A comparison of model-based (2D) and design-based (3D) stereological methods for estimating cell number in the substantia nigra pars compacta (SNpc) of the C57BL/6J mouse
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Highly pathogenic H5N1 influenza virus can enter the central nervous system and induce neuroinflammation and neurodegenerationEstrogen ameliorates microglial activation by inhibiting the Kir2.1 inward-rectifier K(+) channel.Loss of GABAergic cortical neurons underlies the neuropathology of Lafora diseaseResistance to MPTP-neurotoxicity in α-synuclein knockout mice is complemented by human α-synuclein and associated with increased β-synuclein and Akt activationPale body-like inclusion formation and neurodegeneration following depletion of 26S proteasomes in mouse brain neurones are independent of α-synucleinInduction of microglia activation after infection with the non-neurotropic A/CA/04/2009 H1N1 influenza virus.Calibration of the stereological estimation of the number of myelinated axons in the rat sciatic nerve: a multicenter study.Pathway-specific dopaminergic deficits in a mouse model of Angelman syndromeComparison of melanin-concentrating hormone and hypocretin/orexin peptide expression patterns in a current parceling scheme of the lateral hypothalamic zone.The neurotoxicity of DOPAL: behavioral and stereological evidence for its role in Parkinson disease pathogenesis.Exercise protects against MPTP-induced neurotoxicity in mice.CART peptide is a potential endogenous antioxidant and preferentially localized in mitochondria.Genetic dissection of strain dependent paraquat-induced neurodegeneration in the substantia nigra pars compacta.Methylphenidate exposure induces dopamine neuron loss and activation of microglia in the basal ganglia of mice.Exercise does not protect against MPTP-induced neurotoxicity in BDNF haploinsufficient mice.6-OHDA generated ROS induces DNA damage and p53- and PUMA-dependent cell deathNovel para-phenyl substituted diindolylmethanes protect against MPTP neurotoxicity and suppress glial activation in a mouse model of Parkinson's disease.Changes in sensitivity of reward and motor behavior to dopaminergic, glutamatergic, and cholinergic drugs in a mouse model of fragile X syndromeAn animal model mimicking pedunculopontine nucleus cholinergic degeneration in Parkinson's disease.Endocannabinoid hydrolysis generates brain prostaglandins that promote neuroinflammation.Altered dopamine metabolism and increased vulnerability to MPTP in mice with partial deficiency of mitochondrial complex I in dopamine neurons.Inflammatory effects of highly pathogenic H5N1 influenza virus infection in the CNS of mice.HIF1α is necessary for exercise-induced neuroprotection while HIF2α is needed for dopaminergic neuron survival in the substantia nigra pars compacta.Assessment of the Effects of MPTP and Paraquat on Dopaminergic Neurons and Microglia in the Substantia Nigra Pars Compacta of C57BL/6 Mice.Executive function deficits and glutamatergic protein alterations in a progressive 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine mouse model of Parkinson's disease.Sex-specific disruption of murine midbrain astrocytic and dopaminergic developmental trajectories following antenatal GC treatment.A novel phenotype for the dynein heavy chain mutation Loa: altered dendritic morphology, organelle density, and reduced numbers of trigeminal motoneurons.Neuroprotective Properties of a Novel Non-Thiazoledinedione Partial PPAR- γ Agonist against MPTP.Glial cell line-derived neurotrophic factor partially ameliorates motor symptoms without slowing neurodegeneration in mice with respiratory chain-deficient dopamine neurons.Methamphetamine causes degeneration of dopamine cell bodies and terminals of the nigrostriatal pathway evidenced by silver staining.Confocal stereology: an efficient tool for measurement of microscopic structures.Alternative microglial activation is associated with cessation of progressive dopamine neuron loss in mice systemically administered lipopolysaccharide.Admixing of MPTP-Resistant and Susceptible Mice Strains Augments Nigrostriatal Neuronal Correlates to Resist MPTP-Induced Neurodegeneration.Synergistic effects of influenza and 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) can be eliminated by the use of influenza therapeutics: experimental evidence for the multi-hit hypothesis.Pedunculopontine cell loss and protein aggregation direct microglia activation in parkinsonian rats.Fragment C Domain of Tetanus Toxin Mitigates Methamphetamine Neurotoxicity and Its Motor Consequences in Mice.Combining nitric oxide release with anti-inflammatory activity preserves nigrostriatal dopaminergic innervation and prevents motor impairment in a 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model of Parkinson's disease.Anatomical evidence that the uninjured adjacent L4 nerve plays a significant role in the development of peripheral neuropathic pain after L5 spinal nerve ligation in rats.PACAP27 prevents Parkinson-like neuronal loss and motor deficits but not microglia activation induced by prostaglandin J2.Neuropathological changes in the substantia nigra in schizophrenia but not depression.
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A comparison of model-based (2D) and design-based (3D) stereological methods for estimating cell number in the substantia nigra pars compacta (SNpc) of the C57BL/6J mouse
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 17 April 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
A comparison of model-based (2 ...... a (SNpc) of the C57BL/6J mouse
@en
A comparison of model-based
@nl
type
label
A comparison of model-based (2 ...... a (SNpc) of the C57BL/6J mouse
@en
A comparison of model-based
@nl
prefLabel
A comparison of model-based (2 ...... a (SNpc) of the C57BL/6J mouse
@en
A comparison of model-based
@nl
P2093
P2860
P1433
P1476
A comparison of model-based (2 ...... a (SNpc) of the C57BL/6J mouse
@en
P2093
P2860
P304
P356
10.1016/J.NEUROSCIENCE.2009.04.031
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P577
2009-04-17T00:00:00Z