Kidney hypoxia, attributable to increased oxygen consumption, induces nephropathy independently of hyperglycemia and oxidative stress. - Wikidata - awgldk - TriplyDB

Kidney hypoxia, attributable to increased oxygen consumption, induces nephropathy independently of hyperglycemia and oxidative stress.

Kidney hypoxia, attributable to increased oxygen consumption, induces nephropathy independently of hyperglycemia and oxidative stress.

http://www.wikidata.org/entity/Q37402065

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Hypoxia: The Force that Drives Chronic Kidney Disease Chronic renal ischemia in humans: can cell therapy repair the kidney in occlusive renovascular disease?Hyperpolarized 13C urea relaxation mechanism reveals renal changes in diabetic nephropathy.Modeling Glucose Metabolism in the Kidney.Renal Oxygenation in the Pathophysiology of Chronic Kidney Disease.Activation of hypoxia-inducible factors prevents diabetic nephropathy.Insufficient insulin administration to diabetic rats increases substrate utilization and maintains lactate production in the kidney Soluble Tumor Necrosis Factor Receptor 1 Is Associated with Glomerular Filtration Rate Progression and Incidence of Chronic Kidney Disease in Two Community-Based Cohorts of Elderly Individuals.Hypertension: a problem of organ blood flow supply-demand mismatch.Predicted consequences of diabetes and SGLT inhibition on transport and oxygen consumption along a rat nephron.Mitochondrial activation chemicals synergize with surface receptor PD-1 blockade for T cell-dependent antitumor activity.Renal Hypoxia in CKD; Pathophysiology and Detecting Methods.Renal neurohormonal regulation in heart failure decompensation.Hypoxia in diabetic kidneys.How bold is blood oxygenation level-dependent (BOLD) magnetic resonance imaging of the kidney? Opportunities, challenges and future directions.Hypoxia as a key player in the AKI-to-CKD transition.The role of hypoxia and Morg1 in renal injury.Glucose Homeostasis and Cardiovascular Alterations in Diabetes.Mesenchymal stem cells and chronic renal artery stenosis.Expanding roles of the hypoxia-response network in chronic kidney disease.A pseudo-three-dimensional model for quantification of oxygen diffusion from preglomerular arteries to renal tissue and renal venous blood.A mechanistic link between renal ischemia and fibrosis.Mitochondrial Reactive Oxygen Species and Kidney Hypoxia in the Development of Diabetic Nephropathy Nocturnal hypoxemia severity and renin-angiotensin system activity in obstructive sleep apnea.Cooperative Oxygen Sensing by the Kidney and Carotid Body in Blood Pressure Control.Melatonin, mitochondria and hypertension.ET-1 increases reactive oxygen species in hypoxic glomeruli during high salt intake.Carbohydrate restriction ameliorates nephropathy by reducing oxidative stress and upregulating HIF-1α levels in type-1 diabetic rats.Intermittent hypoxia causes histological kidney damage and increases growth factor expression in a mouse model of obstructive sleep apnea.Modeling glucose metabolism and lactate production in the kidney.Accounting for oxygen in the renal cortex: a computational study of factors that predispose the cortex to hypoxia.Cellular transport of l-arginine determines renal medullary blood flow in control rats, but not in diabetic rats despite enhanced cellular uptake capacity.Renal transplantation induces mitochondrial uncoupling, increased kidney oxygen consumption, and decreased kidney oxygen tension.Renal hypoxia in kidney disease: Cause or consequence?Renal fibrosis: Primacy of the proximal tubule.Renal cellular hypoxia in adenine-induced chronic kidney disease.INTRARENAL ACTIVATION OF ENDOTHELIN TYPE B RECEPTORS IMPROVES KIDNEY OXYGENATION IN TYPE 1 DIABETIC RATS.Endothelin type A receptor inhibition normalises intrarenal hypoxia in rats used as a model of type 1 diabetes by improving oxygen delivery.Adenosine A2 a receptor stimulation prevents proteinuria in diabetic rats by promoting an anti-inflammatory phenotype without affecting oxidative stress.Editorial: Hypoxia in Kidney Disease.

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Kidney hypoxia, attributable to increased oxygen consumption, induces nephropathy independently of hyperglycemia and oxidative stress.

http://www.wikidata.org/entity/Q37402065

description

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scientific article published on 09 September 2013

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vedecký článok

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vetenskaplig artikel

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vědecký článek

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Kidney hypoxia, attributable t ...... glycemia and oxidative stress.

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Kidney hypoxia, attributable t ...... glycemia and oxidative stress.

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Kidney hypoxia, attributable t ...... glycemia and oxidative stress.

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Kidney hypoxia, attributable t ...... glycemia and oxidative stress.

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Kidney hypoxia, attributable t ...... glycemia and oxidative stress.

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Kidney hypoxia, attributable t ...... glycemia and oxidative stress.

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HYPERTENSIONAHA.113.01425

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Kidney hypoxia, attributable t ...... rglycemia and oxidative stress

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Erik Thörn

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Fredrik Palm

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Malou Friederich-Persson

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Masaomi Nangaku

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Max Levin

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Global prevalence of diabetes: estimates for the year 2000 and projections for 2030

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Reactive oxygen species cause diabetes-induced decrease in renal oxygen tension.

Reduced oxygenation in diabetic rat kidneys measured by T2* weighted magnetic resonance micro-imaging.

Blood pressure, blood flow, and oxygenation in the clipped kidney of chronic 2-kidney, 1-clip rats: effects of tempol and Angiotensin blockade.

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Dinitrophenol and bioenergetics: an historical perspective.

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The mitochondrial uncoupler 2,4-dinitrophenol attenuates tissue damage and improves mitochondrial homeostasis following transient focal cerebral ischemia.

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