Neonatal intermittent hypoxia leads to long-lasting facilitation of acute hypoxia-evoked catecholamine secretion from rat chromaffin cells.
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Epigenetic Regulation of Carotid Body Oxygen Sensing: Clinical ImplicationsIntermittent hypoxic episodes in preterm infants: do they matter?Peripheral chemoreception and arterial pressure responses to intermittent hypoxiaNeuromolecular mechanisms mediating the effects of chronic intermittent hypoxia on adrenal medullaHypoxia-inducible factor 1 mediates increased expression of NADPH oxidase-2 in response to intermittent hypoxiaH2S mediates O2 sensing in the carotid body.Neonatal intermittent hypoxia impairs neuronal nicotinic receptor expression and function in adrenal chromaffin cells.NADPH oxidase 2 mediates intermittent hypoxia-induced mitochondrial complex I inhibition: relevance to blood pressure changes in ratsMechanisms of sympathetic activation and blood pressure elevation by intermittent hypoxiaIntermittent hypoxia augments acute hypoxic sensing via HIF-mediated ROSEnhanced neuropeptide Y synthesis during intermittent hypoxia in the rat adrenal medulla: role of reactive oxygen species-dependent alterations in precursor peptide processingHypoxia. 4. Hypoxia and ion channel functionHIF-1α activation by intermittent hypoxia requires NADPH oxidase stimulation by xanthine oxidaseAdenosine A₂a receptors and O₂ sensing in developmentEpigenetic regulation of hypoxic sensing disrupts cardiorespiratory homeostasis.Sympatho-adrenal activation by chronic intermittent hypoxia.Effect of postnatal intermittent hypoxia on growth and cardiovascular regulation of rat pups.Calpain activation by ROS mediates human ether-a-go-go-related gene protein degradation by intermittent hypoxia.Neuropeptide y gates a stress-induced, long-lasting plasticity in the sympathetic nervous system.Developmental programming of O(2) sensing by neonatal intermittent hypoxia via epigenetic mechanisms.Adaptive and maladaptive cardiorespiratory responses to continuous and intermittent hypoxia mediated by hypoxia-inducible factors 1 and 2.Mechanisms of intermittent hypoxia induced hypertension.Cardiorespiratory events in preterm infants: interventions and consequences.Carotid body chemoreflex: a driver of autonomic abnormalities in sleep apnoea.DNA methylation in the central and efferent limbs of the chemo reflex requires carotid body neural activity.Epigenetic regulation of redox state mediates persistent cardiorespiratory abnormalities after long-term intermittent hypoxia.Increased cardiac output contributes to the development of chronic intermittent hypoxia-induced hypertension.
P2860
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P2860
Neonatal intermittent hypoxia leads to long-lasting facilitation of acute hypoxia-evoked catecholamine secretion from rat chromaffin cells.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on April 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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name
Neonatal intermittent hypoxia ...... ion from rat chromaffin cells.
@en
Neonatal intermittent hypoxia ...... ion from rat chromaffin cells.
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type
label
Neonatal intermittent hypoxia ...... ion from rat chromaffin cells.
@en
Neonatal intermittent hypoxia ...... ion from rat chromaffin cells.
@nl
prefLabel
Neonatal intermittent hypoxia ...... ion from rat chromaffin cells.
@en
Neonatal intermittent hypoxia ...... ion from rat chromaffin cells.
@nl
P2093
P2860
P356
P1476
Neonatal intermittent hypoxia ...... ion from rat chromaffin cells.
@en
P2093
Dangjai Souvannakitti
Ganesh K Kumar
Nanduri R Prabhakar
P2860
P304
P356
10.1152/JN.00036.2009
P407
P577
2009-04-01T00:00:00Z