Attenuation of angiotensin II-induced vascular dysfunction and hypertension by overexpression of Thioredoxin 2. - Wikidata - awgldk - TriplyDB

Attenuation of angiotensin II-induced vascular dysfunction and hypertension by overexpression of Thioredoxin 2.

Attenuation of angiotensin II-induced vascular dysfunction and hypertension by overexpression of Thioredoxin 2.

http://www.wikidata.org/entity/Q37363718

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Targeting mitochondrial reactive oxygen species as novel therapy for inflammatory diseases and cancers Systemic and renal oxidative stress in the pathogenesis of hypertension: modulation of long-term control of arterial blood pressure by resveratrol Thioredoxins, glutaredoxins, and peroxiredoxins--molecular mechanisms and health significance: from cofactors to antioxidants to redox signaling Cross talk between mitochondria and NADPH oxidases Paradoxical Roles of Antioxidant Enzymes: Basic Mechanisms and Health Implications Transcriptome profiling of peripheral blood cells identifies potential biomarkers for doxorubicin cardiotoxicity in a rat model Mitochondria, endothelial cell function, and vascular diseases.Mechanisms of pathogenesis in drug hepatotoxicity putting the stress on mitochondria.Therapeutic targeting of mitochondrial superoxide in hypertension.Natural antioxidants and hypertension: promise and challenges.Enhanced expression of mitochondrial superoxide dismutase leads to prolonged in vivo cell cycle progression and up-regulation of mitochondrial thioredoxin.Reactive oxygen species, NADPH oxidases, and hypertension.Vascular endothelial function and hypertension: insights and directions Role of arterial telomere dysfunction in hypertension: relative contributions of telomere shortening and telomere uncapping.The impact of age-related dysregulation of the angiotensin system on mitochondrial redox balance The emerging role of the thioredoxin system in angiogenesis Cardiac NO signalling in the metabolic syndrome.Renal oxidative stress, oxygenation, and hypertension Deletion of thioredoxin-interacting protein in mice impairs mitochondrial function but protects the myocardium from ischemia-reperfusion injury Cardiovascular redox and ox stress proteomics.Studies of mitochondrial and nonmitochondrial sources implicate nicotinamide adenine dinucleotide phosphate oxidase(s) in the increased skeletal muscle superoxide generation that occurs during contractile activity Mitochondria and endothelial function Mitochondrial ROS in the prohypertensive immune response.Angiotensin II and oxidative stress in the failing heart Angiotensin II-induced production of mitochondrial reactive oxygen species: potential mechanisms and relevance for cardiovascular disease.Role of mitochondrial oxidative stress in hypertension.Oxidative stress in hypertension: role of the kidney.Thioredoxin, oxidative stress, cancer and aging.Vascular dysfunction in cerebrovascular disease: mechanisms and therapeutic intervention.Endothelial mitochondria and heart disease.Angiotensin II blockade: a strategy to slow ageing by protecting mitochondria?Regulation of endothelial function by mitochondrial reactive oxygen species.Systems biology of antioxidants.Evolving concepts of oxidative stress and reactive oxygen species in cardiovascular disease.New roles for old pathways? A circuitous relationship between reactive oxygen species and cyclo-oxygenase in hypertension.Thioredoxin superfamily and its effects on cardiac physiology and pathology.Mitochondria in endothelial cells: Sensors and integrators of environmental cues.AT1 receptor signaling pathways in the cardiovascular system.Mitochondria in Structural and Functional Cardiac Remodeling.The Future Challenge of Reactive Oxygen Species (ROS) in Hypertension: From Bench to Bed Side.

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Attenuation of angiotensin II-induced vascular dysfunction and hypertension by overexpression of Thioredoxin 2.

http://www.wikidata.org/entity/Q37363718

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 08 June 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Attenuation of angiotensin II- ...... erexpression of Thioredoxin 2.

@en

Attenuation of angiotensin II- ...... erexpression of Thioredoxin 2.

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type

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Attenuation of angiotensin II- ...... erexpression of Thioredoxin 2.

@en

Attenuation of angiotensin II- ...... erexpression of Thioredoxin 2.

@nl

prefLabel

Attenuation of angiotensin II- ...... erexpression of Thioredoxin 2.

@en

Attenuation of angiotensin II- ...... erexpression of Thioredoxin 2.

@nl

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HYPERTENSIONAHA.108.127928

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Attenuation of angiotensin II- ...... erexpression of Thioredoxin 2.

@en

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Daniela Fraccarollo

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Dean P Jones

http://www.w3.org/2001/XMLSchema#string

Georg Ertl

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Jason M Hansen

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Johann Bauersachs

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Julian D Widder

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Paolo Galuppo

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P2860

Overexpressed human mitochondrial thioredoxin confers resistance to oxidant-induced apoptosis in human osteosarcoma cells

Nox1 is involved in angiotensin II-mediated hypertension: a study in Nox1-deficient mice

Cardiac hypertrophy is associated with altered thioredoxin and ASK-1 signaling in a mouse model of menopause

Endothelial dihydrofolate reductase: critical for nitric oxide bioavailability and role in angiotensin II uncoupling of endothelial nitric oxide synthase

Angiotensin II-mediated hypertension in the rat increases vascular superoxide production via membrane NADH/NADPH oxidase activation. Contribution to alterations of vasomotor tone.

Role of extracellular superoxide dismutase in hypertension.

Renin, angiotensin, sodium and organ damage.

Is angiotensin II a direct mediator of left ventricular hypertrophy? Time for another look.

Endothelial-specific expression of mitochondrial thioredoxin improves endothelial cell function and reduces atherosclerotic lesions.

Reactive oxygen species in hypertension; An update.

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338-344

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scholarly article

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10.1161/HYPERTENSIONAHA.108.127928

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2

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54

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2009-06-08T00:00:00Z

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19506101

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P921

arterial hypertension

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2752391

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