Role of endogenous TRPV1 agonists in a postburn pain model of partial-thickness injury. - Wikidata - awgldk - TriplyDB

Role of endogenous TRPV1 agonists in a postburn pain model of partial-thickness injury.

Role of endogenous TRPV1 agonists in a postburn pain model of partial-thickness injury.

http://www.wikidata.org/entity/Q37290034

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Synthesis of lipid mediators during UVB-induced inflammatory hyperalgesia in rats and mice Anatomical and physiological factors contributing to chronic muscle pain Obesity superimposed on aging magnifies inflammation and delays the resolving response after myocardial infarction Serum levels of oxylipins in achilles tendinopathy: an exploratory study.Persistent Nociception Triggered by Nerve Growth Factor (NGF) Is Mediated by TRPV1 and Oxidative Mechanisms.Association between plasma concentrations of linoleic acid-derived oxylipins and the perceived pain scores in an exploratory study in women with chronic neck pain.Molecular and cellular mechanisms that initiate pain and itch.Dietary linoleic acid-induced alterations in pro- and anti-nociceptive lipid autacoids: Implications for idiopathic pain syndromes?Transient receptor potential ion channels in primary sensory neurons as targets for novel analgesics.Role of Oxidized Lipids and TRP Channels in Orofacial Pain and Inflammation.Released lipids regulate transient receptor potential channel (TRP)-dependent oral cancer pain.Central activation of TRPV1 and TRPA1 by novel endogenous agonists contributes to mechanical allodynia and thermal hyperalgesia after burn injury.Sound-stress-induced altered nociceptive behaviors are associated with increased spinal CRFR2 gene expression in a rat model of burn injury.Burn Pain: A Systematic and Critical Review of Epidemiology, Pathophysiology, and Treatment.

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Role of endogenous TRPV1 agonists in a postburn pain model of partial-thickness injury.

http://www.wikidata.org/entity/Q37290034

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 26 July 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Role of endogenous TRPV1 agonists in a postburn pain model of partial-thickness injury.

@en

Role of endogenous TRPV1 agonists in a postburn pain model of partial-thickness injury.

@nl

type

label

Role of endogenous TRPV1 agonists in a postburn pain model of partial-thickness injury.

@en

Role of endogenous TRPV1 agonists in a postburn pain model of partial-thickness injury.

@nl

prefLabel

Role of endogenous TRPV1 agonists in a postburn pain model of partial-thickness injury.

@en

Role of endogenous TRPV1 agonists in a postburn pain model of partial-thickness injury.

@nl

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J.PAIN.2013.07.040

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Role of endogenous TRPV1 agonists in a postburn pain model of partial-thickness injury.

@en

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Dustin P Green

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Kenneth M Hargreaves

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Linda Roman

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Michael A Henry

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P2860

Activation of TRPV1 in the spinal cord by oxidized linoleic acid metabolites contributes to inflammatory hyperalgesia

Heat generates oxidized linoleic acid metabolites that activate TRPV1 and produce pain in rodents

The cytochrome P450 inhibitor, ketoconazole, inhibits oxidized linoleic acid metabolite-mediated peripheral inflammatory pain

The capsaicin receptor: a heat-activated ion channel in the pain pathway

A simple method for the isolation and purification of total lipides from animal tissues

Pharmacological blockade of the vanilloid receptor TRPV1 elicits marked hyperthermia in humans

A new and sensitive method for measuring thermal nociception in cutaneous hyperalgesia

Increased sodium channel immunofluorescence at myelinated and demyelinated sites following an inflammatory and partial axotomy lesion of the rat infraorbital nerve.

bis-Allylic hydroxylation of linoleic acid and arachidonic acid by human hepatic monooxygenases.

Burn injury triggered dysfunction in dendritic cell response to TLR9 activation and resulted in skewed T cell functions

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2512-2520

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scholarly article

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10.1016/J.PAIN.2013.07.040

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11

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Shivani B. Ruparel

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2013-07-26T00:00:00Z

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