Smooth muscle cell-specific Tgfbr1 deficiency promotes aortic aneurysm formation by stimulating multiple signaling events. - Wikidata - awgldk - TriplyDB

Smooth muscle cell-specific Tgfbr1 deficiency promotes aortic aneurysm formation by stimulating multiple signaling events.

Smooth muscle cell-specific Tgfbr1 deficiency promotes aortic aneurysm formation by stimulating multiple signaling events.

http://www.wikidata.org/entity/Q37339708

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An X-linked Myh11-CreERT2 mouse line resulting from Y to X chromosome-translocation of the Cre allele.Transforming Growth Factor-β1 Inhibits Pseudoaneurysm Formation After Aortic Patch Angioplasty.Further Evidence Supporting a Protective Role of Transforming Growth Factor-β (TGFβ) in Aortic Aneurysm and Dissection.Smooth Muscle Cells Derived From Second Heart Field and Cardiac Neural Crest Reside in Spatially Distinct Domains in the Media of the Ascending Aorta-Brief Report.Enlightening the Association between Bicuspid Aortic Valve and Aortopathy.Aortic Aneurysms.Heterogeneity of aortic smooth muscle cells: A determinant for regional characteristics of thoracic aortic aneurysms?

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Smooth muscle cell-specific Tgfbr1 deficiency promotes aortic aneurysm formation by stimulating multiple signaling events.

http://www.wikidata.org/entity/Q37339708

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 14 October 2016

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Smooth muscle cell-specific Tg ...... ing multiple signaling events.

@en

Smooth muscle cell-specific Tg ...... ing multiple signaling events.

@nl

type

label

Smooth muscle cell-specific Tg ...... ing multiple signaling events.

@en

Smooth muscle cell-specific Tg ...... ing multiple signaling events.

@nl

prefLabel

Smooth muscle cell-specific Tg ...... ing multiple signaling events.

@en

Smooth muscle cell-specific Tg ...... ing multiple signaling events.

@nl

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Scientific Reports

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Smooth muscle cell-specific Tg ...... ing multiple signaling events.

@en

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Bradley M Schmit

http://www.w3.org/2001/XMLSchema#string

Chunhua Fu

http://www.w3.org/2001/XMLSchema#string

Kenneth DeSart

http://www.w3.org/2001/XMLSchema#string

Pu Yang

http://www.w3.org/2001/XMLSchema#string

S Paul Oh

http://www.w3.org/2001/XMLSchema#string

Zhihua Jiang

http://www.w3.org/2001/XMLSchema#string

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Losartan, an AT1 antagonist, prevents aortic aneurysm in a mouse model of Marfan syndrome

Genetics of thoracic aortic aneurysm: at the crossroad of transforming growth factor-β signaling and vascular smooth muscle cell contractility

RDEA119/BAY 869766: a potent, selective, allosteric inhibitor of MEK1/2 for the treatment of cancer

Multicenter phase I trial of the mitogen-activated protein kinase 1/2 inhibitor BAY 86-9766 in patients with advanced cancer.

Aneurysm syndromes caused by mutations in the TGF-beta receptor

TGFβ signalling in context

Cardiovascular malformations with normal smooth muscle differentiation in neural crest-specific type II TGFbeta receptor (Tgfbr2) mutant mice

Dysregulation of TGF-beta activation contributes to pathogenesis in Marfan syndrome

TGF-beta signaling-deficient hematopoietic stem cells have normal self-renewal and regenerative ability in vivo despite increased proliferative capacity in vitro.

TGFB2 mutations cause familial thoracic aortic aneurysms and dissections associated with mild systemic features of Marfan syndrome.

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35444

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scholarly article

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10.1038/SREP35444

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6

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Scott A. Berceli

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2016-10-14T00:00:00Z

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1028107685

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27739498

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