High-mobility group box protein 1 (HMGB1): an alarmin mediating the pathogenesis of rheumatic disease
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Cell death in the pathogenesis of immune-mediated diseases: the role of HMGB1 and DAMP-PAMP complexesMicroparticles as mediators and biomarkers of rheumatic diseaseHMGB1 conveys immunosuppressive characteristics on regulatory and conventional T cellsA non-aggressive, highly efficient, enzymatic method for dissociation of human brain-tumors and brain-tissues to viable single-cellsToll-like receptor-mediated IRE1α activation as a therapeutic target for inflammatory arthritisHologenome analysis of two marine sponges with different microbiomesA New-Age for Biologic Therapies: Long-Term Drug-Free Therapy with BiP?Association of increased Treg and Th17 with pathogenesis of moyamoya disease.Electroacupuncture Alleviates Postoperative Cognitive Dysfunction in Aged Rats by Inhibiting Hippocampal Neuroinflammation Activated via Microglia/TLRs Pathway.Nucleic acid sensing receptors in systemic lupus erythematosus: development of novel DNA- and/or RNA-like analogues for treating lupus.DAMPening inflammation by modulating TLR signalling.High mobility group box 1 potentiates the pro-inflammatory effects of interleukin-1β in osteoarthritic synoviocytes.Chondroprotective effects and mechanisms of resveratrol in advanced glycation end products-stimulated chondrocytesHuman schistosomiasis is associated with endotoxemia and Toll-like receptor 2- and 4-bearing B cells.HMGB1 in health and disease.Extracellular high-mobility group box 1 is increased in patients with Behçet's disease with intestinal involvementTemporal and spatial patterns of endogenous danger signal expression after wound healing and in response to lymphedema.High-fat diet-induced adipocyte cell death occurs through a cyclophilin D intrinsic signaling pathway independent of adipose tissue inflammationClearance kinetics and matrix binding partners of the receptor for advanced glycation end products.Expression of RAGE and HMGB1 in thymic epithelial tumors, thymic hyperplasia and regular thymic morphology.Effects of progesterone and estradiol sex hormones on the release of microparticles by RAW 264.7 macrophages stimulated by Poly(I:C).HMGB1 and microparticles as mediators of the immune response to cell death.Innate immunity sensors participating in pathophysiology of joint diseases: a brief overview.Pathogenesis and potential therapeutic targets in systemic lupus erythematosus: from bench to bedsideExpression of the receptor for advanced glycation end products, a target for high mobility group box 1 protein, and its role in chronic recalcitrant rhinosinusitis with nasal polyps.HMGB1, an alarmin promoting HIV dissemination and latency in dendritic cellsToll-like receptor deficiency worsens inflammation and lymphedema after lymphatic injury.[Nle4, D-Phe7]-α-MSH Inhibits Toll-Like Receptor (TLR)2- and TLR4-Induced Microglial Activation and Promotes a M2-Like Phenotype.Renal expression and serum levels of high mobility group box 1 protein in lupus nephritisHMGB1 May Be a Biomarker for Predicting the Outcome in Patients with Polymyositis /Dermatomyositis with Interstitial Lung Disease.Up-regulated S100 calcium binding protein A8 in Plasmodium-infected patients correlates with CD4(+)CD25(+)Foxp3 regulatory T cell generation.Staphylococcus aureus activation of caspase 1/calpain signaling mediates invasion through human keratinocytesEffects of bone marrow MSCs transfected with sRAGE on the intervention of HMGB1 induced immuno-inflammatory reactionHMGB1, a novel biomarker of inflammatory demyelinating disease: an interview with Dr. Robert A. HarrisThe selective alpha7 agonist GTS-21 attenuates cytokine production in human whole blood and human monocytes activated by ligands for TLR2, TLR3, TLR4, TLR9, and RAGE.HMGB1 acts in synergy with lipopolysaccharide in activating rheumatoid synovial fibroblasts via p38 MAPK and NF-κB signaling pathways.Thrombomodulin Protects Against Bacterial Keratitis, Is Anti-Inflammatory, but Not Angiogenic.Tempering the wrath of RAGE: an emerging therapeutic strategy against diabetic complications, neurodegeneration, and inflammationFungal morphogenetic pathways are required for the hallmark inflammatory response during Candida albicans vaginitis.The translocation of HMGB1 during cell activation and cell death.
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High-mobility group box protein 1 (HMGB1): an alarmin mediating the pathogenesis of rheumatic disease
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 30 June 2008
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
@da
vědecký článek
@cs
name
High-mobility group box protei ...... hogenesis of rheumatic disease
@en
High-mobility group box protein 1
@nl
type
label
High-mobility group box protei ...... hogenesis of rheumatic disease
@en
High-mobility group box protein 1
@nl
prefLabel
High-mobility group box protei ...... hogenesis of rheumatic disease
@en
High-mobility group box protein 1
@nl
P2860
P921
P356
P1476
High-mobility group box protei ...... hogenesis of rheumatic disease
@en
P2093
David S Pisetsky
Helena Erlandsson-Harris
P2860
P2888
P356
10.1186/AR2440
P577
2008-06-30T00:00:00Z
P5875
P6179
1020531111