Persistent Nociception Triggered by Nerve Growth Factor (NGF) Is Mediated by TRPV1 and Oxidative Mechanisms. - Wikidata - awgldk - TriplyDB

Persistent Nociception Triggered by Nerve Growth Factor (NGF) Is Mediated by TRPV1 and Oxidative Mechanisms.

Persistent Nociception Triggered by Nerve Growth Factor (NGF) Is Mediated by TRPV1 and Oxidative Mechanisms.

http://www.wikidata.org/entity/Q35675208

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Integrating TRPV1 Receptor Function with Capsaicin Psychophysics Nociceptive TRP Channels: Sensory Detectors and Transducers in Multiple Pain Pathologies Advanced oxidation protein products sensitized the transient receptor potential vanilloid 1 via NADPH oxidase 1 and 4 to cause mechanical hyperalgesia.Local upregulation of transient receptor potential ankyrin 1 and transient receptor potential vanilloid 1 ion channels in rectosigmoid deep infiltrating endometriosis The G2A receptor (GPR132) contributes to oxaliplatin-induced mechanical pain hypersensitivity.Inflammatory pain control by blocking oxidized phospholipid-mediated TRP channel activation.Association between plasma concentrations of linoleic acid-derived oxylipins and the perceived pain scores in an exploratory study in women with chronic neck pain.The Complement System Component C5a Produces Thermal Hyperalgesia via Macrophage-to-Nociceptor Signaling That Requires NGF and TRPV1 Dietary linoleic acid-induced alterations in pro- and anti-nociceptive lipid autacoids: Implications for idiopathic pain syndromes?EXPRESS: NGF-trkA signaling modulates the analgesic effects of prostatic acid phosphatase in resiniferatoxin-induced neuropathy.Nerve Growth Factor Regulation by TNF-α and IL-1β in Synovial Macrophages and Fibroblasts in Osteoarthritic Mice Targeting CYP2J to reduce paclitaxel-induced peripheral neuropathic pain.Mechanism and therapeutic effectiveness of nerve growth factor in osteoarthritis pain Role of Oxidized Lipids and TRP Channels in Orofacial Pain and Inflammation.Transmission pathways and mediators as the basis for clinical pharmacology of pain.Nociceptor Sensory Neuron-Immune Interactions in Pain and Inflammation.Nerve growth factor: a neuroimmune crosstalk mediator for all seasons.Is it Sjögren's syndrome or burning mouth syndrome? Distinct pathoses with similar oral symptoms.Nociceptor plasticity: A closer look.Pain in ankylosing spondylitis: a neuro-immune collaboration.Warming Up to New Possibilities with the Capsaicin Receptor TRPV1: mTOR, AMPK, and Erythropoietin.Serotonin-gated inward currents are three times more frequent in rat hairy skin sensory afferents than in those innervating the skeletal muscle.SHANK3 Deficiency Impairs Heat Hyperalgesia and TRPV1 Signaling in Primary Sensory Neurons.Neuroinflammation, Mast Cells, and Glia: Dangerous Liaisons.Mast cell-neural interactions contribute to pain and itch.A Pilot Study Investigating Clinical Responses and Biological Pathways of Azelastine/Fluticasone in Nonallergic Vasomotor Rhinitis before and after Cold Dry Air Provocation.Pain Mechanisms in Peritoneal Diseases Might Be Partially Regulated by Estrogen.Constitutive activity of transient receptor potential vanilloid type 1 triggers spontaneous firing in nerve growth factor-treated dorsal root ganglion neurons of rats

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P2860

Persistent Nociception Triggered by Nerve Growth Factor (NGF) Is Mediated by TRPV1 and Oxidative Mechanisms.

http://www.wikidata.org/entity/Q35675208

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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name

Persistent Nociception Trigger ...... RPV1 and Oxidative Mechanisms.

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Persistent Nociception Trigger ...... RPV1 and Oxidative Mechanisms.

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Persistent Nociception Trigger ...... RPV1 and Oxidative Mechanisms.

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Persistent Nociception Trigger ...... RPV1 and Oxidative Mechanisms.

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Persistent Nociception Trigger ...... RPV1 and Oxidative Mechanisms.

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Persistent Nociception Trigger ...... RPV1 and Oxidative Mechanisms.

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JNEUROSCI.3993-14.2015

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Journal of Neuroscience

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Persistent Nociception Trigger ...... RPV1 and Oxidative Mechanisms.

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P2093

Dustin P Green

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Eric R Flores

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Michael A Eskander

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Nathaniel A Jeske

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Paul B Chen

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Xiaoli Gao

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P2860

Activation of TRPV1 in the spinal cord by oxidized linoleic acid metabolites contributes to inflammatory hyperalgesia

Heat generates oxidized linoleic acid metabolites that activate TRPV1 and produce pain in rodents

The cytochrome P450 inhibitor, ketoconazole, inhibits oxidized linoleic acid metabolite-mediated peripheral inflammatory pain

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

The spinal phospholipase-cyclooxygenase-prostanoid cascade in nociceptive processing

20-Hydroxyeicosatetraenoic acid (20-HETE) is a novel activator of transient receptor potential vanilloid 1 (TRPV1) channel

TRPV1 receptors in the CNS play a key role in broad-spectrum analgesia of TRPV1 antagonists

Spinal 12-lipoxygenase-derived hepoxilin A3 contributes to inflammatory hyperalgesia via activation of TRPV1 and TRPA1 receptors

Role of cytochrome P450 enzymes in the bioactivation of polyunsaturated fatty acids

Direct activation of capsaicin receptors by products of lipoxygenases: endogenous capsaicin-like substances

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8593-8603

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scholarly article

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10.1523/JNEUROSCI.3993-14.2015

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22

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35

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Shivani B. Ruparel

Kenneth M Hargreaves

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2015-06-01T00:00:00Z

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26041925

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4452557

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