Reovirus oncolysis: the Ras/RalGEF/p38 pathway dictates host cell permissiveness to reovirus infection - Wikidata - awgldk - TriplyDB

Reovirus oncolysis: the Ras/RalGEF/p38 pathway dictates host cell permissiveness to reovirus infection

Reovirus oncolysis: the Ras/RalGEF/p38 pathway dictates host cell permissiveness to reovirus infection

http://www.wikidata.org/entity/Q37285576

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Synergistic antitumor activity of oncolytic reovirus and chemotherapeutic agents in non-small cell lung cancer cells Oncolytic virus therapy: A new era of cancer treatment at dawn Clinical development of reovirus for cancer therapy: An oncolytic virus with immune-mediated antitumor activity Exploring Reovirus Plasticity for Improving Its Use as Oncolytic Virus Diminished reovirus capsid stability alters disease pathogenesis and littermate transmission Oncolytic viruses against cancer stem cells: A promising approach for gastrointestinal cancer Modification of mammalian reoviruses for use as oncolytic agents A phase I trial of single-agent reolysin in patients with relapsed multiple myeloma Permissiveness of human cancer cells to oncolytic bovine herpesvirus 1 is mediated in part by KRAS activity Oncolytic virotherapy: molecular targets in tumor-selective replication and carrier cell-mediated delivery of oncolytic viruses Reovirus exerts potent oncolytic effects in head and neck cancer cell lines that are independent of signalling in the EGFR pathway.Antiviral potential of ERK/MAPK and PI3K/AKT/mTOR signaling modulation for Middle East respiratory syndrome coronavirus infection as identified by temporal kinome analysis Isolation of reovirus T3D mutants capable of infecting human tumor cells independent of junction adhesion molecule-A.Attenuated reovirus displays oncolysis with reduced host toxicity Reovirus-associated reduction of microRNA-let-7d is related to the increased apoptotic death of cancer cells in clinical samples Oncolytic reovirus in canine mast cell tumor.Effect of human activated NRAS on replication of delNS1 H5N1 influenza virus in MDCK cells.Oncolytic bluetongue viruses: promise, progress, and perspectives Glycan engagement dictates hydrocephalus induction by serotype 1 reovirus.Myxoma virus is a novel oncolytic virus with significant antitumor activity against experimental human gliomas.Oncolytic virotherapy synergism with signaling inhibitors: Rapamycin increases myxoma virus tropism for human tumor cells.Unlocking the promise of oncolytic virotherapy in glioma: combination with chemotherapy to enhance efficacy Mechanisms of reovirus-induced cell death and tissue injury: role of apoptosis and virus-induced perturbation of host-cell signaling and transcription factor activation.PUMA and NF-kB Are Cell Signaling Predictors of Reovirus Oncolysis of Breast Cancer.Unshackling the links between reovirus oncolysis, Ras signaling, translational control and cancer.Oncolytic adenoviruses - selective retargeting to tumor cells.Reovirus receptors, cell entry, and proapoptotic signaling.Vesicular stomatitis virus oncolysis of T lymphocytes requires cell cycle entry and translation initiation.The sweet spot: defining virus-sialic acid interactions.Synergistic cytotoxicity of oncolytic reovirus in combination with cisplatin-paclitaxel doublet chemotherapy.Reolysin is a novel reovirus-based agent that induces endoplasmic reticular stress-mediated apoptosis in pancreatic cancer.Coronavirus genetically redirected to the epidermal growth factor receptor exhibits effective antitumor activity against a malignant glioblastoma Intravenous administration of Reolysin, a live replication competent RNA virus is safe in patients with advanced solid tumors.Reovirus-based therapy for cancer.Progress of oncolytic viruses in sarcomas.Approaches to Ras signaling modulation and treatment of Ras-dependent disorders: a patent review (2007--present).Evolution of oncolytic viruses: novel strategies for cancer treatment.Oncolytic viral therapy: targeting cancer stem cells Activated ras signaling pathways and reovirus oncolysis: an update on the mechanism of preferential reovirus replication in cancer cells.Virotherapy in cancer

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Reovirus oncolysis: the Ras/RalGEF/p38 pathway dictates host cell permissiveness to reovirus infection

http://www.wikidata.org/entity/Q37285576

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 19 July 2004

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Reovirus oncolysis: the Ras/Ra ...... siveness to reovirus infection

@en

Reovirus oncolysis: the Ras/Ra ...... iveness to reovirus infection.

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type

label

Reovirus oncolysis: the Ras/Ra ...... siveness to reovirus infection

@en

Reovirus oncolysis: the Ras/Ra ...... iveness to reovirus infection.

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prefLabel

Reovirus oncolysis: the Ras/Ra ...... siveness to reovirus infection

@en

Reovirus oncolysis: the Ras/Ra ...... iveness to reovirus infection.

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Proceedings of the National Academy of Sciences of the United States of America

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Reovirus oncolysis: the Ras/Ra ...... siveness to reovirus infection

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An-Dao Yang

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Kara L Norman

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Kensuke Hirasawa

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Michael A Shields

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Patrick W K Lee

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P2860

The Ras-RasGAP complex: structural basis for GTPase activation and its loss in oncogenic Ras mutants

Mitogen-activated protein kinases activate the serine/threonine kinases Mnk1 and Mnk2

The molecular basis of viral oncolysis: usurpation of the Ras signaling pathway by reovirus

Elevated phospholipase D activity in H-Ras- but not K-Ras-transformed cells by the synergistic action of RalA and ARF6

Differential sensitivity of normal and transformed human cells to reovirus infection

Cleavage of structural proteins during the assembly of the head of bacteriophage T4

Three-dimensional structures of H-ras p21 mutants: molecular basis for their inability to function as signal switch molecules

Amplification of PPM1D in human tumors abrogates p53 tumor-suppressor activity

Stimulation of gene induction and cell growth by the Ras effector Rlf

Involvement of multiple signaling pathways in follicular lymphoma transformation: p38-mitogen-activated protein kinase as a target for therapy

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