Cellular effector mechanisms against Plasmodium liver stages. - Wikidata - awgldk - TriplyDB

Cellular effector mechanisms against Plasmodium liver stages.

Cellular effector mechanisms against Plasmodium liver stages.

http://www.wikidata.org/entity/Q37223839

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Tissue signatures influence the activation of intrahepatic CD8(+) T cells against malaria sporozoites In vivo CD8+ T cell dynamics in the liver of Plasmodium yoelii immunized and infected mice Plasmodium cellular effector mechanisms and the hepatic microenvironment The IL-4/STAT6 signaling axis establishes a conserved microRNA signature in human and mouse macrophages regulating cell survival via miR-342-3p T cell responses to the RTS,S/AS01(E) and RTS,S/AS02(D) malaria candidate vaccines administered according to different schedules to Ghanaian children CD8+ T cells specific for a malaria cytoplasmic antigen form clusters around infected hepatocytes and are protective at the liver stage of infection.Identification and localization of minimal MHC-restricted CD8+ T cell epitopes within the Plasmodium falciparum AMA1 protein Cytotoxic markers associate with protection against malaria in human volunteers immunized with Plasmodium falciparum sporozoites.Prospects and Pitfalls of Pregnancy-Associated Malaria Vaccination Based on the Natural Immune Response to Plasmodium falciparum VAR2CSA-Expressing Parasites.Protective effect of berberine chloride on Plasmodium chabaudi-induced hepatic tissue injury in mice.CD8(+) T Cell Responses to Plasmodium and Intracellular Parasites.Recent clinical experience with vaccines using MPL- and QS-21-containing adjuvant systems.Immunobiology of Plasmodium in liver and brain.Interferon-γ, a valuable surrogate marker of Plasmodium falciparum pre-erythrocytic stages protective immunity.Testosterone persistently dysregulates hepatic expression of Tlr6 and Tlr8 induced by Plasmodium chabaudi malaria.Apicomplexa: Malaria

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Cellular effector mechanisms against Plasmodium liver stages.

http://www.wikidata.org/entity/Q37223839

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 18 July 2008

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Cellular effector mechanisms against Plasmodium liver stages.

@en

Cellular effector mechanisms against Plasmodium liver stages.

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type

label

Cellular effector mechanisms against Plasmodium liver stages.

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Cellular effector mechanisms against Plasmodium liver stages.

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prefLabel

Cellular effector mechanisms against Plasmodium liver stages.

@en

Cellular effector mechanisms against Plasmodium liver stages.

@nl

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J.1462-5822.2008.01211.X

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Cellular Microbiology

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Cellular effector mechanisms against Plasmodium liver stages.

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Elizabeth Nardin

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P2860

The site of primary T cell activation is a determinant of the balance between intrahepatic tolerance and immunity.

Plasmodium berghei-infected primary hepatocytes process and present the circumsporozoite protein to specific CD8+ T cells in vitro.

Dendritic cells acquire antigens from live cells for cross-presentation to CTL.

A totally synthetic polyoxime malaria vaccine containing Plasmodium falciparum B cell and universal T cell epitopes elicits immune responses in volunteers of diverse HLA types.

Cutting edge: the IgG response to the circumsporozoite protein is MHC class II-dependent and CD1d-independent: exploring the role of GPIs in NK T cell activation and antimalarial responses.

The complexity of protective immunity against liver-stage malaria.

Hepatocytes express abundant surface class I MHC and efficiently use transporter associated with antigen processing, tapasin, and low molecular weight polypeptide proteasome subunit components of antigen processing and presentation pathway.

Induction in humans of CD8+ and CD4+ T cell and antibody responses by sequential immunization with malaria DNA and recombinant protein.

Electron microscopic analysis of circumsporozoite protein trail formation by gliding malaria sporozoites.

Human CD4+ T cells induced by synthetic peptide malaria vaccine are comparable to cells elicited by attenuated Plasmodium falciparum sporozoites.

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