Prolonged exposure to high dietary lipids is not associated with lipotoxicity in heart failure.
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Hearts from mice fed a non-obesogenic high-fat diet exhibit changes in their oxidative state, calcium and mitochondria in parallel with increased susceptibility to reperfusion injuryPhysiological and structural differences in spatially distinct subpopulations of cardiac mitochondria: influence of cardiac pathologiesCardioprotective effects of dietary lipids evident in the time-dependent alterations of cardiac function and gene expression following myocardial infarction.High-fat feeding-induced hyperinsulinemia increases cardiac glucose uptake and mitochondrial function despite peripheral insulin resistance.Metabolic Remodeling in Diabetic Cardiomyopathy.Lipotoxicity in the heart.High-fat, low-carbohydrate diet promotes arrhythmic death and increases myocardial ischemia-reperfusion injury in rats.The myocardial contractile response to physiological stress improves with high saturated fat feeding in heart failure.Myocardial insulin resistance induced by high fat feeding in heart failure is associated with preserved contractile function.High fat feeding in mice is insufficient to induce cardiac dysfunction and does not exacerbate heart failure.Chronic high-fat diet-induced obesity decreased survival and increased hypertrophy of rats with experimental eccentric hypertrophy from chronic aortic regurgitation.High intake of saturated fat, but not polyunsaturated fat, improves survival in heart failure despite persistent mitochondrial defects.Dietary linoleate preserves cardiolipin and attenuates mitochondrial dysfunction in the failing rat heartNormalizing the metabolic phenotype after myocardial infarction: impact of subchronic high fat feedingCardiac-specific deletion of acetyl CoA carboxylase 2 prevents metabolic remodeling during pressure-overload hypertrophyCeramide synthase 5 mediates lipid-induced autophagy and hypertrophy in cardiomyocytes.Diabetes, perioperative ischaemia and volatile anaesthetics: consequences of derangements in myocardial substrate metabolism.Aldehyde stress and up-regulation of Nrf2-mediated antioxidant systems accompany functional adaptations in cardiac mitochondria from mice fed n-3 polyunsaturated fatty acids.Dietary omega-3 fatty acids alter cardiac mitochondrial phospholipid composition and delay Ca2+-induced permeability transition.Impact of dietary fatty acids on cardiac arrhythmogenesis.Creating and curing fatty hearts.Metabolic therapy at the crossroad: how to optimize myocardial substrate utilization?Cardiac metabolism and its interactions with contraction, growth, and survival of cardiomyocytes.Hyperleptinemia Exacerbates High-Fat Diet-Mediated Atrial Fibrosis and Fibrillation.A high fat diet increases mitochondrial fatty acid oxidation and uncoupling to decrease efficiency in rat heartCritical role of complex III in the early metabolic changes following myocardial infarction.Nox4 reprograms cardiac substrate metabolism via protein O-GlcNAcylation to enhance stress adaptation.
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P2860
Prolonged exposure to high dietary lipids is not associated with lipotoxicity in heart failure.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 03 March 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Prolonged exposure to high die ...... lipotoxicity in heart failure.
@en
Prolonged exposure to high die ...... lipotoxicity in heart failure.
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type
label
Prolonged exposure to high die ...... lipotoxicity in heart failure.
@en
Prolonged exposure to high die ...... lipotoxicity in heart failure.
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prefLabel
Prolonged exposure to high die ...... lipotoxicity in heart failure.
@en
Prolonged exposure to high die ...... lipotoxicity in heart failure.
@nl
P2093
P2860
P1476
Prolonged exposure to high die ...... lipotoxicity in heart failure.
@en
P2093
Brian D Hoit
Charles L Hoppel
Julie H Rennison
Margaret P Chandler
Tracy A McElfresh
Vijay R Anand
Xiaoqin Chen
P2860
P304
P356
10.1016/J.YJMCC.2009.02.019
P577
2009-03-03T00:00:00Z