Effects of oral contraceptives or a gonadotropin-releasing hormone agonist on ovarian carcinogenesis in genetically engineered mice.
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CD95 promotes tumour growthOvarian cancer development and metastasis.Foretinib (GSK1363089), an orally available multikinase inhibitor of c-Met and VEGFR-2, blocks proliferation, induces anoikis, and impairs ovarian cancer metastasis.Metformin inhibits ovarian cancer growth and increases sensitivity to paclitaxel in mouse models.The effects of 17β-estradiol and a selective estrogen receptor modulator, bazedoxifene, on ovarian carcinogenesis.Prior oral contraceptive use in ovarian cancer patients: assessing associations with overall and progression-free survivalMinireview: animal models and mechanisms of ovarian cancer development.Tumor repressor protein 53 and steroid hormones provide a new paradigm for ovarian cancer metastases.Epithelial ovarian cancer experimental models.
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Effects of oral contraceptives or a gonadotropin-releasing hormone agonist on ovarian carcinogenesis in genetically engineered mice.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 08 September 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Effects of oral contraceptives ...... n genetically engineered mice.
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Effects of oral contraceptives ...... n genetically engineered mice.
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type
label
Effects of oral contraceptives ...... n genetically engineered mice.
@en
Effects of oral contraceptives ...... n genetically engineered mice.
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prefLabel
Effects of oral contraceptives ...... n genetically engineered mice.
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Effects of oral contraceptives ...... n genetically engineered mice.
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P2093
P2860
P1476
Effects of oral contraceptives ...... in genetically engineered mice
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Daniela M Dinulescu
Helen H Kim
Ilyssa O Gordon
Keeley L Mui
Melissa L Gilliam
Sujatha Jagadeeswaran
Thomas N Krausz
Woo Seok Lee
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P304
P356
10.1158/1940-6207.CAPR-08-0236
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2009-09-08T00:00:00Z