Experimental sepsis-induced mitochondrial biogenesis is dependent on autophagy, TLR4, and TLR9 signaling in liver.
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Mitochondrial Mechanisms in Septic CardiomyopathyThyroid hormone induction of mitochondrial activity is coupled to mitophagy via ROS-AMPK-ULK1 signalingStaphylococcus aureus sepsis induces early renal mitochondrial DNA repair and mitochondrial biogenesis in mice.CaMKIV-dependent preservation of mTOR expression is required for autophagy during lipopolysaccharide-induced inflammation and acute kidney injury.Importance of Toll-like receptor 2 in mitochondrial dysfunction during polymicrobial sepsisDose response of endotoxin on hepatocyte and muscle mitochondrial respiration in vitroAdenosine monophosphate-activated protein kinase activation protects against sepsis-induced organ injury and inflammationEffects of inhalation of low-dose nitrite or carbon monoxide on post-reperfusion mitochondrial function and tissue injury in hemorrhagic shock swine.MITOCHONDRIAL FUNCTION IN SEPSIS.Biology and Metabolism of Sepsis: Innate Immunity, Bioenergetics, and AutophagyAutophagosome-lysosome fusion triggers a lysosomal response mediated by TLR9 and controlled by OCRLMKK3 regulates mitochondrial biogenesis and mitophagy in sepsis-induced lung injury.Recent developments in severe sepsis research: from bench to bedside and back.Calcium/calmodulin-dependent protein kinase regulates the PINK1/Parkin and DJ-1 pathways of mitophagy during sepsis.Sepsis-induced acute kidney injury.Metabolic reprogramming and tolerance during sepsis-induced AKI.Mechanisms of bile acid mediated inflammation in the liver.Metformin improves anxiety-like behaviors through AMPK-dependent regulation of autophagy following transient forebrain ischemia.Autophagy in sepsis: Degradation into exhaustion?Ginsenoside Rg3 attenuates sepsis-induced injury and mitochondrial dysfunction in liver via AMPK-mediated autophagy flux.Redox regulation of mitophagy in the lung during murine Staphylococcus aureus sepsis.The critical role played by endotoxin-induced liver autophagy in the maintenance of lipid metabolism during sepsis.Bile acids initiate cholestatic liver injury by triggering a hepatocyte-specific inflammatory response.The Role of Autophagy in Critical Illness-induced Liver Damage.Hypoxia mediates mitochondrial biogenesis in hepatocellular carcinoma to promote tumor growth through HMGB1 and TLR9 interaction.Autophagy: A Potential Therapeutic Target for Reversing Sepsis-Induced Immunosuppression.Afzelin ameliorates D-galactosamine and lipopolysaccharide-induced fulminant hepatic failure by modulating mitochondrial quality control and dynamics.Exposure of Monocytic Cells to Lipopolysaccharide Induces Coordinated Endotoxin Tolerance, Mitochondrial Biogenesis, Mitophagy, and Antioxidant Defenses
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Experimental sepsis-induced mitochondrial biogenesis is dependent on autophagy, TLR4, and TLR9 signaling in liver.
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article científic
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article scientifique
@fr
articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 27 August 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Experimental sepsis-induced mi ...... , and TLR9 signaling in liver.
@en
Experimental sepsis-induced mi ...... , and TLR9 signaling in liver.
@nl
type
label
Experimental sepsis-induced mi ...... , and TLR9 signaling in liver.
@en
Experimental sepsis-induced mi ...... , and TLR9 signaling in liver.
@nl
prefLabel
Experimental sepsis-induced mi ...... , and TLR9 signaling in liver.
@en
Experimental sepsis-induced mi ...... , and TLR9 signaling in liver.
@nl
P2093
P2860
P921
P356
P1433
P1476
Experimental sepsis-induced mi ...... , and TLR9 signaling in liver.
@en
P2093
Brian S Zuckerbraun
Evie H Carchman
Kevin Mollen
Matthew R Rosengart
Patricia Loughran
Sean Whelan
Sladjana Stratamirovic
Sruti Shiva
P2860
P304
P356
10.1096/FJ.13-229476
P407
P577
2013-08-27T00:00:00Z