Requirement of arsenic biomethylation for oxidative DNA damage. - Wikidata - awgldk - TriplyDB

Requirement of arsenic biomethylation for oxidative DNA damage.

Requirement of arsenic biomethylation for oxidative DNA damage.

http://www.wikidata.org/entity/Q37478021

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Renal, hepatic, pulmonary and adrenal tumors induced by prenatal inorganic arsenic followed by dimethylarsinic acid in adulthood in CD1 mice Arsenic contamination of groundwater: a review of sources, prevalence, health risks, and strategies for mitigation Exposure to inorganic arsenic can lead to gut microbe perturbations and hepatocellular carcinoma Long isoforms of NRF1 contribute to arsenic-induced antioxidant response in human keratinocytes Environmental toxicants, epigenetics, and cancer Metallothionein blocks oxidative DNA damage induced by acute inorganic arsenic exposure Arsenic transformation predisposes human skin keratinocytes to UV-induced DNA damage yet enhances their survival apparently by diminishing oxidant response Arsenic biotransformation as a cancer promoting factor by inducing DNA damage and disruption of repair mechanisms Chronic inorganic arsenic exposure in vitro induces a cancer cell phenotype in human peripheral lung epithelial cells The Use of Chemical-Chemical Interaction and Chemical Structure to Identify New Candidate Chemicals Related to Lung Cancer Methylarsonous acid causes oxidative DNA damage in cells independent of the ability to biomethylate inorganic arsenic Monomethylarsonous acid produces irreversible events resulting in malignant transformation of a human bladder cell line following 12 weeks of low-level exposure Arsenic, stem cells, and the developmental basis of adult cancer.The ATP-binding cassette transporter ABCB6 is induced by arsenic and protects against arsenic cytotoxicity.Site-specific radical formation in DNA induced by Cu(II)-H₂O₂ oxidizing system, using ESR, immuno-spin trapping, LC-MS, and MS/MS Individual variations in inorganic arsenic metabolism associated with AS3MT genetic polymorphisms.Nitric oxide donor, V-PROLI/NO, provides protection against arsenical induced toxicity in rat liver cells: requirement for Cyp1a1 Chronic administration of 2-acetylaminofluorene alters the cellular iron metabolism in rat liver.TP53 R72P polymorphism modulates DNA methylation in hepatocellular carcinoma.Differential DNA methylation profile of key genes in malignant prostate epithelial cells transformed by inorganic arsenic or cadmium Detection and imaging of the free radical DNA in cells--site-specific radical formation induced by Fenton chemistry and its repair in cellular DNA as seen by electron spin resonance, immuno-spin trapping and confocal microscopy.Arsenic-transformed malignant prostate epithelia can convert noncontiguous normal stem cells into an oncogenic phenotype.Identification of an S-adenosylmethionine (SAM) dependent arsenic methyltransferase in Danio rerio.Translational attenuation differentially alters the fate of disease-associated fibulin proteins Development of immunoblotting techniques for DNA radical detection Transcriptional Modulation of the ERK1/2 MAPK and NF-κB Pathways in Human Urothelial Cells After Trivalent Arsenical Exposure: Implications for Urinary Bladder Cancer.Obesity-induced tissue free radical generation: an in vivo immuno-spin trapping study.Arsenite Regulates Prolongation of Glycan Residues of Membrane Glycoprotein: A Pivotal Study via Wax Physisorption Kinetics and FTIR Imaging.In Vivo Immuno-Spin Trapping: Imaging the Footprints of Oxidative Stress.Chronic exposure of renal stem cells to inorganic arsenic induces a cancer phenotype.Aberrant microRNA expression likely controls RAS oncogene activation during malignant transformation of human prostate epithelial and stem cells by arsenic.Arsenic in the treatment of newly diagnosed acute promyelocytic leukemia: current status and future research direction.Oxidative DNA damage after acute exposure to arsenite and monomethylarsonous acid in biomethylation-deficient human cells.A semi-mechanistic integrated toxicokinetic-toxicodynamic (TK/TD) model for arsenic(III) in hepatocytes Oxidative DNA damage enhances the carcinogenic potential of in vitro chronic arsenic exposures.Ogg1 genetic background determines the genotoxic potential of environmentally relevant arsenic exposures.Superoxide dismutase protects cells from DNA damage induced by trivalent methylated arsenicals.Fish micronucleus assay to assess genotoxic potential of arsenic at its guideline exposure in aquatic environment.Effects of Inorganic Arsenic, Methylated Arsenicals, and Arsenobetaine on Atherosclerosis in the Mouse Model and the Role of As3mt-Mediated Methylation.Exosomal circRNA_100284 from arsenite-transformed cells, via microRNA-217 regulation of EZH2, is involved in the malignant transformation of human hepatic cells by accelerating the cell cycle and promoting cell proliferation.

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Requirement of arsenic biomethylation for oxidative DNA damage.

http://www.wikidata.org/entity/Q37478021

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on December 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Requirement of arsenic biomethylation for oxidative DNA damage.

@en

Requirement of arsenic biomethylation for oxidative DNA damage.

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type

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Requirement of arsenic biomethylation for oxidative DNA damage.

@en

Requirement of arsenic biomethylation for oxidative DNA damage.

@nl

prefLabel

Requirement of arsenic biomethylation for oxidative DNA damage.

@en

Requirement of arsenic biomethylation for oxidative DNA damage.

@nl

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Journal of the National Cancer Institute

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Requirement of arsenic biomethylation for oxidative DNA damage.

@en

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Chikara Kojima

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Dario C Ramirez

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Erik J Tokar

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Michael P Waalkes

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Miroslav Stýblo

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Ronald P Mason

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Seiichiro Himeno

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Zuzana Drobná

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P2860

Evidence for induction of oxidative stress caused by chronic exposure of Chinese residents to arsenic contained in drinking water

Association of arsenic-induced malignant transformation with DNA hypomethylation and aberrant gene expression

Further studies on aberrant gene expression associated with arsenic-induced malignant transformation in rat liver TRL1215 cells

Chronic exposure to methylated arsenicals stimulates arsenic excretion pathways and induces arsenic tolerance in rat liver cells

Comprehensive profiling and localisation of the matrix metalloproteinases in urothelial carcinoma.

Monomethylarsonous acid induces transformation of human bladder cells.

Monomethylarsonous acid (MMA(III)) is more toxic than arsenite in Chang human hepatocytes.

Methylated trivalent arsenic species are genotoxic.

Transcription factor Nrf2 activation by inorganic arsenic in cultured keratinocytes: involvement of hydrogen peroxide.

The cellular metabolism and systemic toxicity of arsenic.

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scholarly article

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10.1093/JNCI/DJP414

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2009-12-01T00:00:00Z

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19933942

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