TLR2 synergizes with both TLR4 and TLR9 for induction of the MyD88-dependent splenic cytokine and chemokine response to Streptococcus pneumoniae.
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Inflammasome/IL-1β Responses to Streptococcal PathogensLimited anti-inflammatory role for interleukin-1 receptor like 1 (ST2) in the host response to murine postinfluenza pneumococcal pneumonia.Streptococcus pneumoniae synergizes with nontypeable Haemophilus influenzae to induce inflammation via upregulating TLR2Streptococcus pneumoniae nasopharyngeal colonization induces type I interferons and interferon-induced gene expression.TLR1/2 activation during heterologous prime-boost vaccination (DNA-MVA) enhances CD8+ T Cell responses providing protection against Leishmania (Viannia)TLR-2 independent recognition of Mycobacterium tuberculosis by CD11c+ pulmonary cells from old miceTargeting the TLR9-MyD88 pathway in the regulation of adaptive immune responses.TLR-mediated inflammatory responses to Streptococcus pneumoniae are highly dependent on surface expression of bacterial lipoproteinsThe role of TLR2 in the host response to pneumococcal pneumonia in absence of the spleen.Immune receptors involved in Streptococcus suis recognition by dendritic cells.Toll-like receptors in health and disease in the brain: mechanisms and therapeutic potential.Morphine inhibits murine dendritic cell IL-23 production by modulating Toll-like receptor 2 and Nod2 signaling.Macrophages sense and kill bacteria through carbon monoxide-dependent inflammasome activation.Role of phosphoinositide 3-kinase-Akt signaling pathway in the age-related cytokine dysregulation in splenic macrophages stimulated via TLR-2 or TLR-4 receptorsMechanisms of interferon-γ production by neutrophils and its function during Streptococcus pneumoniae pneumonia.Angptl4 is upregulated under inflammatory conditions in the bone marrow of mice, expands myeloid progenitors, and accelerates reconstitution of platelets after myelosuppressive therapyToll-Like Receptor 9-Mediated Inflammation Triggers Alveolar Bone Loss in Experimental Murine PeriodontitisImportance of Toll-like receptor 9 in host defense against M1T1 group A Streptococcus infectionsSynergistic interactions of TLR2/6 and TLR9 induce a high level of resistance to lung infection in mice.Macrophages pulsed with Streptococcus pneumoniae elicit a T cell-dependent antibody response upon transfer into naive miceNoncovalent association of protein and capsular polysaccharide on bacteria-sized latex beads as a model for polysaccharide-specific humoral immunity to intact gram-positive extracellular bacteriaRole of p38 and early growth response factor 1 in the macrophage response to group B streptococcus.Anatomical site-specific contributions of pneumococcal virulence determinants.B and CD4+ T-cell expression of TLR2 is critical for optimal induction of a T-cell-dependent humoral immune response to intact Streptococcus pneumoniaePneumococci: immunology of the innate host response.Alcohol abuse and Streptococcus pneumoniae infections: consideration of virulence factors and impaired immune responses.TLR9: an important molecule in the fight against hepatitis B virus.Immune ageing and susceptibility to Streptococcus pneumoniae.Distinct Toll-like receptor signals regulate cerebral parasite load and interferon α/β and tumor necrosis factor α-dependent T-cell infiltration in the brains of Trypanosoma brucei-infected mice.Group A streptococcus activates type I interferon production and MyD88-dependent signaling without involvement of TLR2, TLR4, and TLR9.Differential use of TLR2 and TLR9 in the regulation of immune responses during the infection with Trypanosoma cruzi.Recognition of Streptococcus pneumoniae and muramyl dipeptide by NOD2 results in potent induction of MMP-9, which can be controlled by lipopolysaccharide stimulation.Oridonin attenuates the release of pro-inflammatory cytokines in lipopolysaccharide-induced RAW264.7 cells and acute lung injuryType I interferons protect from Toll-like receptor 9-associated liver injury and regulate IL-1 receptor antagonist in mice.Possible involvement of toll-like receptors in the pathogenesis of myasthenia gravis.Pathogen-associated molecular patterns alter molecular clock gene expression in mouse splenocytes.Enhancement of natural killer activity and IFN-γ production in an IL-12-dependent manner by a Brassica rapa L.Innate immunity to pneumococcal infection of the central nervous system depends on toll-like receptor (TLR) 2 and TLR4.Expression of Toll-like receptors 2, 4 and 9 in patients with Guillain-Barré syndrome.
P2860
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P2860
TLR2 synergizes with both TLR4 and TLR9 for induction of the MyD88-dependent splenic cytokine and chemokine response to Streptococcus pneumoniae.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on February 2007
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
TLR2 synergizes with both TLR4 ...... e to Streptococcus pneumoniae.
@en
TLR2 synergizes with both TLR4 ...... e to Streptococcus pneumoniae.
@nl
type
label
TLR2 synergizes with both TLR4 ...... e to Streptococcus pneumoniae.
@en
TLR2 synergizes with both TLR4 ...... e to Streptococcus pneumoniae.
@nl
prefLabel
TLR2 synergizes with both TLR4 ...... e to Streptococcus pneumoniae.
@en
TLR2 synergizes with both TLR4 ...... e to Streptococcus pneumoniae.
@nl
P2093
P2860
P1433
P1476
TLR2 synergizes with both TLR4 ...... e to Streptococcus pneumoniae.
@en
P2093
Charles A Scanga
Clifford M Snapper
Eric M Bachelder
Katherine S Lee
Quanyi Chen
P2860
P304
P356
10.1016/J.CELLIMM.2007.04.003
P577
2007-02-01T00:00:00Z