Abnormal insulin sensitivity persists up to three years in pediatric patients post-burn - Wikidata - awgldk - TriplyDB

Abnormal insulin sensitivity persists up to three years in pediatric patients post-burn

Abnormal insulin sensitivity persists up to three years in pediatric patients post-burn

http://www.wikidata.org/entity/Q37198029

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Alcohol's Burden on Immunity Following Burn, Hemorrhagic Shock, or Traumatic Brain Injury Physical rehabilitation of pediatric burns Stress hyperglycemia in pediatric critical illness: the intensive care unit adds to the stress!The impact of severe burns on skeletal muscle mitochondrial function Thrombocytopenia induces multiple intracranial hemorrhages in patients with severe burns: A review of 16 cases The leading causes of death after burn injury in a single pediatric burn center Impact of stress-induced diabetes on outcomes in severely burned children Clinical review: Glucose control in severely burned patients - current best practice.Extent and magnitude of catecholamine surge in pediatric burned patients.Insulin effects on glucose tolerance, hypermetabolic response, and circadian-metabolic protein expression in a rat burn and disuse model Effects of burn injury, cold stress and cutaneous wound injury on the morphology and energy metabolism of murine brown adipose tissue (BAT) in vivo.Long-term persistance of the pathophysiologic response to severe burn injury.Intensive insulin therapy in severely burned pediatric patients: a prospective randomized trial.Predictors of insulin resistance in pediatric burn injury survivors 24 to 36 months postburn.Leukocyte infiltration and activation of the NLRP3 inflammasome in white adipose tissue following thermal injury Five-year outcomes after oxandrolone administration in severely burned children: a randomized clinical trial of safety and efficacy.The effect of ketoconazole on post-burn inflammation, hypermetabolism and clinical outcomes.Long-term oxandrolone treatment increases muscle protein net deposition via improving amino acid utilization in pediatric patients 6 months after burn injury.Endoplasmic reticulum stress and insulin resistance post-trauma: similarities to type 2 diabetes.Effects of pharmacological interventions on muscle protein synthesis and breakdown in recovery from burns.Long-term mortality among older adults with burn injury: a population-based study in Australia Adult patients are more catabolic than children during acute phase after burn injury: a retrospective analysis on muscle protein kinetics.Decreased skin-mediated detoxification contributes to oxidative stress and insulin resistance.Pathophysiologic Response to Burns in the Elderly Postburn Hypermetabolism: Past, Present, and Future.Signals from fat after injury: plasma adipokines and ghrelin concentrations in the severely burned.17β-Estradiol reappropriates mass lost to the hypermetabolic state in thermally injured rats Whole body and skeletal muscle protein turnover in recovery from burns.The skin function: a factor of anti-metabolic syndrome Skeletal muscle is anabolically unresponsive to an amino acid infusion in pediatric burn patients 6 months postinjury.Burned Adults Develop Profound Glucose Intolerance.Survivors versus nonsurvivors postburn: differences in inflammatory and hypermetabolic trajectories Nutrition in burns: Galveston contributions Lack of Th17 cell generation in patients with severe burn injuries The hepatic response to thermal injury: is the liver important for postburn outcomes?Fenofibrate does not affect burn-induced hepatic endoplasmic reticulum stress Nephrilin peptide modulates a neuroimmune stress response in rodent models of burn trauma and sepsis.Effects of exercise training on resting energy expenditure and lean mass during pediatric burn rehabilitation.Post-burn hepatic insulin resistance is associated with endoplasmic reticulum (ER) stress Nicotinamide overload may play a role in the development of type 2 diabetes

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Abnormal insulin sensitivity persists up to three years in pediatric patients post-burn

http://www.wikidata.org/entity/Q37198029

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on 24 February 2009

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

Abnormal insulin sensitivity persists up to three years in pediatric patients post-burn

@en

Abnormal insulin sensitivity persists up to three years in pediatric patients post-burn.

@nl

type

label

Abnormal insulin sensitivity persists up to three years in pediatric patients post-burn

@en

Abnormal insulin sensitivity persists up to three years in pediatric patients post-burn.

@nl

prefLabel

Abnormal insulin sensitivity persists up to three years in pediatric patients post-burn

@en

Abnormal insulin sensitivity persists up to three years in pediatric patients post-burn.

@nl

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The Journal of Clinical Endocrinology and Metabolism

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Abnormal insulin sensitivity persists up to three years in pediatric patients post-burn

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David N Herndon

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Gabriela A Kulp

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Gerd G Gauglitz

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Marc G Jeschke

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Walter J Meyer

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P2860

Homeostasis model assessment: insulin resistance and ?-cell function from fasting plasma glucose and insulin concentrations in man

Monocyte chemotactic protein-1 is a potential player in the negative cross-talk between adipose tissue and skeletal muscle

Intensive insulin therapy in critically ill patients

Growth hormone and cortisol secretion in patients with burn injury.

Stress-induced hyperglycemia.

Pathophysiologic response to severe burn injury.

The metabolic basis of the increase of the increase in energy expenditure in severely burned patients.

Stress hyperglycaemia and increased risk of death after myocardial infarction in patients with and without diabetes: a systematic overview.

Glucose clamp technique: a method for quantifying insulin secretion and resistance.

The role of hyperglycemia in acute stroke.

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1656-1664

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scholarly article

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10.1210/JC.2008-1947

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5

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94

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2009-02-24T00:00:00Z

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24037751

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19240154

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2684478

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