Obesity in mice with adipocyte-specific deletion of clock component Arntl. - Wikidata - awgldk - TriplyDB

Obesity in mice with adipocyte-specific deletion of clock component Arntl.

Obesity in mice with adipocyte-specific deletion of clock component Arntl.

http://www.wikidata.org/entity/Q37195570

about

Conceptual heuristic models of the interrelationships between obesity and the occupational environment Circadian regulation of metabolic homeostasis: causes and consequences Interdependence of nutrient metabolism and the circadian clock system: Importance for metabolic health Physiological links between circadian rhythms, metabolism and nutrition Nutrients, Clock Genes, and Chrononutrition Circadian regulation of adipose function The chondrocyte clock gene Bmal1 controls cartilage homeostasis and integrity.Circadian clocks, feeding time, and metabolic homeostasis The tick tock of odontogenesis The functional significance of the skeletal muscle clock: lessons from Bmal1 knockout models Therapeutic applications of circadian rhythms for the cardiovascular system Adipose Clocks: Burning the Midnight Oil Physiological responses to food intake throughout the day Effects of Experimental Sleep Restriction on Weight Gain, Caloric Intake, and Meal Timing in Healthy Adults Circadian Rhythms, Metabolism, and Chrononutrition in Rodents and Humans Circadian System and Glucose Metabolism: Implications for Physiology and Disease Metabolism as an integral cog in the mammalian circadian clockwork Contribution of daily and seasonal biorhythms to obesity in humans Circadian Rhythm and Sleep Disruption: Causes, Metabolic Consequences, and Countermeasures Cardiac Per2 functions as novel link between fatty acid metabolism and myocardial inflammation during ischemia and reperfusion injury of the heart Nighttime eating and breast cancer among Chinese women in Hong Kong Nutrition and the circadian system Daily Eating Patterns and Their Impact on Health and Disease.The biological clock is regulated by adrenergic signaling in brown fat but is dispensable for cold-induced thermogenesis.Chronobiology and obesity: Interactions between circadian rhythms and energy regulation.Myeloid cell-specific disruption of Period1 and Period2 exacerbates diet-induced inflammation and insulin resistance Dopamine D2 receptors and the circadian clock reciprocally mediate antipsychotic drug-induced metabolic disturbances.The Role of the Molecular Clock in Skeletal Muscle and What It Is Teaching Us About Muscle-Bone Crosstalk.Hepatic gene therapy rescues high-fat diet responses in circadian Clock mutant mice.Circadian control of glucose metabolism.An Increased Dietary Supply of Medium-Chain Fatty Acids during Early Weaning in Rodents Prevents Excessive Fat Accumulation in Adulthood Effects of obesity on transcriptomic changes and cancer hallmarks in estrogen receptor-positive breast cancer.Increased adipocyte O2 consumption triggers HIF-1α, causing inflammation and insulin resistance in obesity.Circadian gene variants in cancer.Circadian rhythms, Wnt/beta-catenin pathway and PPAR alpha/gamma profiles in diseases with primary or secondary cardiac dysfunction.Circadian Clocks in Articular Cartilage and Bone: A Compass in the Sea of Matrices.Regulation of Mammalian Physiology by Interconnected Circadian and Feeding Rhythms.Dietary exposure to the endocrine disruptor tolylfluanid promotes global metabolic dysfunction in male mice.Impact of nutrients on circadian rhythmicity The Fragile X proteins Fmrp and Fxr2p cooperate to regulate glucose metabolism in mice.

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Obesity in mice with adipocyte-specific deletion of clock component Arntl.

http://www.wikidata.org/entity/Q37195570

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

@zh-hant

name

Obesity in mice with adipocyte-specific deletion of clock component Arntl.

@en

Obesity in mice with adipocyte-specific deletion of clock component Arntl.

@nl

type

label

Obesity in mice with adipocyte-specific deletion of clock component Arntl.

@en

Obesity in mice with adipocyte-specific deletion of clock component Arntl.

@nl

prefLabel

Obesity in mice with adipocyte-specific deletion of clock component Arntl.

@en

Obesity in mice with adipocyte-specific deletion of clock component Arntl.

@nl

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Nature Medicine

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Obesity in mice with adipocyte-specific deletion of clock component Arntl

@en

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Cheryl H Vaughan

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Christopher A Bradfield

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Fenfen Wang

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Garret A Fitzgerald

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Georgios K Paschos

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John A Lawson

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Julian L Griffin

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Michael Eiden

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Salam Ibrahim

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Takeshige Kunieda

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BMAL1 and CLOCK, two essential components of the circadian clock, are involved in glucose homeostasis

The basic-helix-loop-helix-PAS orphan MOP3 forms transcriptionally active complexes with circadian and hypoxia factors

Two defects contribute to hypothalamic leptin resistance in mice with diet-induced obesity

Obesity and metabolic syndrome in circadian Clock mutant mice

GPR120 is an omega-3 fatty acid receptor mediating potent anti-inflammatory and insulin-sensitizing effects

Disruption of the clock components CLOCK and BMAL1 leads to hypoinsulinaemia and diabetes

Adverse metabolic and cardiovascular consequences of circadian misalignment

IL-6 and IL-10 anti-inflammatory activity links exercise to hypothalamic insulin and leptin sensitivity through IKKbeta and ER stress inhibition

Coordination of circadian timing in mammals

Summaries of Affymetrix GeneChip probe level data

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1768-1777

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10.1038/NM.2979

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12

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18

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Gregory R Grant

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2012-11-11T00:00:00Z

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