Thiazolidinediones mimic glucose starvation in facilitating Sp1 degradation through the up-regulation of beta-transducin repeat-containing protein. - Wikidata - awgldk - TriplyDB

Thiazolidinediones mimic glucose starvation in facilitating Sp1 degradation through the up-regulation of beta-transducin repeat-containing protein.

Thiazolidinediones mimic glucose starvation in facilitating Sp1 degradation through the up-regulation of beta-transducin repeat-containing protein.

http://www.wikidata.org/entity/Q37240247

about

Udp-glucose dehydrogenase as a novel field-specific candidate biomarker of prostate cancer Targeting the oncogenic E3 ligase Skp2 in prostate and breast cancer cells with a novel energy restriction-mimetic agent Energy restriction as an antitumor target of thiazolidinediones.Cullins and cancer.Prospects on strategies for therapeutically targeting oncogenic regulatory factors by small-molecule agents Energy restriction-mimetic agents induce apoptosis in prostate cancer cells in part through epigenetic activation of KLF6 tumor suppressor gene expression.Development of a novel class of glucose transporter inhibitors.AKT1 Inhibits Epithelial-to-Mesenchymal Transition in Breast Cancer through Phosphorylation-Dependent Twist1 Degradation.p53-Dependent regulation of metabolic function through transcriptional activation of pantothenate kinase-1 gene.Metformin inhibits pancreatic cancer cell and tumor growth and downregulates Sp transcription factors.Insulin-like growth factor-I receptor is suppressed through transcriptional repression and mRNA destabilization by a novel energy restriction-mimetic agent.Dimethyl Fumarate ameliorates pulmonary arterial hypertension and lung fibrosis by targeting multiple pathways Transcription factor Sp1, also known as specificity protein 1 as a therapeutic target.Sp1 and the 'hallmarks of cancer'.OSU-CG5, a novel energy restriction mimetic agent, targets human colorectal cancer cells in vitro.Gallic acid downregulates matrix metalloproteinase-2 (MMP-2) and MMP-9 in human leukemia cells with expressed Bcr/Abl.A partnership with the proteasome; the destructive nature of GSK3.Sp1 phosphorylation by ATM downregulates BER and promotes cell elimination in response to persistent DNA damage.Long non-coding RNA metastasis-associated lung adenocarcinoma transcript 1 promotes lung adenocarcinoma by directly interacting with specificity protein 1.A systems-wide screen identifies substrates of the SCF TrCP ubiquitin ligase

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Thiazolidinediones mimic glucose starvation in facilitating Sp1 degradation through the up-regulation of beta-transducin repeat-containing protein.

http://www.wikidata.org/entity/Q37240247

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on 16 April 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Thiazolidinediones mimic gluco ...... cin repeat-containing protein.

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Thiazolidinediones mimic gluco ...... cin repeat-containing protein.

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Thiazolidinediones mimic gluco ...... cin repeat-containing protein.

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Thiazolidinediones mimic gluco ...... cin repeat-containing protein.

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Thiazolidinediones mimic gluco ...... cin repeat-containing protein.

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Molecular Pharmacology

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Thiazolidinediones mimic gluco ...... cin repeat-containing protein.

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Andrew J Paterson

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Ching-Shih Chen

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Hsiao-Ching Chuang

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Hsiao-Ching Yang

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Samuel K Kulp

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Shiuh-Rong Ho

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Shuo Wei

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Wan-Chi Tsai

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P2860

Human Sug1/p45 is involved in the proteasome-dependent degradation of Sp1

Dual regulation of Snail by GSK-3beta-mediated phosphorylation in control of epithelial-mesenchymal transition

Structure of a beta-TrCP1-Skp1-beta-catenin complex: destruction motif binding and lysine specificity of the SCF(beta-TrCP1) ubiquitin ligase

Deregulated proteolysis by the F-box proteins SKP2 and beta-TrCP: tipping the scales of cancer

SCFbeta-TRCP controls oncogenic transformation and neural differentiation through REST degradation

Increased expression of AP2 and Sp1 transcription factors in human thyroid tumors: a role in NIS expression regulation?

The human F box protein beta-Trcp associates with the Cul1/Skp1 complex and regulates the stability of beta-catenin.

Regulation of the cell cycle by SCF-type ubiquitin ligases

Fibroblast growth factor-2 represses platelet-derived growth factor receptor-alpha (PDGFR-alpha) transcription via ERK1/2-dependent Sp1 phosphorylation and an atypical cis-acting element in the proximal PDGFR-alpha promoter

ERK and the F-box protein betaTRCP target STAT1 for degradation

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47-57

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scholarly article

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10.1124/MOL.109.055376

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1

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76

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2009-04-16T00:00:00Z

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24283072

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19372209

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2701453

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