Apoptotic transition of senescent cells accompanied with mitochondrial hyper-function. - Wikidata - awgldk - TriplyDB

Apoptotic transition of senescent cells accompanied with mitochondrial hyper-function.

Apoptotic transition of senescent cells accompanied with mitochondrial hyper-function.

http://www.wikidata.org/entity/Q37317537

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Mitochondrial dysfunction and potential anticancer therapy.Polymorphism rs1385129 Within Glut1 Gene SLC2A1 Is Linked to Poor CD4+ T Cell Recovery in Antiretroviral-Treated HIV+ Individuals.TLR4-mediated activation of the ERK pathway following UVA irradiation contributes to increased cytokine and MMP expression in senescent human dermal fibroblasts

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Apoptotic transition of senescent cells accompanied with mitochondrial hyper-function.

http://www.wikidata.org/entity/Q37317537

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on April 2016

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Apoptotic transition of senescent cells accompanied with mitochondrial hyper-function.

@en

Apoptotic transition of senescent cells accompanied with mitochondrial hyper-function.

@nl

type

label

Apoptotic transition of senescent cells accompanied with mitochondrial hyper-function.

@en

Apoptotic transition of senescent cells accompanied with mitochondrial hyper-function.

@nl

prefLabel

Apoptotic transition of senescent cells accompanied with mitochondrial hyper-function.

@en

Apoptotic transition of senescent cells accompanied with mitochondrial hyper-function.

@nl

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ONCOTARGET.8536

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Apoptotic transition of senescent cells accompanied with mitochondrial hyper-function.

@en

P2093

Danli Wang

http://www.w3.org/2001/XMLSchema#string

Feiqiu Wen

http://www.w3.org/2001/XMLSchema#string

Fen Zhang

http://www.w3.org/2001/XMLSchema#string

Hong-Wei Ouyang

http://www.w3.org/2001/XMLSchema#string

Junfeng Ji

http://www.w3.org/2001/XMLSchema#string

Li Chen

http://www.w3.org/2001/XMLSchema#string

Ran Zheng

http://www.w3.org/2001/XMLSchema#string

Rui Zhang

http://www.w3.org/2001/XMLSchema#string

Weihao Sui

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Xiaowen Chen

http://www.w3.org/2001/XMLSchema#string

P2860

Loss of MARCH5 mitochondrial E3 ubiquitin ligase induces cellular senescence through dynamin-related protein 1 and mitofusin 1

Mitochondrial fission and fusion mediators, hFis1 and OPA1, modulate cellular senescence

Reciprocal regulation of p53 and malic enzymes modulates metabolism and senescence

Loss of PINK1 function promotes mitophagy through effects on oxidative stress and mitochondrial fission

The DNA damage response: putting checkpoints in perspective

Formation of elongated giant mitochondria in DFO-induced cellular senescence: involvement of enhanced fusion process through modulation of Fis1

Regulation of mitochondrial morphology through proteolytic cleavage of OPA1

The serial cultivation of human diploid cell strains

Senescent cells, tumor suppression, and organismal aging: good citizens, bad neighbors

Mitochondrial dysfunction accounts for the stochastic heterogeneity in telomere-dependent senescence.

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28286-28300

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scholarly article

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10.18632/ONCOTARGET.8536

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19

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7

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2016-04-01T00:00:00Z

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299569631

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27056883

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apoptotic process

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5053727

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