Insulin signaling in skeletal muscle and liver of neonatal pigs during endotoxemia.

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http://www.wikidata.org/entity/Q37414140

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

@wuu

2008年学术文章

@zh-cn

2008年学术文章

@zh-hans

2008年学术文章

@zh-my

2008年学术文章

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2008年學術文章

@yue

2008年學術文章

@zh

2008年學術文章

@zh-hant

name

Insulin signaling in skeletal muscle and liver of neonatal pigs during endotoxemia.

@en

Insulin signaling in skeletal muscle and liver of neonatal pigs during endotoxemia.

@nl

type

Item

label

Insulin signaling in skeletal muscle and liver of neonatal pigs during endotoxemia.

@en

Insulin signaling in skeletal muscle and liver of neonatal pigs during endotoxemia.

@nl

prefLabel

Insulin signaling in skeletal muscle and liver of neonatal pigs during endotoxemia.

@en

Insulin signaling in skeletal muscle and liver of neonatal pigs during endotoxemia.

@nl

P1476

Insulin signaling in skeletal muscle and liver of neonatal pigs during endotoxemia.

@en

P2093

Agus Suryawan

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Guoyao Wu

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Hanh V Nguyen

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Leonard S Jefferson

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Renán A Orellana

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Scot R Kimball

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Teresa A Davis

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P2860

Arginine metabolism: nitric oxide and beyond

IRS-1-mediated inhibition of insulin receptor tyrosine kinase activity in TNF-alpha- and obesity-induced insulin resistance

4E-BP1, a repressor of mRNA translation, is phosphorylated and inactivated by the Akt(PKB) signaling pathway

Control of Ser2448 phosphorylation in the mammalian target of rapamycin by insulin and skeletal muscle load.

Nitric oxide in septic shock.

Potential role of TNF-alpha in the pathogenesis of insulin resistance and type 2 diabetes.

Regulation of muscle protein synthesis during sepsis and inflammation.

A phosphatidylinositol 3-kinase/Akt/mTOR pathway mediates and PTEN antagonizes tumor necrosis factor inhibition of insulin signaling through insulin receptor substrate-1.

Activation of RAC-protein kinase by heat shock and hyperosmolarity stress through a pathway independent of phosphatidylinositol 3-kinase.

Stimulation of protein synthesis, eukaryotic translation initiation factor 4E phosphorylation, and PHAS-I phosphorylation by insulin requires insulin receptor substrate 1 and phosphatidylinositol 3-kinase.

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505-510

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scholarly article

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10.1203/PDR.0B013E318183FD4C

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English

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2008-11-01T00:00:00Z

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5255936

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1038784366

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