Disruption of the arsenic (+3 oxidation state) methyltransferase gene in the mouse alters the phenotype for methylation of arsenic and affects distribution and retention of orally administered arsenate.

Disruption of the arsenic (+3 oxidation state) methyltransferase gene in the mouse alters the phenotype for methylation of arsenic and affects distribution and retention of orally administered arsenate.

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http://www.wikidata.org/entity/Q37392124

Q37392124

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Nutritional manipulation of one-carbon metabolism: effects on arsenic methylation and toxicity Gut microbiome perturbations induced by bacterial infection affect arsenic biotransformation Arsenite selectively inhibits mouse bone marrow lymphoid progenitor cell development in vivo and in vitro and suppresses humoral immunity in vivo Renal function is associated with indicators of arsenic methylation capacity in Bangladeshi adults Determinants and Consequences of Arsenic Metabolism Efficiency among 4,794 Individuals: Demographics, Lifestyle, Genetics, and Toxicity Arsenic exposure and toxicology: a historical perspective Differential methylation of the arsenic (III) methyltransferase promoter according to arsenic exposure Arsenic Exposure and Type 2 Diabetes: MicroRNAs as Mechanistic Links?Influence of cobalamin on arsenic metabolism in Bangladesh AS3MT-mediated tolerance to arsenic evolved by multiple independent horizontal gene transfers from bacteria to eukaryotes.Metabolism, toxicity and anticancer activities of arsenic compounds.Interspecies differences in metabolism of arsenic by cultured primary hepatocytes Arsenic (+ 3 oxidation state) methyltransferase and the methylation of arsenicals in the invertebrate chordate Ciona intestinalis.Polymorphisms in arsenic(+III oxidation state) methyltransferase (AS3MT) predict gene expression of AS3MT as well as arsenic metabolism.Evaluation of the carcinogenicity of inorganic arsenic.Human adaptation to arsenic-rich environments.An emerging role for epigenetic dysregulation in arsenic toxicity and carcinogenesis.Creatinine, arsenic metabolism, and renal function in an arsenic-exposed population in Bangladesh Arsenic immunotoxicity: a review.A transgenic Drosophila model for arsenic methylation suggests a metabolic rationale for differential dose-dependent toxicity endpoints.Involvement of N-6 adenine-specific DNA methyltransferase 1 (N6AMT1) in arsenic biomethylation and its role in arsenic-induced toxicity Regulation of iNOS function and cellular redox state by macrophage Gch1 reveals specific requirements for tetrahydrobiopterin in NRF2 activation.Differential susceptibility of human peripheral blood T cells to suppression by environmental levels of sodium arsenite and monomethylarsonous acid Mathematical model insights into arsenic detoxification Arsenic toxicology: translating between experimental models and human pathology.Identification of Small Molecule Inhibitors of Human As(III) S-Adenosylmethionine Methyltransferase (AS3MT)Interactive Effects of N6AMT1 and As3MT in Arsenic Biomethylation.Identification of an S-adenosylmethionine (SAM) dependent arsenic methyltransferase in Danio rerio.High-throughput screening-compatible assays of As(III) S-adenosylmethionine methyltransferase activity.Altered arsenic disposition in experimental nonalcoholic fatty liver disease.Methylated arsenic metabolites bind to PML protein but do not induce cellular differentiation and PML-RARα protein degradation Biological and behavioral factors modify urinary arsenic metabolic profiles in a U.S. population.N-6-adenine-specific DNA methyltransferase 1 (N6AMT1) polymorphisms and arsenic methylation in Andean women Analysis of maternal polymorphisms in arsenic (+3 oxidation state)-methyltransferase AS3MT and fetal sex in relation to arsenic metabolism and infant birth outcomes: Implications for risk analysis Body composition and arsenic metabolism: a cross-sectional analysis in the Strong Heart Study.Requirement of arsenic biomethylation for oxidative DNA damage.Oxidation state specific analysis of arsenic species in tissues of wild-type and arsenic (+3 oxidation state) methyltransferase-knockout mice.Linking selenium biogeochemistry to the sulfur-dependent biological detoxification of arsenic.Metabolism of arsenic and its toxicological relevance.Natural Antioxidants Against Arsenic-Induced Genotoxicity.

P2860

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P2860

Disruption of the arsenic (+3 oxidation state) methyltransferase gene in the mouse alters the phenotype for methylation of arsenic and affects distribution and retention of orally administered arsenate.

Item

http://www.wikidata.org/entity/Q37392124

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on October 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Disruption of the arsenic (+3 ...... orally administered arsenate.

@en

Disruption of the arsenic

@nl

type

Item

label

Disruption of the arsenic (+3 ...... orally administered arsenate.

@en

Disruption of the arsenic

@nl

prefLabel

Disruption of the arsenic (+3 ...... orally administered arsenate.

@en

Disruption of the arsenic

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P1476

Disruption of the arsenic (+3 ...... f orally administered arsenate

@en

P2093

Brenda C Edwards

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David J Thomas

http://www.w3.org/2001/XMLSchema#string

John T Creed

http://www.w3.org/2001/XMLSchema#string

Karen Herbin-Davis

http://www.w3.org/2001/XMLSchema#string

Kevin M Kubachka

http://www.w3.org/2001/XMLSchema#string

Michael F Hughes

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Miroslav Styblo

http://www.w3.org/2001/XMLSchema#string

Noboyu Maeda

http://www.w3.org/2001/XMLSchema#string

Zuzana Drobna

http://www.w3.org/2001/XMLSchema#string

P2860

Comparative genomic analysis of the gut bacterium Bifidobacterium longum reveals loci susceptible to deletion during pure culture growth

Microbial methylation of metalloids: arsenic, antimony, and bismuth.

Methylation study of a population environmentally exposed to arsenic in drinking water

Metabolism of low-dose inorganic arsenic in a central European population: influence of sex and genetic polymorphisms

Profile of urinary arsenic metabolites during pregnancy.

Elucidating the pathway for arsenic methylation

A comparative study of the sub-chronic toxic effects of three organic arsenical compounds on the urothelium in F344 rats; gender-based differences in response.

Chromatographic separation and identification of products from the reaction of dimethylarsinic acid with hydrogen sulfide.

Arsenic (+ 3 oxidation state) methyltransferase and the methylation of arsenicals in the invertebrate chordate Ciona intestinalis.

Arsenic methylation is a process of detoxification through accelerated excretion.

P304

1713-1720

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scholarly article

P356

10.1021/TX900179R

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P478

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X Le

Hua Naranmandura

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2009-10-01T00:00:00Z

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26752121

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19691357

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2763928

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Disruption of the arsenic (+3 oxidation state) methyltransferase gene in the mouse alters the phenotype for methylation of arsenic and affects distribution and retention of orally administered arsenate.

about

P2860

P2860

Disruption of the arsenic (+3 oxidation state) methyltransferase gene in the mouse alters the phenotype for methylation of arsenic and affects distribution and retention of orally administered arsenate.

description

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type

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P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P5875

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P1433

P1476

P2093

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P433

P478

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P932