Alternatively spliced mRNAs predicted to yield frame-shift proteins and stable intron 1 RNAs of the herpes simplex virus 1 regulatory gene alpha 0 accumulate in the cytoplasm of infected cells.
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Herpes simplex virus 1 regulatory protein ICP22 interacts with a new cell cycle-regulated factor and accumulates in a cell cycle-dependent fashion in infected cells.A novel cellular protein, p60, interacting with both herpes simplex virus 1 regulatory proteins ICP22 and ICP0 is modified in a cell-type-specific manner and Is recruited to the nucleus after infectionFunctional anatomy of herpes simplex virus 1 overlapping genes encoding infected-cell protein 22 and US1.5 proteinHerpes simplex virus IE63 (ICP27) protein interacts with spliceosome-associated protein 145 and inhibits splicing prior to the first catalytic stepThe degradation of promyelocytic leukemia and Sp100 proteins by herpes simplex virus 1 is mediated by the ubiquitin-conjugating enzyme UbcH5aThe molecular basis of herpes simplex virus latencyPersistence and expression of the herpes simplex virus genome in the absence of immediate-early proteins.Processing of alpha-globin and ICP0 mRNA in cells infected with herpes simplex virus type 1 ICP27 mutants.Colocalization of the herpes simplex virus 1 UL4 protein with infected cell protein 22 in small, dense nuclear structures formed prior to onset of DNA synthesisICP22 and the UL13 protein kinase are both required for herpes simplex virus-induced modification of the large subunit of RNA polymerase II.Numerous conserved and divergent microRNAs expressed by herpes simplex viruses 1 and 2.Herpes simplex virus 1 ICP22 regulates the accumulation of a shorter mRNA and of a truncated US3 protein kinase that exhibits altered functionsHSV-1-based vectors for gene therapy of neurological diseases and brain tumors: part I. HSV-1 structure, replication and pathogenesis.Neither LAT nor open reading frame P mutations increase expression of spliced or intron-containing ICP0 transcripts in mouse ganglia latently infected with herpes simplex virusAn early regulatory function required in a cell type-dependent manner is expressed by the genomic but not the cDNA copy of the herpes simplex virus 1 gene encoding infected cell protein 0.Detecting selection in noncoding regions of nucleotide sequences.Prediction and identification of herpes simplex virus 1-encoded microRNAs.Role of ICP0 in the strategy of conquest of the host cell by herpes simplex virus 1Selection of a nonconsensus branch point is influenced by an RNA stem-loop structure and is important to confer stability to the herpes simplex virus 2-kilobase latency-associated transcriptAnalysis of human alphaherpesvirus microRNA expression in latently infected human trigeminal ganglia.During its nuclear phase the multifunctional regulatory protein ICP0 undergoes proteolytic cleavage characteristic of polyproteins.Herpes simplex virus type 1 ICP0 protein does not accumulate in the nucleus of primary neurons in cultureICP0 gene expression is a herpes simplex virus type 1 apoptotic triggerTranscriptional regulation of the Herpes Simplex Virus 1alpha-gene by the viral immediate-early protein ICP22 in association with VP16.Molecular aspects of herpes simplex virus I latency, reactivation, and recurrence.
P2860
Q22003919-D995A11D-2BF2-4BB4-8458-A92D999E7BE2Q22009164-FA76257E-72FE-4413-88FC-1450E3E8A4DEQ24517275-ECC56565-08C6-4EAD-B220-8CE7D51C508CQ24529096-4F743375-E837-4C71-896B-83CB4B3F1727Q24678629-2DCB1D0F-92FB-4A49-9BC8-FD83D60180ACQ27001272-D13DE81B-C703-4890-AF14-80B993C67AA7Q33783411-75869353-9778-459B-BC71-128374DDB15DQ33809422-FF93C09D-FE89-4375-A8DA-DBC5841C925DQ33814451-CE035F30-C25C-46A2-A384-5C71445C7EE9Q33815308-4AB722E5-973E-4BD0-BA36-9A43B6952DC0Q33826615-0ACEE3AA-808E-4185-9E71-7A6127707018Q33843248-55884342-E736-47D7-94F4-F1EFC75BBE4FQ33995747-19037538-F264-41D1-83C8-81E191774B13Q34338858-48AFC66F-5F5B-4373-AF14-5E9DFD926F31Q34346572-9C96408E-92C8-4751-89A1-910B3068C740Q34644986-5B9C30F8-60A0-43FE-B9D1-5B3378790822Q34648394-30C5B532-971E-416B-82A1-6BADFD02BA21Q35667107-014F073D-9167-4C31-A0BC-7F875B5ACCD3Q35889694-BD71FEF4-BAB1-40F4-B4B2-CF4B9938F13CQ37365331-A2F42502-DEC0-4DF6-839D-5C465211228CQ37419216-F979F642-6703-4E37-BD6B-45D00F8CC3EEQ39539586-0AE7D267-F09F-49EB-8A30-ABCC010DC597Q41495440-0158E3E0-C3B6-4DAD-A3F1-E2ADEDE2DE27Q44561051-8874DEDC-DDDC-4E7D-8A3D-D3AE270E7ECAQ45752692-A5AF8FCB-7C06-4F59-8CC9-0E90E4EA6DA2
P2860
Alternatively spliced mRNAs predicted to yield frame-shift proteins and stable intron 1 RNAs of the herpes simplex virus 1 regulatory gene alpha 0 accumulate in the cytoplasm of infected cells.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on October 1996
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Alternatively spliced mRNAs pr ...... e cytoplasm of infected cells.
@en
Alternatively spliced mRNAs pr ...... e cytoplasm of infected cells.
@nl
type
label
Alternatively spliced mRNAs pr ...... e cytoplasm of infected cells.
@en
Alternatively spliced mRNAs pr ...... e cytoplasm of infected cells.
@nl
prefLabel
Alternatively spliced mRNAs pr ...... e cytoplasm of infected cells.
@en
Alternatively spliced mRNAs pr ...... e cytoplasm of infected cells.
@nl
P2860
P356
P1476
Alternatively spliced mRNAs pr ...... e cytoplasm of infected cells.
@en
P2093
K L Carter
P2860
P304
12535-12540
P356
10.1073/PNAS.93.22.12535
P407
P577
1996-10-01T00:00:00Z