Role of transiently altered sarcolemmal membrane permeability and basic fibroblast growth factor release in the hypertrophic response of adult rat ventricular myocytes to increased mechanical activity in vitro
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Sarcospan-deficient mice maintain normal muscle functionPathophysiological roles of FGF signaling in the heartFibroblast growth factor receptor 1 signaling in adult cardiomyocytes increases contractility and results in a hypertrophic cardiomyopathyThe fibrosis-cell death axis in heart failureFibroblast growth factors in cardiovascular disease: The emerging role of FGF21Study familial hypertrophic cardiomyopathy using patient-specific induced pluripotent stem cellsFibroblast growth factor-2 mediates pressure-induced hypertrophic responseOverexpression of FGF-2 increases cardiac myocyte viability after injury in isolated mouse hearts.Intramyocardial fibroblast myocyte communication.Bone marrow-derived cells contribute to fibrosis in the chronically failing heartImpaired cerebral cortex development and blood pressure regulation in FGF-2-deficient miceModulation of membrane traffic by mechanical stimuli.Fibroblast Growth Factor 2 Mediates Isoproterenol-induced Cardiac Hypertrophy through Activation of the Extracellular Regulated Kinase.Molecular pathogenesis of muscle degeneration in the delta-sarcoglycan-deficient hamsterDilated cardiomyopathy and impaired cardiac hypertrophic response to angiotensin II in mice lacking FGF-2.Animal models for muscular dystrophy show different patterns of sarcolemmal disruption.Electrical stimulation of neonatal cardiomyocytes results in the sequential activation of nuclear genes governing mitochondrial proliferation and differentiationIncreased troponin levels in nonischemic cardiac conditions and noncardiac diseases.Biological functions of the low and high molecular weight protein isoforms of fibroblast growth factor-2 in cardiovascular development and disease.Measurement of circulating concentrations of cardiac troponin I and T in healthy subjects: a tool for monitoring myocardial tissue renewal?The diagnostic utility of cardiac biomarkers in dialysis patients.Regulatory RNAs and paracrine networks in the heart.Cardiac actions of fibroblast growth factor 23.Relationship between strain levels and permeability of the plasma membrane in statically stretched myoblasts.Increased c-fos mRNA expression by human fibroblasts contracting stressed collagen matrices.Cell Injury-Induced Release of Fibroblast Growth Factor 2: Relevance to Intracerebral Mesenchymal Stromal Cell Transplantations.Angiotensin II-induced cardiac hypertrophy and fibrosis are promoted in mice lacking Fgf16.Release of cardiac troponin I from viable cardiomyocytes is mediated by integrin stimulation.Integrin stimulation favors uptake of macromolecules by cardiomyocytes in vitro.FGF-2 and FGF-16 protect isolated perfused mouse hearts from acute doxorubicin-induced contractile dysfunction.Exacerbation of myocardial injury in transgenic mice overexpressing FGF-2 is T cell dependent.Protection by endogenous FGF-2 against isoproterenol-induced cardiac dysfunction is attenuated by cyclosporine A.Myocyte contractile activity modulates norepinephrine cytotoxicity and survival effects of neuregulin-1beta.Sequence analysis of the fibroblast growth factor 2 gene from the spontaneously hypertensive and hypertrophic heart rats.Densification of Type I Collagen Matrices as a Model for Cardiac Fibrosis.Bed rest decreases mechanically induced myofiber wounding and consequent wound-mediated FGF release.NRG-1-induced cardiomyocyte hypertrophy. Role of PI-3-kinase, p70(S6K), and MEK-MAPK-RSK.Cardiac troponin T elevation at dialysis initiation is associated with all-cause and cardiovascular mortality on dialysis in patients without diabetic nephropathy.Calcitriol downregulates fibroblast growth factor receptor 1 through histone deacetylase activation in HL-1 atrial myocytes.Modelling multi-scale cell-tissue interaction of tissue-engineered muscle constructs
P2860
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P2860
Role of transiently altered sarcolemmal membrane permeability and basic fibroblast growth factor release in the hypertrophic response of adult rat ventricular myocytes to increased mechanical activity in vitro
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 1996
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Role of transiently altered sa ...... d mechanical activity in vitro
@en
Role of transiently altered sa ...... mechanical activity in vitro.
@nl
type
label
Role of transiently altered sa ...... d mechanical activity in vitro
@en
Role of transiently altered sa ...... mechanical activity in vitro.
@nl
prefLabel
Role of transiently altered sa ...... d mechanical activity in vitro
@en
Role of transiently altered sa ...... mechanical activity in vitro.
@nl
P2093
P2860
P356
P1476
Role of transiently altered sa ...... d mechanical activity in vitro
@en
P2093
D Pimental
H J Berger
P L McNeil
P2860
P304
P356
10.1172/JCI118414
P407
P577
1996-01-01T00:00:00Z