Spontaneous relapsing-remitting EAE in the SJL/J mouse: MOG-reactive transgenic T cells recruit endogenous MOG-specific B cells. - Wikidata - awgldk - TriplyDB

Spontaneous relapsing-remitting EAE in the SJL/J mouse: MOG-reactive transgenic T cells recruit endogenous MOG-specific B cells.

Spontaneous relapsing-remitting EAE in the SJL/J mouse: MOG-reactive transgenic T cells recruit endogenous MOG-specific B cells.

http://www.wikidata.org/entity/Q37273211

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Oligodendrogenesis in the normal and pathological central nervous system B Cells in the Multiple Sclerosis Central Nervous System: Trafficking and Contribution to CNS-Compartmentalized Inflammation Novel Insights and Therapeutics in Multiple Sclerosis Selected Aspects in the Pathogenesis of Autoimmune Diseases Immunology of neuromyelitis optica: a T cell-B cell collaboration Functional and pathogenic differences of Th1 and Th17 cells in experimental autoimmune encephalomyelitis Selective accumulation of pro-inflammatory T cells in the intestine contributes to the resistance to autoimmune demyelinating disease The gut-brain connection: triggering of brain autoimmune disease by commensal gut bacteria Autoimmune encephalitis in humans: how closely does it reflect multiple sclerosis ?Characterization of dysferlin deficient SJL/J mice to assess preclinical drug efficacy: fasudil exacerbates muscle disease phenotype Treatment of autoimmune inflammation by a TLR7 ligand regulating the innate immune system Models of autoimmune demyelination in the central nervous system: on the way to translational medicine.Relapsing-remitting central nervous system autoimmunity mediated by GFAP-specific CD8 T cells.Myelin Oligodendrocyte Glycoprotein: Deciphering a Target in Inflammatory Demyelinating Diseases.Conventional housing conditions attenuate the development of experimental autoimmune encephalomyelitis.Dysregulation of lysophosphatidic acids in multiple sclerosis and autoimmune encephalomyelitis.Functional characterization of aquaporin-4 specific T cells: towards a model for neuromyelitis optica Recombinant TCR ligand reverses clinical signs and CNS damage of EAE induced by recombinant human MOG Cytokine-regulated neutrophil recruitment is required for brain but not spinal cord inflammation during experimental autoimmune encephalomyelitis.Experimental autoimmune encephalomyelitis as a testing paradigm for adjuvants and vaccines Real-time in vivo analysis of T cell activation in the central nervous system using a genetically encoded calcium indicator.DQB1*06:02-Associated Pathogenic Anti-Myelin Autoimmunity in Multiple Sclerosis-Like Disease: Potential Function of DQB1*06:02 as a Disease-Predisposing Allele Antibodies to MOG have a demyelination phenotype and affect oligodendrocyte cytoskeleton.Passive experimental autoimmune encephalomyelitis in C57BL/6 with MOG: evidence of involvement of B cells.Immune tolerance in multiple sclerosis.Characterization of antigen-specific B cells using nominal antigen-coated flow-beads.NFATc1 affects mouse splenic B cell function by controlling the calcineurin--NFAT signaling network.Substrain differences reveal novel disease-modifying gene candidates that alter the clinical course of a rodent model of multiple sclerosis.Axonal pathology and demyelination in viral models of multiple sclerosis.Brain-reactive antibodies and disease.Experimental Autoimmune Encephalomyelitis in Mice B cell antigen presentation is sufficient to drive neuroinflammation in an animal model of multiple sclerosis.IFN-gamma signaling in the central nervous system controls the course of experimental autoimmune encephalomyelitis independently of the localization and composition of inflammatory foci.TNFR2 Deficiency Acts in Concert with Gut Microbiota To Precipitate Spontaneous Sex-Biased Central Nervous System Demyelinating Autoimmune Disease.Time-Dependent Progression of Demyelination and Axonal Pathology in MP4-Induced Experimental Autoimmune Encephalomyelitis.IL-10 producing B cells partially restore E2-mediated protection against EAE in PD-L1 deficient mice.Glycoproteins as targets of autoantibodies in CNS inflammation: MOG and more.B-cell activation influences T-cell polarization and outcome of anti-CD20 B-cell depletion in central nervous system autoimmunity Platelets contribute to the pathogenesis of experimental autoimmune encephalomyelitis.Meningeal Infiltration of the Spinal Cord by Non-Classically Activated B Cells is Associated with Chronic Disease Course in a Spontaneous B Cell-Dependent Model of CNS Autoimmune Disease

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Spontaneous relapsing-remitting EAE in the SJL/J mouse: MOG-reactive transgenic T cells recruit endogenous MOG-specific B cells.

http://www.wikidata.org/entity/Q37273211

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on June 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Spontaneous relapsing-remittin ...... dogenous MOG-specific B cells.

@en

Spontaneous relapsing-remittin ...... dogenous MOG-specific B cells.

@nl

type

label

Spontaneous relapsing-remittin ...... dogenous MOG-specific B cells.

@en

Spontaneous relapsing-remittin ...... dogenous MOG-specific B cells.

@nl

prefLabel

Spontaneous relapsing-remittin ...... dogenous MOG-specific B cells.

@en

Spontaneous relapsing-remittin ...... dogenous MOG-specific B cells.

@nl

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Journal of Experimental Medicine

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Spontaneous relapsing-remittin ...... ndogenous MOG-specific B cells

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Andreas Holz

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Bernadette Pöllinger

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Florian C Kurschus

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Hartmut Wekerle

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Kerstin Berer

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Michael R Bösl

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Robert Dunn

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Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

Control of regulatory T cell development by the transcription factor Foxp3

Multiple sclerosis in the Japanese population.

Molecular characterization of antibody specificities against myelin/oligodendrocyte glycoprotein in autoimmune demyelination

T cell tolerance to Mlsa encoded antigens in T cell receptor V beta 8.1 chain transgenic mice.

B cells regulate autoimmunity by provision of IL-10.

An overview of real-time quantitative PCR: applications to quantify cytokine gene expression.

Anti-IL-23 therapy inhibits multiple inflammatory pathways and ameliorates autoimmune encephalomyelitis

Regulatory B cells inhibit EAE initiation in mice while other B cells promote disease progression.

Spontaneous opticospinal encephalomyelitis in a double-transgenic mouse model of autoimmune T cell/B cell cooperation.

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1303-1316

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scholarly article

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10.1084/JEM.20090299

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6

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206

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Helena S. Domingues

Gurumoorthy Krishnamoorthy

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2009-06-01T00:00:00Z

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26258191

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19487416

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2715069

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