Herpes simplex virus type-1 induces IFN-alpha production via Toll-like receptor 9-dependent and -independent pathways.
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Sendai virus infection induces efficient adaptive immunity independently of type I interferonsPathogen recognition and inflammatory signaling in innate immune defensesMacrophages and cytokines in the early defence against herpes simplex virusInnate immune responses to DNA virusesA Novel Toll-Like Receptor (TLR) Influences Compatibility between the Gastropod Biomphalaria glabrata, and the Digenean Trematode Schistosoma mansoniKey role of splenic myeloid DCs in the IFN-alphabeta response to adenoviruses in vivoTranslational mini-review series on Toll-like receptors: recent advances in understanding the role of Toll-like receptors in anti-viral immunityPlasmacytoid dendritic cells and type I IFN: 50 years of convergent historyCytoplasmic DNA innate immune pathwaysInnate immune DNA sensing pathways: STING, AIMII and the regulation of interferon production and inflammatory responsesCritical role for Cryopyrin/Nalp3 in activation of caspase-1 in response to viral infection and double-stranded RNAIkappaB kinase-alpha is critical for interferon-alpha production induced by Toll-like receptors 7 and 9Nucleic acid scavenging polymers inhibit extracellular DNA-mediated innate immune activation without inhibiting anti-viral responsesAn image-based genetic assay identifies genes in T1D susceptibility loci controlling cellular antiviral immunity in mouseAspartate-glutamate-alanine-histidine box motif (DEAH)/RNA helicase A helicases sense microbial DNA in human plasmacytoid dendritic cells.Superior immunogenicity of inactivated whole virus H5N1 influenza vaccine is primarily controlled by Toll-like receptor signalling.Interferon lambda inhibits herpes simplex virus type I infection of human astrocytes and neuronsThe virion host shut-off (vhs) protein blocks a TLR-independent pathway of herpes simplex virus type 1 recognition in human and mouse dendritic cells.Innate immune recognition of viruses and viral vectors.Plasmacytoid dendritic cells in HIV infection: striking a delicate balance.MyD88-dependent pro-interleukin-1β induction in dendritic cells exposed to food-grade synthetic amorphous silica.Early alpha/beta interferon production by myeloid dendritic cells in response to UV-inactivated virus requires viral entry and interferon regulatory factor 3 but not MyD88.Targeting the TLR9-MyD88 pathway in the regulation of adaptive immune responses.Endocytosis of HIV-1 activates plasmacytoid dendritic cells via Toll-like receptor-viral RNA interactionsProphylactic and therapeutic modulation of innate and adaptive immunity against mucosal infection of herpes simplex virusToll-like receptor (TLR) 2 and TLR9 expressed in trigeminal ganglia are critical to viral control during herpes simplex virus 1 infectionInnate immune response of human plasmacytoid dendritic cells to poxvirus infection is subverted by vaccinia E3 via its Z-DNA/RNA binding domainInvolvement of TLR2 and TLR4 in cell responses to Rickettsia akariUNC93B1 mediates innate inflammation and antiviral defense in the liver during acute murine cytomegalovirus infection.Matrix protein mutant of vesicular stomatitis virus stimulates maturation of myeloid dendritic cellsCD200R1 supports HSV-1 viral replication and licenses pro-inflammatory signaling functions of TLR2.Promoter organization of the interferon-A genes differentially affects virus-induced expression and responsiveness to TBK1 and IKKepsilon.Toll-like receptor-independent triggering of dendritic cell maturation by viruses.In vivo ablation of CD11c-positive dendritic cells increases susceptibility to herpes simplex virus type 1 infection and diminishes NK and T-cell responses.Recombinant modified vaccinia virus Ankara generating excess early double-stranded RNA transiently activates protein kinase R and triggers enhanced innate immune responses.A herpesvirus virulence factor inhibits dendritic cell maturation through protein phosphatase 1 and Ikappa B kinase.Mesenchymal stem cells detect and defend against gammaherpesvirus infection via the cGAS-STING pathway.Novel drugs targeting Toll-like receptors for antiviral therapy.Optimization of allergen standardizationPlasmacytoid dendritic cells contribute to systemic but not local antiviral responses to HSV infections.
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P2860
Herpes simplex virus type-1 induces IFN-alpha production via Toll-like receptor 9-dependent and -independent pathways.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 22 July 2004
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Herpes simplex virus type-1 in ...... ent and -independent pathways.
@en
Herpes simplex virus type-1 in ...... ent and -independent pathways.
@nl
type
label
Herpes simplex virus type-1 in ...... ent and -independent pathways.
@en
Herpes simplex virus type-1 in ...... ent and -independent pathways.
@nl
prefLabel
Herpes simplex virus type-1 in ...... ent and -independent pathways.
@en
Herpes simplex virus type-1 in ...... ent and -independent pathways.
@nl
P2093
P2860
P356
P1476
Herpes simplex virus type-1 in ...... ent and -independent pathways.
@en
P2093
Beatrix Schlatter
Cornelia Wagner
Frank Schmitz
Hermann Wagner
Hubertus Hochrein
Mark Suter
Matthias Schiemann
Meredith O'Keeffe
Stefan Bauer
P2860
P304
11416-11421
P356
10.1073/PNAS.0403555101
P407
P577
2004-07-22T00:00:00Z