Enhanced glucose tolerance by SK4 channel inhibition in pancreatic beta-cells. - Wikidata - awgldk - TriplyDB

Enhanced glucose tolerance by SK4 channel inhibition in pancreatic beta-cells.

Enhanced glucose tolerance by SK4 channel inhibition in pancreatic beta-cells.

http://www.wikidata.org/entity/Q37267135

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Mathematical modeling of heterogeneous electrophysiological responses in human β-cells Ionic mechanisms and Ca2+ dynamics underlying the glucose response of pancreatic β cells: a simulation study.Therapeutic potential of KCa3.1 blockers: recent advances and promising trends.Bile acids acutely stimulate insulin secretion of mouse β-cells via farnesoid X receptor activation and K(ATP) channel inhibition Bursting and calcium oscillations in pancreatic beta-cells: specific pacemakers for specific mechanisms.Type 2 Diabetes-Associated K+ Channel TALK-1 Modulates β-Cell Electrical Excitability, Second-Phase Insulin Secretion, and Glucose Homeostasis Modeling of glucose-induced cAMP oscillations in pancreatic β cells: cAMP rocks when metabolism rolls Trafficking of intermediate (KCa3.1) and small (KCa2.x) conductance, Ca(2+)-activated K(+) channels: a novel target for medicinal chemistry efforts?Uncoupling of Cav1.2 from Ca(2+)-induced Ca(2+) release and SK channel regulation in pancreatic β-cells.BK channels affect glucose homeostasis and cell viability of murine pancreatic beta cells.Role of K(ATP) channels in β-cell resistance to oxidative stress.Anoctamin 1 (Ano1) is required for glucose-induced membrane potential oscillations and insulin secretion by murine β-cells.The triggering pathway to insulin secretion: Functional similarities and differences between the human and the mouse β cells and their translational relevance.Mathematical modeling demonstrates how multiple slow processes can provide adjustable control of islet bursting.Ca2+ activated K+ channel Kca3.1 as a determinant of gastric acid secretion.TALK-1 channels control β cell endoplasmic reticulum Ca2+ homeostasis.A TRPM4 Inhibitor 9-Phenanthrol Inhibits Glucose- and Glucagon-Like Peptide 1-Induced Insulin Secretion from Rat Islets of Langerhans.Pancreatic β-Cell Electrical Activity and Insulin Secretion: Of Mice and Men.Pharmacological approach to understanding the control of insulin secretion in human islets.Cytokine-mediated changes in K+ channel activity promotes an adaptive Ca2+ response that sustains β-cell insulin secretion during inflammation.Regulation of KATP Channel Trafficking in Pancreatic β Cells by Protein Histidine Phosphorylation.

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Enhanced glucose tolerance by SK4 channel inhibition in pancreatic beta-cells.

http://www.wikidata.org/entity/Q37267135

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 28 April 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Enhanced glucose tolerance by SK4 channel inhibition in pancreatic beta-cells.

@en

Enhanced glucose tolerance by SK4 channel inhibition in pancreatic beta-cells.

@nl

type

label

Enhanced glucose tolerance by SK4 channel inhibition in pancreatic beta-cells.

@en

Enhanced glucose tolerance by SK4 channel inhibition in pancreatic beta-cells.

@nl

prefLabel

Enhanced glucose tolerance by SK4 channel inhibition in pancreatic beta-cells.

@en

Enhanced glucose tolerance by SK4 channel inhibition in pancreatic beta-cells.

@nl

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Enhanced glucose tolerance by SK4 channel inhibition in pancreatic beta-cells.

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P2093

Belinda Gier

http://www.w3.org/2001/XMLSchema#string

Daniela Wolpers

http://www.w3.org/2001/XMLSchema#string

Gisela Drews

http://www.w3.org/2001/XMLSchema#string

Martina Düfer

http://www.w3.org/2001/XMLSchema#string

Peter Krippeit-Drews

http://www.w3.org/2001/XMLSchema#string

Peter Ruth

http://www.w3.org/2001/XMLSchema#string

P2860

Delayed rectifying and calcium-activated K+ channels and their significance for action potential repolarization in mouse pancreatic beta-cells

A human intermediate conductance calcium-activated potassium channel

Design of a potent and selective inhibitor of the intermediate-conductance Ca2+-activated K+ channel, IKCa1: a potential immunosuppressant

The hSK4 (KCNN4) isoform is the Ca2+-activated K+ channel (Gardos channel) in human red blood cells

A new generation of Ca2+ indicators with greatly improved fluorescence properties

Expression of voltage-gated potassium channels in human and rhesus pancreatic islets.

Action potentials and insulin secretion: new insights into the role of Kv channels.

Electrophysiology of the pancreatic beta-cell

GUIDE study: double-blind comparison of once-daily gliclazide MR and glimepiride in type 2 diabetic patients

International Union of Pharmacology. LII. Nomenclature and molecular relationships of calcium-activated potassium channels

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1835-1843

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scholarly article

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10.2337/DB08-1324

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8

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19401418

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