Oleic acid directly regulates POMC neuron excitability in the hypothalamus - Wikidata - awgldk - TriplyDB

Oleic acid directly regulates POMC neuron excitability in the hypothalamus

Oleic acid directly regulates POMC neuron excitability in the hypothalamus

http://www.wikidata.org/entity/Q37190525

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Control of metabolic and cardiovascular function by the leptin-brain melanocortin pathway FAT/CD36: a major regulator of neuronal fatty acid sensing and energy homeostasis in rats and mice Antagonistic modulation of NPY/AgRP and POMC neurons in the arcuate nucleus by noradrenalin.Developmental gene x environment interactions affecting systems regulating energy homeostasis and obesity.Gene-environment interactions controlling energy and glucose homeostasis and the developmental origins of obesity ATP-Sensitive Potassium Channel-Deficient Mice Show Hyperphagia but Are Resistant to Obesity.Metabolic sensing and the brain: who, what, where, and how?Regulation of the orexigenic neuropeptide, enkephalin, by PPARδ and fatty acids in neurons of the hypothalamus and forebrain.Brainstem nutrient sensing in the nucleus of the solitary tract inhibits feeding.New aspects of melanocortin signaling: a role for PRCP in α-MSH degradation Fatty acid-induced astrocyte ketone production and the control of food intake.Effects of maternal genotype and diet on offspring glucose and fatty acid-sensing ventromedial hypothalamic nucleus neurons.Regulation of hypothalamic neuronal sensing and food intake by ketone bodies and fatty acids.Targeting lipid sensing in the central nervous system: new therapy against the development of obesity and type 2 diabetes.Integration of satiety signals by the central nervous system.Physiological and pathophysiological implications of lipid sensing in the brain.Does nutrient sensing determine how we "see" food?Stereoselective oxidation of regioisomeric octadecenoic acids by fatty acid dioxygenases.POMC Neurons: From Birth to Death.Tanycytes and a differential fatty acid metabolism in the hypothalamus.Energy imbalance alters Ca2+ handling and excitability of POMC neurons.Oleate induces KATP channel-dependent hyperpolarization in mouse hypothalamic glucose-excited neurons without altering cellular energy charge.Mediobasal hypothalamic leucine sensing regulates food intake through activation of a hypothalamus-brainstem circuit.Dietary intake of unsaturated fatty acids modulates physiological properties of entorhinal cortex neurons in mice.[Role of the nucleus of the solitary tract in the detection and integration of multiple metabolic signals].Impact of dietary fats on brain functions.Oleic Acid in the Ventral Tegmental Area Inhibits Feeding, Food Reward, and Dopamine Tone.Rapid sensing of l-leucine by human and murine hypothalamic neurons: Neurochemical and mechanistic insights.[Role of brain lipid sensing in nervous regulation of energy balance].

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Oleic acid directly regulates POMC neuron excitability in the hypothalamus

http://www.wikidata.org/entity/Q37190525

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 04 March 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Oleic acid directly regulates POMC neuron excitability in the hypothalamus

@en

Oleic acid directly regulates POMC neuron excitability in the hypothalamus.

@nl

type

label

Oleic acid directly regulates POMC neuron excitability in the hypothalamus

@en

Oleic acid directly regulates POMC neuron excitability in the hypothalamus.

@nl

prefLabel

Oleic acid directly regulates POMC neuron excitability in the hypothalamus

@en

Oleic acid directly regulates POMC neuron excitability in the hypothalamus.

@nl

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Journal of Neurophysiology

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Oleic acid directly regulates POMC neuron excitability in the hypothalamus

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Roger Gutierrez-Juarez

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Streamson Chua

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Ya Su

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Young-Hwan Jo

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P2860

Phosphatidylinositol 3'-kinase is activated by association with IRS-1 during insulin stimulation

Leptin and insulin stimulation of signalling pathways in arcuate nucleus neurones: PI3K dependent actin reorganization and KATP channel activation

The orphan G protein-coupled receptor GPR40 is activated by medium and long chain fatty acids.

Free fatty acids regulate insulin secretion from pancreatic beta cells through GPR40.

Agouti protein is an antagonist of the melanocyte-stimulating-hormone receptor

Obesity wars: molecular progress confronts an expanding epidemic

Mechanism of cloned ATP-sensitive potassium channel activation by oleoyl-CoA

Role of melanocortinergic neurons in feeding and the agouti obesity syndrome

Inhibition of hypothalamic carnitine palmitoyltransferase-1 decreases food intake and glucose production

Central nervous system control of food intake

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2305-2316

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scholarly article

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10.1152/JN.91294.2008

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5

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101

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2009-03-04T00:00:00Z

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19261705

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2681442

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