Neutralization of vascular endothelial growth factor antiangiogenic isoforms is more effective than treatment with proangiogenic isoforms in stimulating vascular development and follicle progression in the perinatal rat ovary.
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Vascular endothelial growth factor A: just one of multiple mechanisms for sex-specific vascular development within the testis?The anti-angiogenic isoforms of VEGF in health and disease.Molecular diversity of VEGF-A as a regulator of its biological activity.Do anti-angiogenic VEGF (VEGFxxxb) isoforms exist? A cautionary taleThe carboxyl terminus of VEGF-A is a potential target for anti-angiogenic therapyLoss of vascular endothelial growth factor A (VEGFA) isoforms in granulosa cells using pDmrt-1-Cre or Amhr2-Cre reduces fertility by arresting follicular development and by reducing litter size in female mice.Neutralization of vascular endothelial growth factor antiangiogenic isoforms or administration of proangiogenic isoforms stimulates vascular development in the rat testis.Transcriptomic diversification of developing cumulus and mural granulosa cells in mouse ovarian follicles.Ovarian VEGF(165)b expression regulates follicular development, corpus luteum function and fertilityThe balance of proangiogenic and antiangiogenic VEGFA isoforms regulate follicle development.The role of VEGF 165b in pathophysiologyLoss of vascular endothelial growth factor A (VEGFA) isoforms in the testes of male mice causes subfertility, reduces sperm numbers, and alters expression of genes that regulate undifferentiated spermatogonia.VEGF(121)b, a new member of the VEGF(xxx)b family of VEGF-A splice isoforms, inhibits neovascularisation and tumour growth in vivoWT1 mutants reveal SRPK1 to be a downstream angiogenesis target by altering VEGF splicing.The VEGF165b "ICE-o-form" puts a chill on the VEGF story.Impaired vascular permeability regulation caused by the VEGF₁₆₅b splice variant in pre-eclampsia.Anti-angiogenic VEGFAxxxb transcripts are not expressed in the medio-basal hypothalamus of the seasonal sheep.
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P2860
Neutralization of vascular endothelial growth factor antiangiogenic isoforms is more effective than treatment with proangiogenic isoforms in stimulating vascular development and follicle progression in the perinatal rat ovary.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
@tr
scientific article published on 15 July 2009
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Neutralization of vascular end ...... on in the perinatal rat ovary.
@en
Neutralization of vascular end ...... on in the perinatal rat ovary.
@nl
type
label
Neutralization of vascular end ...... on in the perinatal rat ovary.
@en
Neutralization of vascular end ...... on in the perinatal rat ovary.
@nl
prefLabel
Neutralization of vascular end ...... on in the perinatal rat ovary.
@en
Neutralization of vascular end ...... on in the perinatal rat ovary.
@nl
P2093
P2860
P1476
Neutralization of vascular end ...... on in the perinatal rat ovary.
@en
P2093
Andrea S Cupp
Debra T Clopton
Michelle M Baltes-Breitwisch
Renee M McFee
Robin A Artac
Robyn A Longfellow Smith
P2860
P304
P356
10.1095/BIOLREPROD.109.078097
P407
P577
2009-07-15T00:00:00Z