Prolonged cell cycle response of HeLa cells to low-level alkylation exposure. - Wikidata - awgldk - TriplyDB

Prolonged cell cycle response of HeLa cells to low-level alkylation exposure.

Prolonged cell cycle response of HeLa cells to low-level alkylation exposure.

http://www.wikidata.org/entity/Q37331796

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Structural, molecular and cellular functions of MSH2 and MSH6 during DNA mismatch repair, damage signaling and other noncanonical activities Nuclear reorganization of DNA mismatch repair proteins in response to DNA damage.HOXA10 is associated with temozolomide resistance through regulation of the homologous recombinant DNA repair pathway in glioblastoma cell lines.Phosphorylated hMSH6: DNA mismatch versus DNA damage recognition.Spontaneous production of immunoglobulin M in human epithelial cancer cells Major differences between tumor and normal human cell fates after exposure to chemotherapeutic monofunctional alkylator Multifaceted roles of alkyltransferase and related proteins in DNA repair, DNA damage, resistance to chemotherapy, and research tools.Complex DNA repair pathways as possible therapeutic targets to overcome temozolomide resistance in glioblastoma.TAT-CC fusion protein depresses the oncogenicity of BCR-ABL in vitro and in vivo through interrupting its oligomerization.The homologous recombination protein RAD51D mediates the processing of 6-thioguanine lesions downstream of mismatch repair.

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Prolonged cell cycle response of HeLa cells to low-level alkylation exposure.

http://www.wikidata.org/entity/Q37331796

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 28 July 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Prolonged cell cycle response of HeLa cells to low-level alkylation exposure.

@en

Prolonged cell cycle response of HeLa cells to low-level alkylation exposure.

@nl

type

label

Prolonged cell cycle response of HeLa cells to low-level alkylation exposure.

@en

Prolonged cell cycle response of HeLa cells to low-level alkylation exposure.

@nl

prefLabel

Prolonged cell cycle response of HeLa cells to low-level alkylation exposure.

@en

Prolonged cell cycle response of HeLa cells to low-level alkylation exposure.

@nl

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0008-5472.CAN-09-0899

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Cancer Research

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Prolonged cell cycle response of HeLa cells to low-level alkylation exposure.

@en

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Allen G Schroering

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Anbarasi Kothandapani

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Kandace J Williams

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Saravanan Kaliyaperumal

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Steve M Patrick

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Vishal P Sharma

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P2860

MSH2 and ATR form a signaling module and regulate two branches of the damage response to DNA methylation

ATR kinase activation mediated by MutSalpha and MutLalpha in response to cytotoxic O6-methylguanine adducts

Cell death by mitotic catastrophe: a molecular definition

DNA mismatch repair

DNA mismatch repair: functions and mechanisms

An Msh2 point mutation uncouples DNA mismatch repair and apoptosis

Dominant effects of an Msh6 missense mutation on DNA repair and cancer susceptibility

A simple technique for quantitation of low levels of DNA damage in individual cells

RPA phosphorylation in mitosis alters DNA binding and protein-protein interactions.

Mismatch repair-dependent G2 checkpoint induced by low doses of SN1 type methylating agents requires the ATR kinase.

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6307-6314

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scholarly article

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10.1158/0008-5472.CAN-09-0899

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15

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69

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2009-07-28T00:00:00Z

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19638578

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2737279

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