about
Hypoperfusion in the renal outer medulla after injection of contrast media in rats.Renal hyaluronan accumulation and hyaluronan synthase expression after ischaemia-reperfusion injury in the rat.Reactive oxygen species cause diabetes-induced decrease in renal oxygen tension.Adenosine A1 receptors in contrast media-induced renal dysfunction in the normal rat.Polyol-pathway-dependent disturbances in renal medullary metabolism in experimental insulin-deficient diabetes mellitus in rats.Altered response in renal blood flow and oxygen tension to contrast media in diabetic rats.The effects of carbon dioxide versus ioxaglate in the rat kidney.Influence of iotrolan on renal cortical and outer medullary blood flow in the rat.Reduced nitric oxide concentration in the renal cortex of streptozotocin-induced diabetic rats: effects on renal oxygenation and microcirculation.Influence of iothalamate on renal medullary perfusion and oxygenation in the rat.Uncoupling protein-2 in diabetic kidneys: increased protein expression correlates to increased non-transport related oxygen consumption.Reduced oxygenation in diabetic rat kidneys measured by T2* weighted magnetic resonance micro-imaging.Identification and distribution of uncoupling protein isoforms in the normal and diabetic rat kidney.Iodinated contrast media decrease renomedullary blood flow. A possible cause of contrast media-induced nephropathy.Nitric oxide originating from NOS1 controls oxygen utilization and electrolyte transport efficiency in the diabetic kidney.Tubular reabsorption and diabetes-induced glomerular hyperfiltration.Hypoxia in the diabetic kidney is independent of advanced glycation end-products.Activation of hypoxia-inducible factors prevents diabetic nephropathy.NADPH oxidase inhibition reduces tubular sodium transport and improves kidney oxygenation in diabetes.High Sensitivity Method to Estimate Distribution of Hyaluronan Molecular Sizes in Small Biological Samples Using Gas-Phase Electrophoretic Mobility Molecular Analysis.Pathophysiology of contrast medium-induced nephropathy.Effects of intravenous contrast media on cortical and medullary blood flow in the rat kidney.Diabetes-induced decrease in renal oxygen tension: effects of an altered metabolism.Inhibition of mTOR activity in diabetes mellitus reduces proteinuria but not renal accumulation of hyaluronanNitric oxide and kidney oxygenation.Kidney hypoxia, attributable to increased oxygen consumption, induces nephropathy independently of hyperglycemia and oxidative stress.Diabetes, oxidative stress, nitric oxide and mitochondria function.Determinants of kidney oxygen consumption and their relationship to tissue oxygen tension in diabetes and hypertension.Renal interstitial hyaluronan: functional aspects during normal and pathological conditions.A role for the extracellular matrix component hyaluronan in kidney dysfunction during ACE-inhibitor fetopathy.Iodinated contrast media inhibit oxygen consumption in freshly isolated proximal tubular cells from elderly humans and diabetic rats: Influence of nitric oxide.Renal effects of CO2 and iodinated contrast media in patients undergoing renovascular intervention: a prospective, randomized study.CNS-induced natriuresis, neurohypophyseal peptides and renal dopamine and noradrenaline excretion in prehypertensive salt-sensitive Dahl rats.Angiotensin converting enzyme inhibition blocks interstitial hyaluronan dissipation in the neonatal rat kidney via hyaluronan synthase 2 and hyaluronidase 1.Inhibition of hyaluronan synthesis in rats reduces renal ability to excrete fluid and electrolytes during acute hydration.Renomedullary interstitial cells in culture; the osmolality and oxygen tension influence the extracellular amounts of hyaluronan and cellular expression of CD44.Fluid balance in patients with chronic renal failure assessed with N-terminal proatrial natriuretic peptide, atrial natriuretic peptide and ultrasonography.Renal failure in 57 925 patients undergoing coronary procedures using iso-osmolar or low-osmolar contrast media.The role of dopamine-metabolizing enzymes in the regulation of renal sodium excretion in the rat.Renal cortical accumulation of hyaluronan in adult rats exposed neonatally to angiotensin-converting enzyme inhibition.
P50
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P50
description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
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name
Peter Hansell
@ast
Peter Hansell
@en
Peter Hansell
@es
Peter Hansell
@nl
Peter Hansell
@sl
type
label
Peter Hansell
@ast
Peter Hansell
@en
Peter Hansell
@es
Peter Hansell
@nl
Peter Hansell
@sl
prefLabel
Peter Hansell
@ast
Peter Hansell
@en
Peter Hansell
@es
Peter Hansell
@nl
Peter Hansell
@sl
P106
P21
P31
P496
0000-0002-0315-8554