A nonhomologous end-joining pathway is required for protein phosphatase 2A promotion of DNA double-strand break repair. - Wikidata - awgldk - TriplyDB

A nonhomologous end-joining pathway is required for protein phosphatase 2A promotion of DNA double-strand break repair.

A nonhomologous end-joining pathway is required for protein phosphatase 2A promotion of DNA double-strand break repair.

http://www.wikidata.org/entity/Q37350638

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Dinosaurs and ancient civilizations: reflections on the treatment of cancer What goes on must come off: phosphatases gate-crash the DNA damage response The Adenovirus E4orf4 Protein Provides a Novel Mechanism for Inhibition of the DNA Damage Response Valosin-containing protein regulates the proteasome-mediated degradation of DNA-PKcs in glioma cells.Protein phosphatase 6 interacts with the DNA-dependent protein kinase catalytic subunit and dephosphorylates gamma-H2AX.Phosphatases in the cellular response to DNA damage.Minichromosome maintenance 2 bound with retroviral Gp70 is localized to cytoplasm and enhances DNA-damage-induced apoptosis.Inhibition of protein phosphatase 2A with a small molecule LB100 radiosensitizes nasopharyngeal carcinoma xenografts by inducing mitotic catastrophe and blocking DNA damage repair.Role of Ku70 in deubiquitination of Mcl-1 and suppression of apoptosis.Inhibition of protein phosphatase 2A radiosensitizes pancreatic cancers by modulating CDC25C/CDK1 and homologous recombination repair The interconnectedness of cancer cell signaling.Targeting MCM2 function as a novel strategy for the treatment of highly malignant breast tumors.Protein phosphatase 2A and DNA-dependent protein kinase are involved in mediating rapamycin-induced Akt phosphorylation.Silencing CDK4 radiosensitizes breast cancer cells by promoting apoptosis.The War on Cancer rages on.Radiation induces premature chromatid separation via the miR-142-3p/Bod1 pathway in carcinoma cells Methylation of promoter of RBL1 enhances the radioresistance of three dimensional cultured carcinoma cells Detection and repair of ionizing radiation-induced DNA double strand breaks: new developments in nonhomologous end joining.RhoB promotes γH2AX dephosphorylation and DNA double-strand break repair Response to DNA damage: why do we need to focus on protein phosphatases?Inhibition of DNA-dependent protein kinase reduced palmitate and oleate-induced lipid accumulation in HepG2 cells.A Role for the Twins Protein Phosphatase (PP2A-B55) in the Maintenance of Drosophila Genome Integrity.

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A nonhomologous end-joining pathway is required for protein phosphatase 2A promotion of DNA double-strand break repair.

http://www.wikidata.org/entity/Q37350638

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on October 2009

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

A nonhomologous end-joining pa ...... NA double-strand break repair.

@en

A nonhomologous end-joining pa ...... NA double-strand break repair.

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type

label

A nonhomologous end-joining pa ...... NA double-strand break repair.

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A nonhomologous end-joining pa ...... NA double-strand break repair.

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prefLabel

A nonhomologous end-joining pa ...... NA double-strand break repair.

@en

A nonhomologous end-joining pa ...... NA double-strand break repair.

@nl

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A nonhomologous end-joining pa ...... NA double-strand break repair.

@en

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Fengqin Gao

http://www.w3.org/2001/XMLSchema#string

Qinhong Wang

http://www.w3.org/2001/XMLSchema#string

Tammy Flagg

http://www.w3.org/2001/XMLSchema#string

Ton Wang

http://www.w3.org/2001/XMLSchema#string

Xingming Deng

http://www.w3.org/2001/XMLSchema#string

P2860

MDC1 directly binds phosphorylated histone H2AX to regulate cellular responses to DNA double-strand breaks

The B56 family of protein phosphatase 2A (PP2A) regulatory subunits encodes differentiation-induced phosphoproteins that target PP2A to both nucleus and cytoplasm

Megabase chromatin domains involved in DNA double-strand breaks in vivo

DNA double-strand breaks: signaling, repair and the cancer connection

Chromosomal stability and the DNA double-stranded break connection

Reversible phosphorylation of Bcl2 following interleukin 3 or bryostatin 1 is mediated by direct interaction with protein phosphatase 2A

Activation of DNA-PK by ionizing radiation is mediated by protein phosphatase 6.

Replication protein A and gamma-H2AX foci assembly is triggered by cellular response to DNA double-strand breaks.

Bcl2 negatively regulates DNA double-strand-break repair through a nonhomologous end-joining pathway

DNA double strand break repair in mammalian cells.

P304

1012-1021

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scholarly article

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10.1593/NEO.09720

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10

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11

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2009-10-01T00:00:00Z

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19794960

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