The capsule-encoding viaB locus reduces intestinal inflammation by a Salmonella pathogenicity island 1-independent mechanism
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Typhoid fever: "you can't hit what you can't see"The Vi capsular polysaccharide enables Salmonella enterica serovar typhi to evade microbe-guided neutrophil chemotaxisIntraspecies variation in the emergence of hyperinfectious bacterial strains in natureVery long O-antigen chains enhance fitness during Salmonella-induced colitis by increasing bile resistanceSalmonella enterica Serovar Typhi conceals the invasion-associated type three secretion system from the innate immune system by gene regulation.Salmonella enterica serovar Typhi impairs CD4 T cell responses by reducing antigen availabilityA rapid change in virulence gene expression during the transition from the intestinal lumen into tissue promotes systemic dissemination of Salmonella.Taming the elephant: Salmonella biology, pathogenesis, and prevention.The Vi capsular polysaccharide prevents complement receptor 3-mediated clearance of Salmonella enterica serotype Typhi.Reconstructing pathogen evolution from the ruinsComparative growth analysis of capsulated (Vi+) and acapsulated (Vi-) Salmonella typhi isolates in human bloodLoss of Multicellular Behavior in Epidemic African Nontyphoidal Salmonella enterica Serovar Typhimurium ST313 Strain D23580.Loss of very-long O-antigen chains optimizes capsule-mediated immune evasion by Salmonella enterica serovar Typhi.Host specificity of bacterial pathogens.Salmonella Extracellular Matrix Components Influence Biofilm Formation and Gallbladder Colonization.Salmonellae interactions with host processes.Comparative analysis of Salmonella genomes identifies a metabolic network for escalating growth in the inflamed gut.How to become a top model: impact of animal experimentation on human Salmonella disease research.New Insights into the Roles of Long Polar Fimbriae and Stg Fimbriae in Salmonella Interactions with Enterocytes and M Cells.The TviA auxiliary protein renders the Salmonella enterica serotype Typhi RcsB regulon responsive to changes in osmolarityVi capsular polysaccharide: Synthesis, virulence, and application.Cathelicidin antimicrobial peptide expression is not induced or required for bacterial clearance during salmonella enterica infection of human monocyte-derived macrophages.Salmonella type III effector SpvC, a phosphothreonine lyase, contributes to reduction in inflammatory response during intestinal phase of infection.Foodborne Pathogens and Host Predilection
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P2860
The capsule-encoding viaB locus reduces intestinal inflammation by a Salmonella pathogenicity island 1-independent mechanism
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 18 May 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The capsule-encoding viaB locu ...... island 1-independent mechanism
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The capsule-encoding viaB locu ...... sland 1-independent mechanism.
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type
label
The capsule-encoding viaB locu ...... island 1-independent mechanism
@en
The capsule-encoding viaB locu ...... sland 1-independent mechanism.
@nl
prefLabel
The capsule-encoding viaB locu ...... island 1-independent mechanism
@en
The capsule-encoding viaB locu ...... sland 1-independent mechanism.
@nl
P2093
P2860
P356
P1476
The capsule-encoding viaB locu ...... island 1-independent mechanism
@en
P2093
Brian P Butler
Cagla Tükel
Ivan Godinez
Maria G Winter
R Paul Wilson
Sebastian E Winter
Takeshi Haneda
P2860
P304
P356
10.1128/IAI.00172-09
P407
P577
2009-05-18T00:00:00Z