Role of transiently altered sarcolemmal membrane permeability and basic fibroblast growth factor release in the hypertrophic response of adult rat ventricular myocytes to increased mechanical activity in vitro

Role of transiently altered sarcolemmal membrane permeability and basic fibroblast growth factor release in the hypertrophic response of adult rat ventricular myocytes to increased mechanical activity in vitro

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Sarcospan-deficient mice maintain normal muscle function Pathophysiological roles of FGF signaling in the heart Fibroblast growth factor receptor 1 signaling in adult cardiomyocytes increases contractility and results in a hypertrophic cardiomyopathy The fibrosis-cell death axis in heart failure Fibroblast growth factors in cardiovascular disease: The emerging role of FGF21 Study familial hypertrophic cardiomyopathy using patient-specific induced pluripotent stem cells Fibroblast growth factor-2 mediates pressure-induced hypertrophic response Overexpression of FGF-2 increases cardiac myocyte viability after injury in isolated mouse hearts.Intramyocardial fibroblast myocyte communication.Bone marrow-derived cells contribute to fibrosis in the chronically failing heart Impaired cerebral cortex development and blood pressure regulation in FGF-2-deficient mice Modulation of membrane traffic by mechanical stimuli.Fibroblast Growth Factor 2 Mediates Isoproterenol-induced Cardiac Hypertrophy through Activation of the Extracellular Regulated Kinase.Molecular pathogenesis of muscle degeneration in the delta-sarcoglycan-deficient hamster Dilated cardiomyopathy and impaired cardiac hypertrophic response to angiotensin II in mice lacking FGF-2.Animal models for muscular dystrophy show different patterns of sarcolemmal disruption.Electrical stimulation of neonatal cardiomyocytes results in the sequential activation of nuclear genes governing mitochondrial proliferation and differentiation Increased troponin levels in nonischemic cardiac conditions and noncardiac diseases.Biological functions of the low and high molecular weight protein isoforms of fibroblast growth factor-2 in cardiovascular development and disease.Measurement of circulating concentrations of cardiac troponin I and T in healthy subjects: a tool for monitoring myocardial tissue renewal?The diagnostic utility of cardiac biomarkers in dialysis patients.Regulatory RNAs and paracrine networks in the heart.Cardiac actions of fibroblast growth factor 23.Relationship between strain levels and permeability of the plasma membrane in statically stretched myoblasts.Increased c-fos mRNA expression by human fibroblasts contracting stressed collagen matrices.Cell Injury-Induced Release of Fibroblast Growth Factor 2: Relevance to Intracerebral Mesenchymal Stromal Cell Transplantations.Angiotensin II-induced cardiac hypertrophy and fibrosis are promoted in mice lacking Fgf16.Release of cardiac troponin I from viable cardiomyocytes is mediated by integrin stimulation.Integrin stimulation favors uptake of macromolecules by cardiomyocytes in vitro.FGF-2 and FGF-16 protect isolated perfused mouse hearts from acute doxorubicin-induced contractile dysfunction.Exacerbation of myocardial injury in transgenic mice overexpressing FGF-2 is T cell dependent.Protection by endogenous FGF-2 against isoproterenol-induced cardiac dysfunction is attenuated by cyclosporine A.Myocyte contractile activity modulates norepinephrine cytotoxicity and survival effects of neuregulin-1beta.Sequence analysis of the fibroblast growth factor 2 gene from the spontaneously hypertensive and hypertrophic heart rats.Densification of Type I Collagen Matrices as a Model for Cardiac Fibrosis.Bed rest decreases mechanically induced myofiber wounding and consequent wound-mediated FGF release.NRG-1-induced cardiomyocyte hypertrophy. Role of PI-3-kinase, p70(S6K), and MEK-MAPK-RSK.Cardiac troponin T elevation at dialysis initiation is associated with all-cause and cardiovascular mortality on dialysis in patients without diabetic nephropathy.Calcitriol downregulates fibroblast growth factor receptor 1 through histone deacetylase activation in HL-1 atrial myocytes.Modelling multi-scale cell-tissue interaction of tissue-engineered muscle constructs

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P2860

Role of transiently altered sarcolemmal membrane permeability and basic fibroblast growth factor release in the hypertrophic response of adult rat ventricular myocytes to increased mechanical activity in vitro

Item

http://www.wikidata.org/entity/Q37350080

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on January 1996

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Role of transiently altered sa ...... d mechanical activity in vitro

@en

Role of transiently altered sa ...... mechanical activity in vitro.

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type

Item

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Role of transiently altered sa ...... d mechanical activity in vitro

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Role of transiently altered sa ...... mechanical activity in vitro.

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Role of transiently altered sa ...... d mechanical activity in vitro

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Role of transiently altered sa ...... mechanical activity in vitro.

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P1476

Role of transiently altered sa ...... d mechanical activity in vitro

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D Kaye

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D Pimental

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H J Berger

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P L McNeil

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R A Kelly

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S Prasad

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T Mäki

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T W Smith

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P2860

Molecular cloning of a novel cytokine cDNA encoding the ninth member of the fibroblast growth factor family, which has a unique secretion property

Alternative translation of human fibroblast growth factor 2 mRNA occurs by internal entry of ribosomes

Signal transduction from the extracellular matrix

Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

Regulation of cardiac gene expression during myocardial growth and hypertrophy: molecular studies of an adaptive physiologic response

Autocrine release of angiotensin II mediates stretch-induced hypertrophy of cardiac myocytes in vitro

Molecular characterization of angiotensin II--induced hypertrophy of cardiac myocytes and hyperplasia of cardiac fibroblasts. Critical role of the AT1 receptor subtype

Acidic fibroblast growth factor mRNA is expressed by cardiac myocytes in culture and the protein is localized to the extracellular matrix.

Fibroblast growth factor stimulates angiotensin converting enzyme expression in vascular smooth muscle cells. Possible mediator of the response to vascular injury.

Isolation, characterization, and localization of heparin-binding growth factors in the heart

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281-291

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scholarly article

P356

10.1172/JCI118414

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English

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1996-01-01T00:00:00Z

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14632473

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8567946

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507016

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Role of transiently altered sarcolemmal membrane permeability and basic fibroblast growth factor release in the hypertrophic response of adult rat ventricular myocytes to increased mechanical activity in vitro

about

P2860

P2860

Role of transiently altered sarcolemmal membrane permeability and basic fibroblast growth factor release in the hypertrophic response of adult rat ventricular myocytes to increased mechanical activity in vitro

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P577

P5875

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P577

P5875

P698

P932