Mclk1+/- mice are not resistant to the development of atherosclerosis. - Wikidata - awgldk - TriplyDB

Mclk1+/- mice are not resistant to the development of atherosclerosis.

Mclk1+/- mice are not resistant to the development of atherosclerosis.

http://www.wikidata.org/entity/Q37196450

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Is there a place for coenzyme Q in the management of metabolic disorders associated with obesity?

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Mclk1+/- mice are not resistant to the development of atherosclerosis.

http://www.wikidata.org/entity/Q37196450

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 05 May 2009

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

Mclk1+/- mice are not resistant to the development of atherosclerosis.

@en

Mclk1+/- mice are not resistant to the development of atherosclerosis.

@nl

type

label

Mclk1+/- mice are not resistant to the development of atherosclerosis.

@en

Mclk1+/- mice are not resistant to the development of atherosclerosis.

@nl

prefLabel

Mclk1+/- mice are not resistant to the development of atherosclerosis.

@en

Mclk1+/- mice are not resistant to the development of atherosclerosis.

@nl

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Lipids in Health and Disease

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Mclk1+/- mice are not resistant to the development of atherosclerosis.

@en

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Bryan G Hughes

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Siegfried Hekimi

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P2860

Evolutionary conservation of the clk-1-dependent mechanism of longevity: loss of mclk1 increases cellular fitness and lifespan in mice

A new member of the family of di-iron carboxylate proteins. Coq7 (clk-1), a membrane-bound hydroxylase involved in ubiquinone biosynthesis.

A review of recent studies on malondialdehyde as toxic molecule and biological marker of oxidative stress

Malondialdehyde and thiobarbituric acid-reactivity as diagnostic indices of lipid peroxidation and peroxidative tissue injury

Mouse homologue of coq7/clk-1, longevity gene in Caenorhabditis elegans, is essential for coenzyme Q synthesis, maintenance of mitochondrial integrity, and neurogenesis

Reversal of the mitochondrial phenotype and slow development of oxidative biomarkers of aging in long-lived Mclk1+/- mice

Early mitochondrial dysfunction in long-lived Mclk1+/- mice

Ubiquinone is necessary for mouse embryonic development but is not essential for mitochondrial respiration

Hypercholesterolemia in low density lipoprotein receptor knockout mice and its reversal by adenovirus-mediated gene delivery

Atherosclerosis. the road ahead

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16

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scholarly article

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10.1186/1476-511X-8-16

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8

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2009-05-05T00:00:00Z

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24402740

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1013490034

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19416523

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atherosclerosis

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2683836

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