Proteomic studies of nitrated alpha-synuclein microglia regulation by CD4+CD25+ T cells. - Wikidata - awgldk - TriplyDB

Proteomic studies of nitrated alpha-synuclein microglia regulation by CD4+CD25+ T cells.

Proteomic studies of nitrated alpha-synuclein microglia regulation by CD4+CD25+ T cells.

http://www.wikidata.org/entity/Q37355257

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Regulatory T cell in stroke: a new paradigm for immune regulation Neuroimmunological processes in Parkinson's disease and their relation to α-synuclein: microglia as the referee between neuronal processes and peripheral immunity Role of neuroinflammation in neurodegenerative diseases (Review)Regulatory T cells attenuate Th17 cell-mediated nigrostriatal dopaminergic neurodegeneration in a model of Parkinson's disease.Control of neuroinflammation as a therapeutic strategy for amyotrophic lateral sclerosis and other neurodegenerative disorders Functional proteomic analysis for regulatory T cell surveillance of the HIV-1-infected macrophage.Adaptive immune regulation of glial homeostasis as an immunization strategy for neurodegenerative diseases T-Lymphocyte Deficiency Exacerbates Behavioral Deficits in the 6-OHDA Unilateral Lesion Rat Model for Parkinson's Disease.Granulocyte-macrophage colony stimulating factor exerts protective and immunomodulatory effects in cortical trauma Effect of thymic stimulation of CD4+ T cell expansion on disease onset and progression in mutant SOD1 mice.A53T-alpha-synuclein-overexpression in the mouse nigrostriatal pathway leads to early increase of 14-3-3 epsilon and late increase of GFAP Neuroinflammation is a key player in Parkinson's disease and a prime target for therapy.A model of nitric oxide induced α-synuclein misfolding in Parkinson's disease.Taking the Perspective that a Depressive State Reflects Inflammation: Implications for the Use of Antidepressants A Perspective on Roles Played by Innate and Adaptive Immunity in the Pathobiology of Neurodegenerative Disorders Regulatory T-lymphocytes mediate amyotrophic lateral sclerosis progression and survival CNS infiltration of peripheral immune cells: D-Day for neurodegenerative disease?GM-CSF induces neuroprotective and anti-inflammatory responses in 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine intoxicated mice.Regulatory T lymphocytes from ALS mice suppress microglia and effector T lymphocytes through different cytokine-mediated mechanisms.Neurodegenerative disorders and nanoformulated drug development.A coat of many colors: neuroimmune crosstalk in human immunodeficiency virus infection.ALS patients' regulatory T lymphocytes are dysfunctional, and correlate with disease progression rate and severity.Combined analysis of the glia secretome and the CSF proteome: neuroinflammation and novel biomarkers.Inflammation in neurodegenerative diseases.Understanding the molecular basis of Parkinson's disease, identification of biomarkers and routes to therapy.Activated immune cells in Parkinson's disease.Targeting metabolic inflammation in Parkinson's disease: implications for prospective therapeutic strategies.Inflammation and adaptive immunity in Parkinson's disease.T-cell-mediated regulation of neuroinflammation involved in neurodegenerative diseases.Complex Changes in the Innate and Adaptive Immunity Accompany Progressive Degeneration of the Nigrostriatal Pathway Induced by Intrastriatal Injection of 6-Hydroxydopamine in the Rat.The role of regulatory T cells in nervous system pathologies.14-3-3ε marks the amyloid-stimulated microglia long-term activation.Lymphocytes reduce nigrostriatal deficits in the 6-hydroxydopamine mouse model of Parkinson's disease.Brain ingress of regulatory T cells in a murine model of HIV-1 encephalitis.Immune responses in Parkinson's disease: interplay between central and peripheral immune systems.Immunity and inflammation in neurodegenerative diseases.Bacillus Calmette-Guerin vaccine-mediated neuroprotection is associated with regulatory T-cell induction in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine mouse model of Parkinson's disease.Updates on immunity and inflammation in Parkinson disease pathology.Combined Active Humoral and Cellular Immunization Approaches for the Treatment of Synucleinopathies.The TGF-β System As a Potential Pathogenic Player in Disease Modulation of Amyotrophic Lateral Sclerosis.

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Proteomic studies of nitrated alpha-synuclein microglia regulation by CD4+CD25+ T cells.

http://www.wikidata.org/entity/Q37355257

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on July 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Proteomic studies of nitrated alpha-synuclein microglia regulation by CD4+CD25+ T cells.

@en

Proteomic studies of nitrated alpha-synuclein microglia regulation by CD4+CD25+ T cells.

@nl

type

label

Proteomic studies of nitrated alpha-synuclein microglia regulation by CD4+CD25+ T cells.

@en

Proteomic studies of nitrated alpha-synuclein microglia regulation by CD4+CD25+ T cells.

@nl

prefLabel

Proteomic studies of nitrated alpha-synuclein microglia regulation by CD4+CD25+ T cells.

@en

Proteomic studies of nitrated alpha-synuclein microglia regulation by CD4+CD25+ T cells.

@nl

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Journal of Proteome Research

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Proteomic studies of nitrated alpha-synuclein microglia regulation by CD4+CD25+ T cells.

@en

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Ashley D Reynolds

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David K Stone

http://www.w3.org/2001/XMLSchema#string

Howard E Gendelman

http://www.w3.org/2001/XMLSchema#string

R Lee Mosley

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P2860

Microglial inflammation in the parkinsonian substantia nigra: relationship to alpha-synuclein deposition

Alpha-synuclein in Lewy bodies

The influence of age on T cell generation and TCR diversity

Identification of novel proteins associated with both alpha-synuclein and DJ-1

Parkinson's disease: mechanisms and models

Identification of cathepsin B as a mediator of neuronal death induced by Abeta-activated microglial cells using a functional genomics approach.

Proteome analysis of human substantia nigra in Parkinson's disease.

Proteomic fingerprints distinguish microglia, bone marrow, and spleen macrophage populations.

Involvement of proinflammatory factors, apoptosis, caspase-3 activation and Ca2+ disturbance in microglia activation-mediated dopaminergic cell degeneration.

Detection of biomarkers with a multiplex quantitative proteomic platform in cerebrospinal fluid of patients with neurodegenerative disorders.

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3497-3511

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scholarly article

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10.1021/PR9001614

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7

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8

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2009-07-01T00:00:00Z

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2747638

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