Linking epithelial polarity and carcinogenesis by multitasking Helicobacter pylori virulence factor CagA.
about
Function, regulation and pathological roles of the Gab/DOS docking proteinsFunctional regulation of phospholipase D expression in cancer and inflammationHelicobacter and salmonella persistent infection strategiesTransgenic expression of the Helicobacter pylori virulence factor CagA promotes apoptosis or tumorigenesis through JNK activation in DrosophilaHelicobacter pylori infection induced gastric cancer; advance in gastric stem cell research and the remaining challengesHelicobacter pylori infection, oncogenic pathways and epigenetic mechanisms in gastric carcinogenesis.A model to explain specific cellular communications and cellular harmony:- a hypothesis of coupled cells and interactive coupling molecules.Helicobacter pylori perturbs iron trafficking in the epithelium to grow on the cell surfaceNature meets nurture: molecular genetics of gastric cancer.Sequence Polymorphism and Intrinsic Structural Disorder as Related to Pathobiological Performance of the Helicobacter pylori CagA OncoproteinEpidemiological link between gastric disease and polymorphisms in VacA and CagA.Helicobacter pylori CagA triggers expression of the bactericidal lectin REG3γ via gastric STAT3 activationMicroRNAs up-regulated by CagA of Helicobacter pylori induce intestinal metaplasia of gastric epithelial cellsPolymorphisms in the intermediate region of VacA impact Helicobacter pylori-induced disease development.Mapping of p140Cap phosphorylation sites: the EPLYA and EGLYA motifs have a key role in tyrosine phosphorylation and Csk binding, and are substrates of the Abl kinase.Presence of terminal EPIYA phosphorylation motifs in Helicobacter pylori CagA contributes to IL-8 secretion, irrespective of the number of repeats.Helicobacter pylori CagA disrupts epithelial patterning by activating myosin light chain.Complex cellular responses of Helicobacter pylori-colonized gastric adenocarcinoma cellsPotentiation of Helicobacter pylori CagA protein virulence through homodimerizationDeconstructing host-pathogen interactions in Drosophila.Statins Attenuate Helicobacter pylori CagA Translocation and Reduce Incidence of Gastric Cancer: In Vitro and Population-Based Case-Control StudiesA hypothesis of couplet molecules and couplet cells in gastric function and an association with Helicobacter pylori.Pathogen-driven gastrointestinal cancers: Time for a change in treatment paradigm?Association between infection of virulence cagA gene Helicobacter pylori and laryngeal squamous cell carcinomaHelicobacter pylori CagA: From Pathogenic Mechanisms to Its Use as an Anti-Cancer Vaccine.Gastric cancer-molecular and clinical dimensions.Role of the tumor microenvironment in the pathogenesis of gastric carcinoma.The stress polarity pathway: AMPK 'GIV'-es protection against metabolic insults.Genetic predisposition to Helicobacter pylori-induced gastric precancerous conditions.Modulation of connexin signaling by bacterial pathogens and their toxins.Regulation of the actin cytoskeleton in Helicobacter pylori-induced migration and invasive growth of gastric epithelial cells.Helicobacter pylori CagA: a critical destroyer of the gastric epithelial barrier.The functional interplay of Helicobacter pylori factors with gastric epithelial cells induces a multi-step process in pathogenesis.Current perspectives toward the identification of key players in gastric cancer microRNA dysregulation.Impact of structural polymorphism for the Helicobacter pylori CagA oncoprotein on binding to polarity-regulating kinase PAR1b.Analysis of T4SS-induced signaling by H. pylori using quantitative phosphoproteomics.Helicobacter pylori CagA protein induces factors involved in the epithelial to mesenchymal transition (EMT) in infected gastric epithelial cells in an EPIYA- phosphorylation-dependent manner.Rebamipide abolishes Helicobacter pylori CagA-induced phospholipase D1 expression via inhibition of NFκB and suppresses invasion of gastric cancer cells.Outer inflammatory protein a (OipA) of Helicobacter pylori is regulated by host cell contact and mediates CagA translocation and interleukin-8 response only in the presence of a functional cag pathogenicity island type IV secretion system.Curcuma wenyujin Y. H. Chen et C. Ling n-Butyl alcohol extract inhibits AGS cell Helicobacter pylori CagA+VacA+ promoted invasiveness by down-regulating caudal type homeobox transcription factor and claudin-2 expression.
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Linking epithelial polarity and carcinogenesis by multitasking Helicobacter pylori virulence factor CagA.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Linking epithelial polarity an ...... pylori virulence factor CagA.
@en
Linking epithelial polarity an ...... pylori virulence factor CagA.
@nl
type
label
Linking epithelial polarity an ...... pylori virulence factor CagA.
@en
Linking epithelial polarity an ...... pylori virulence factor CagA.
@nl
prefLabel
Linking epithelial polarity an ...... pylori virulence factor CagA.
@en
Linking epithelial polarity an ...... pylori virulence factor CagA.
@nl
P2860
P356
P1433
P1476
Linking epithelial polarity an ...... pylori virulence factor CagA.
@en
P2093
M Hatakeyama
P2860
P2888
P304
P356
10.1038/ONC.2008.353
P407
P577
2008-11-01T00:00:00Z