Nicotinamide overload may play a role in the development of type 2 diabetes - Wikidata - awgldk - TriplyDB

Nicotinamide overload may play a role in the development of type 2 diabetes

Nicotinamide overload may play a role in the development of type 2 diabetes

http://www.wikidata.org/entity/Q37461169

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Vitamin paradox in obesity: Deficiency or excess?Vitamins and type 2 diabetes mellitus Novel applications of trophic factors, Wnt and WISP for neuronal repair and regeneration in metabolic disease Excess vitamin intake: An unrecognized risk factor for obesity Diabetes mellitus: channeling care through cellular discovery B-vitamin consumption and the prevalence of diabetes and obesity among the US adults: population based ecological study.Assessing the metabolic effects of prednisolone in healthy volunteers using urine metabolic profiling.Rapid Elimination of Blood Alcohol Using Erythrocytes: Mathematical Modeling and In Vitro Study.Chronic niacin overload may be involved in the increased prevalence of obesity in US children.A review of nicotinamide: treatment of skin diseases and potential side effects.Novel avenues of drug discovery and biomarkers for diabetes mellitus.Association of nicotinamide-N-methyltransferase mRNA expression in human adipose tissue and the plasma concentration of its product, 1-methylnicotinamide, with insulin resistance.Serum N(1)-Methylnicotinamide Is Associated With Obesity and Diabetes in Chinese.Decreased skin-mediated detoxification contributes to oxidative stress and insulin resistance.Novel directions for diabetes mellitus drug discovery The skin function: a factor of anti-metabolic syndrome Early infant exposure to excess multivitamin: a risk factor for autism?Gut microbiota from metabolic disease-resistant, macrophage-specific RIP140 knockdown mice improves metabolic phenotype and gastrointestinal integrity.Dietary methyl-consuming compounds and metabolic syndrome.Effect of non-surgical periodontal treatment on visfatin concentrations in serum and gingival crevicular fluid of patients with chronic periodontitis and type 2 diabetes mellitus.Effect of combined therapy of diabinese and nicotinic acid on liver enzymes in rabbits with dithizone-induced diabetes.Nicotinamide supplementation induces detrimental metabolic and epigenetic changes in developing rats.Maternal nicotinamide supplementation causes global DNA hypomethylation, uracil hypo-incorporation and gene expression changes in fetal rats.Excess nicotinamide inhibits methylation-mediated degradation of catecholamines in normotensives and hypertensives.Excessive nicotinic acid increases methyl consumption and hydrogen peroxide generation in rats.Management of nicotinamide N-methyltransferase overexpression: inhibit the enzyme or reduce nicotinamide intake? Reply to Zhou S, Li D, Zhou Y [letter].A Discussion on the Relationship between Skin Lipid Metabolism and Whole-Body Glucose and Lipid Metabolism: Systematic Review

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Nicotinamide overload may play a role in the development of type 2 diabetes

http://www.wikidata.org/entity/Q37461169

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on December 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Nicotinamide overload may play a role in the development of type 2 diabetes

@en

Nicotinamide overload may play a role in the development of type 2 diabetes.

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type

label

Nicotinamide overload may play a role in the development of type 2 diabetes

@en

Nicotinamide overload may play a role in the development of type 2 diabetes.

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prefLabel

Nicotinamide overload may play a role in the development of type 2 diabetes

@en

Nicotinamide overload may play a role in the development of type 2 diabetes.

@nl

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World Journal of Gastroenterology

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Nicotinamide overload may play a role in the development of type 2 diabetes

@en

P2093

Chang-Bin Sun

http://www.w3.org/2001/XMLSchema#string

Cong-Long Zheng

http://www.w3.org/2001/XMLSchema#string

Da Li

http://www.w3.org/2001/XMLSchema#string

Dong-Ju Jiang

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Fu-Cheng Xiao

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Fu-Ning Bian

http://www.w3.org/2001/XMLSchema#string

Lai-Bin Dong

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Li-Bin Zhang

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Li-Xin Jing

http://www.w3.org/2001/XMLSchema#string

Ming Guo

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P2860

Reactive oxygen species have a causal role in multiple forms of insulin resistance

Validation of an HPLC method for the determination of urinary and plasma levels of N1-methylnicotinamide, an endogenous marker of renal cationic transport and plasma flow.

Waging war on modern chronic diseases: primary prevention through exercise biology.

Safety of high-dose nicotinamide: a review.

Increased consumption of refined carbohydrates and the epidemic of type 2 diabetes in the United States: an ecologic assessment.

Antipsychotic medications: metabolic and cardiovascular risk.

NAD+/NADH and NADP+/NADPH in cellular functions and cell death: regulation and biological consequences.

Human liver aldehyde oxidase: inhibition by 239 drugs.

Lipid-lowering agents that cause drug-induced hepatotoxicity.

Poly(ADP-ribose) polymerase: the nuclear target in signal transduction and its role in brain ischemia-reperfusion injury.

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5674-5684

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scholarly article

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10.3748/WJG.15.5674

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45

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15

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2009-12-01T00:00:00Z

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19960564

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type-2 diabetes

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2789220

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