injury-induced pain

Neuronal plasticity: increasing the gain in pain

Quantitative assessment of tactile allodynia in the rat paw

ERK/MAP kinase is chronically activated in human reactive astrocytes.

Activation of the neuronal extracellular signal-regulated kinase 2 in the spinal cord dorsal horn is required for complete Freund's adjuvant-induced pain hypersensitivity

MAP kinase and pain.

Transcriptional and posttranslational plasticity and the generation of inflammatory pain

Adeno-associated virus effectively mediates conditional gene modification in the brain

Central sensitization and LTP: do pain and memory share similar mechanisms?

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237-246

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scholarly article

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10.1016/J.PAIN.2009.11.003

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2

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148

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Amanda R Weyerbacher

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2009-12-11T00:00:00Z

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40682876

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20005044

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2831745

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