Preferential use of B7.2 and not B7.1 in priming of vaccinia virus-specific CD8 T cells.
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CD8 T cells use IFN-γ to protect against the lethal effects of a respiratory poxvirus infection.The TNFR family members OX40 and CD27 link viral virulence to protective T cell vaccines in miceDifferential requirements for CD80/86-CD28 costimulation in primary and memory CD4 T cell responses to vaccinia virus.Immunodomination during peripheral vaccinia virus infection.B cell-specific expression of B7-2 is required for follicular Th cell function in response to vaccinia virus.CD4+ T cell help is dispensable for protective CD8+ T cell memory against mousepox virus following vaccinia virus immunization.CD8 T cell memory to a viral pathogen requires trans cosignaling between HVEM and BTLA.T cell receptor signaling can directly enhance the avidity of CD28 ligand binding.Targeting OX40 promotes lung-resident memory CD8 T cell populations that protect against respiratory poxvirus infectionUbiquitin-mediated regulation of CD86 protein expression by the ubiquitin ligase membrane-associated RING-CH-1 (MARCH1).Multiple layers of CD80/86-dependent costimulatory activity regulate primary, memory, and secondary lymphocytic choriomeningitis virus-specific T cell immunityTransient IL-10 receptor blockade can enhance CD8(+) T cell responses to a simian adenovirus-vectored HIV-1 conserved region immunogenCD8 T cells are essential for recovery from a respiratory vaccinia virus infection.Natural Killer Cells and Innate Interferon Gamma Participate in the Host Defense against Respiratory Vaccinia Virus Infection.Anatomically restricted synergistic antiviral activities of innate and adaptive immune cells in the skin.Viral Persistence Induces Antibody Inflation without Altering Antibody AvidityTumor necrosis factor receptor/tumor necrosis factor family members in antiviral CD8 T-cell immunity.OX40:OX40L axis: emerging targets for improving poxvirus-based CD8(+) T-cell vaccines against respiratory virusesCD8+ T Cells Orchestrate pDC-XCR1+ Dendritic Cell Spatial and Functional Cooperativity to Optimize Priming.HVEM Imprints Memory Potential on Effector CD8 T Cells Required for Protective Mucosal Immunity.Inflammatory monocytes contribute to the persistence of CXCR3hi CX3CR1lo circulating and lung-resident memory CD8+ T cells following respiratory virus infection.
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P2860
Preferential use of B7.2 and not B7.1 in priming of vaccinia virus-specific CD8 T cells.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on March 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Preferential use of B7.2 and not B7.1 in priming of vaccinia virus-specific CD8 T cells.
@en
Preferential use of B7.2 and not B7.1 in priming of vaccinia virus-specific CD8 T cells.
@nl
type
label
Preferential use of B7.2 and not B7.1 in priming of vaccinia virus-specific CD8 T cells.
@en
Preferential use of B7.2 and not B7.1 in priming of vaccinia virus-specific CD8 T cells.
@nl
prefLabel
Preferential use of B7.2 and not B7.1 in priming of vaccinia virus-specific CD8 T cells.
@en
Preferential use of B7.2 and not B7.1 in priming of vaccinia virus-specific CD8 T cells.
@nl
P2093
P2860
P356
P1476
Preferential use of B7.2 and not B7.1 in priming of vaccinia virus-specific CD8 T cells.
@en
P2093
Michael Croft
Rachel Flynn
Ramon Arens
Shahram Salek-Ardakani
P2860
P304
P356
10.4049/JIMMUNOL.0803545
P407
P577
2009-03-01T00:00:00Z