Profiling of dynamically changed gene expression in dorsal root ganglia post peripheral nerve injury and a critical role of injury-induced glial fibrillary acidic protein in maintenance of pain behaviors [corrected].

Profiling of dynamically changed gene expression in dorsal root ganglia post peripheral nerve injury and a critical role of injury-induced glial fibrillary acidic protein in maintenance of pain behaviors [corrected].

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Evidence for a role of endocannabinoids, astrocytes and p38 phosphorylation in the resolution of postoperative pain Thrombospondin-4 contributes to spinal sensitization and neuropathic pain states Central Mechanisms Mediating Thrombospondin-4-induced Pain States.A comparison of RNA-seq and exon arrays for whole genome transcription profiling of the L5 spinal nerve transection model of neuropathic pain in the rat.Painful nerve injury upregulates thrombospondin-4 expression in dorsal root ganglia Application of pulsed radiofrequency currents to rat dorsal root ganglia modulates nerve injury-induced tactile allodynia Differential transcriptional profiling of damaged and intact adjacent dorsal root ganglia neurons in neuropathic pain.Gene expression in skin, muscle, and dorsal root ganglion after plantar incision in the rat.Plasticity of cytochrome P450 isozyme expression in rat trigeminal ganglia neurons during inflammation.Spinal 5-HT3 receptors facilitate behavioural hypersensitivity induced by elevated calcium channel alpha-2-delta-1 protein The Effect of GCSB-5 a New Herbal Medicine on Changes in Pain Behavior and Neuroglial Activation in a Rat Model of Lumbar Disc Herniation.Upregulation of nuclear factor of activated T-cells by nerve injury contributes to development of neuropathic pain.Thrombospondin-4 contributes to spinal cord injury-induced changes in nociception.Pathobiology and management of prostate cancer-induced bone pain: recent insights and future treatments.Glial activation in the collagenase model of nociception associated with osteoarthritis.Electrophysiological characterization of spinal neuron sensitization by elevated calcium channel alpha-2-delta-1 subunit protein.Targeting astrocyte signaling for chronic pain.Systems biology approaches to finding novel pain mediators.The neuroimmunology of degeneration and regeneration in the peripheral nervous system.Genetics of chronic post-surgical pain: a crucial step toward personal pain medicine.Gene expression profiles reveal key pathways and genes associated with neuropathic pain in patients with spinal cord injury.A rapamycin-sensitive signaling pathway is essential for the full expression of persistent pain states.Satellite glial cell proliferation in the trigeminal ganglia after chronic constriction injury of the infraorbital nerve.Quantification of gene expression after painful nerve injury: validation of optimal reference genes Thrombospondin-4 divergently regulates voltage-gated Ca2+ channel subtypes in sensory neurons after nerve injury Intrathecal cannabilactone CB(2)R agonist, AM1710, controls pathological pain and restores basal cytokine levels.Differential effects of voltage-gated calcium channel blockers on calcium channel alpha-2-delta-1 subunit protein-mediated nociception Glial fibrillary acidic protein promoter determines transgene expression in satellite glial cells following intraganglionic adeno-associated virus delivery in adult rats.Peripheral and central sensitization in remote spinal cord regions contribute to central neuropathic pain after spinal cord injury.Changes in the Expressions of Iba1 and Calcitonin Gene-Related Peptide in Adjacent Lumbar Spinal Segments after Lumbar Disc Herniation in a Rat Model.The α7 nicotinic receptor dual allosteric agonist and positive allosteric modulator GAT107 reverses nociception in mouse models of inflammatory and neuropathic pain.Effect of subpressor dose of angiotensin II on pain-related behavior in relation with neuronal injury and activation of satellite glial cells in the rat dorsal root ganglia.Attenuation of pain-related behaviour evoked by carrageenan injection through blockade of neuropeptide Y Y1 and Y2 receptors.Thrombospondin-4 acts on pre-synaptic calcium channel alpha-2-delta-1 subunit to induce synaptogenesis between sensory and spinal cord neurons through a T-type calcium channel dependent pathway that is blocked by early, low-dose gabapentin treatment

P2860

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P2860

Profiling of dynamically changed gene expression in dorsal root ganglia post peripheral nerve injury and a critical role of injury-induced glial fibrillary acidic protein in maintenance of pain behaviors [corrected].

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http://www.wikidata.org/entity/Q37345895

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article científic

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scientific article published on May 2009

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