Prostaglandins increase trabecular meshwork outflow facility in cultured human anterior segments. - Wikidata - awgldk - TriplyDB

Prostaglandins increase trabecular meshwork outflow facility in cultured human anterior segments.

Prostaglandins increase trabecular meshwork outflow facility in cultured human anterior segments.

http://www.wikidata.org/entity/Q37351340

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Current understanding of conventional outflow dysfunction in glaucoma From the bedside to the bench and back again: predicting and improving the outcomes of SLT glaucoma therapy.Stanniocalcin-1 Is an Ocular Hypotensive Agent and a Downstream Effector Molecule That Is Necessary for the Intraocular Pressure-Lowering Effects of Latanoprost.Prostaglandin pathway gene therapy for sustained reduction of intraocular pressure Clinical utility and differential effects of prostaglandin analogs in the management of raised intraocular pressure and ocular hypertension The influence of topical prostaglandin analogues in inflammation after selective laser trabeculoplasty treatment.[Effectiveness of selective laser trabeculoplasty in patients with insufficient control of intraocular pressure despite maximum tolerated medical therapy].Baseline Factors Predictive of SLT Response: A Prospective Study.Effects of prostaglandin analogues on aqueous humor outflow pathways.Recent Patents on Emerging Therapeutics for the Treatment of Glaucoma, Age Related Macular Degeneration and Uveitis.Endothelin antagonism as an active principle for glaucoma therapy Clinical options for the reduction of elevated intraocular pressure Cellular basis for bimatoprost effects on human conventional outflow.Trans-conjunctival aqueous humor outflow in glaucomatous patients treated with prostaglandin analogues: an in vivo confocal microscopy study.ATP-sensitive potassium (KATP) channel activation decreases intraocular pressure in the anterior chamber of the eye Expression profile of the matricellular protein osteopontin in primary open-angle glaucoma and the normal human eye.Aqueous humor outflow: dynamics and disease.Effects of Latanoprost and Bimatoprost on the Expression of Molecules Relevant to Ocular Inflow and Outflow Pathways Pharmacologic manipulation of conventional outflow facility in ex vivo mouse eyes Effect of topical latanoprost 0.005% on intraocular pressure and pupil diameter in normal and glaucomatous cats Biological properties of trabecular meshwork cells.Bioengineered glaucomatous 3D human trabecular meshwork as an in vitro disease model.Differential effects of prostaglandin E2-sensitive receptors on contractility of human ocular cells that regulate conventional outflow Imaging the effects of prostaglandin analogues on cultured trabecular meshwork cells by coherent anti-stokes Raman scattering Circadian variation of aqueous humor dynamics in older healthy adults.Update on the mechanism of action of topical prostaglandins for intraocular pressure reduction.Extracellular matrix turnover and outflow resistance.The influence of genetic background on conventional outflow facility in mice.Endogenous Bioactive Lipids and the Regulation of Conventional Outflow Facility.Emerging drugs for ocular hypertension.Pharmacology of topical prostaglandin F2 α analogs and their place in the treatment of glaucoma in small animals.Safety and efficacy of travoprost solution for the treatment of elevated intraocular pressure.Unconventional aqueous humor outflow: A review.Emerging drugs to treat glaucoma: targeting prostaglandin F and E receptors.ATP sensitive potassium channel openers: A new class of ocular hypotensive agents.Extracellular release of ATP mediated by cyclic mechanical stress leads to mobilization of AA in trabecular meshwork cells.Biomechanical, ultrastructural, and electrophysiological characterization of the non-human primate experimental glaucoma model.Open-angle glaucoma: therapeutically targeting the extracellular matrix of the conventional outflow pathway.Recreating a human trabecular meshwork outflow system on microfabricated porous structures.[Functional morphology of the outflow pathways of aqueous humor and their changes in open angle glaucoma].

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P2860

Prostaglandins increase trabecular meshwork outflow facility in cultured human anterior segments.

http://www.wikidata.org/entity/Q37351340

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2007 nî lūn-bûn

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Cindy K Bahler

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Douglas H Johnson

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Kyle G Howell

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P2860

Effects of AGN 192024, a new ocular hypotensive agent, on aqueous dynamics.

Prostaglandins increase matrix metalloproteinase release from human ciliary smooth muscle cells.

The prostanoid EP2 receptor agonist butaprost increases uveoscleral outflow in the cynomolgus monkey.

Prostaglandin F2 alpha-isopropylester eye drops: effects in normal human eyes.

Mechanism of action of bimatoprost, latanoprost, and travoprost in healthy subjects. A crossover study.

Human scleral hydraulic conductivity: age-related changes, topographical variation, and potential scleral outflow facility.

Differential effects of vasodilatory prostaglandins on focal adhesions, cytoskeletal architecture, and migration in human aortic smooth muscle cells.

Increased human scleral permeability with prostaglandin exposure.

Ocular and systemic pharmacokinetics of latanoprost in humans.

A six-month randomized clinical trial comparing the intraocular pressure-lowering efficacy of bimatoprost and latanoprost in patients with ocular hypertension or glaucoma.

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