Response to adversity: molecular control of gene activation following genotoxic stress.
about
Redox activation of Fos-Jun DNA binding activity is mediated by a DNA repair enzymeSulphur mustards inhibit binding of transcription factor AP2 in vitroUse of high-throughput RT-qPCR to assess modulations of gene expression profiles related to genomic stability and interactions by cadmiumOctyl methoxycinnamate modulates gene expression and prevents cyclobutane pyrimidine dimer formation but not oxidative DNA damage in UV-exposed human cell lines.Tumor suppressor p53 can participate in transcriptional induction of the GADD45 promoter in the absence of direct DNA bindingUp-regulation of base excision repair correlates with enhanced protection against a DNA damaging agent in mouse cell lines.The redox and DNA-repair activities of Ref-1 are encoded by nonoverlapping domainsInduction of RNA-binding proteins in mammalian cells by DNA-damaging agents.Identification of Cyclobutane Pyrimidine Dimer-Responsive Genes Using UVB-Irradiated Human Keratinocytes Transfected with In Vitro-Synthesized Photolyase mRNA.UVB-induced association of tumor necrosis factor (TNF) receptor 1/TNF receptor-associated factor-2 mediates activation of Rel proteins.Induction of the growth arrest and DNA damage-inducible gene GADD153 by cisplatin in vitro and in vivo.Enhancement of drug sensitivity of human malignancies by epidermal growth factor.Cisplatin and taxol activate different signal pathways regulating cellular injury-induced expression of GADD153Development of human cell biosensor system for genotoxicity detection based on DNA damage-induced gene expression.Transcription and activity of antioxidant enzymes after ionizing irradiation in radiation-resistant and radiation-sensitive miceActivation of AP-1 and of a nuclear redox factor, Ref-1, in the response of HT29 colon cancer cells to hypoxia.Induction of cellular p53 activity by DNA-damaging agents and growth arrest.The mdm-2 gene is induced in response to UV light in a p53-dependent manner.Strong and prolonged induction of c-jun and c-fos proto-oncogenes by photodynamic therapy.The redox/DNA repair protein, Ref-1, is essential for early embryonic development in mice.In vivo protein-DNA interactions at the c-jun promoter: preformed complexes mediate the UV response.UV irradiation induces the murine urokinase-type plasminogen activator gene via the c-Jun N-terminal kinase signaling pathway: requirement of an AP1 enhancer element.Exposure of HEp-2 cells to stress conditions influences antinuclear antibody reactivity.Expression of c-Fos and c-Jun in the cornea, lens, and retina after ultraviolet irradiation of the rat eye and effects of topical antisense oligodeoxynucleotides.CP-115,953 stimulates cytokine production by lymphocytes.The level of intracellular glutathione is a key regulator for the induction of stress-activated signal transduction pathways including Jun N-terminal protein kinases and p38 kinase by alkylating agentsLimited effects of temafloxacin compared with ciprofloxacin on T-lymphocyte function.UV-induced activation of AP-1 involves obligatory extranuclear steps including Raf-1 kinaseDepletion of the mitochondrial electron transport abrogates the cytotoxic and gene-inductive effects of TNF.Neuregulin 1-Beta cytoprotective role in AML 12 mouse hepatocytes exposed to pentachlorophenol
P2860
Q24555669-CDD8B7C5-0668-4506-8BD7-915123266A3BQ28367486-1ACEF179-2713-4CF4-B7C3-2DA68A25ECE6Q28821954-EF183E53-9AA8-465B-9FFA-C3C8BF947233Q33733379-3D6B9295-86E4-40E6-9F4C-02A87723AD7CQ33786522-FACE699F-0E17-45EC-9616-968AE6BF855CQ34662309-EB9E234B-6919-4B46-97EB-F6DC20F73685Q34878594-43E81B18-EE2B-4245-BD46-AE2BF1E6B975Q35057947-1E5B1AAD-75E0-45C7-8FAA-D6245B170794Q35677482-58C04ECC-48D5-4B49-B1F9-8EAB3668E630Q35739622-288E9A34-68C5-474B-BB17-451ED2E35FFDQ36080620-590BBC00-70CC-44D3-9D8E-CA8A2C7A28BDQ36081127-00192E0F-0281-4E35-AFD9-E6053A8C1AACQ36134430-1C40732E-E757-46E0-8AE5-E5195EE9AF3DQ36177594-B00704DC-4565-45CC-AA1C-B1B290072D64Q36661144-8BEAD4EB-A3E6-41D6-96A3-85D170456D43Q36665401-800E51A1-F4B8-4D7A-A83A-981D8ED95D6CQ36690595-F2D11888-24AA-4CFF-949B-D1DC7FC932ABQ36717209-CA95ABC7-51D0-44AA-AA01-DFA441890B1BQ36804740-41DCCB7D-7A13-4503-8F06-9B71C6714A95Q37377727-B00796A6-243A-43B6-A1F7-DE8518B22178Q38316471-788C1EC2-82A7-4616-B252-E79F94F8D67DQ39575499-245154E4-5D80-4486-A796-2B6ACA293F46Q39625508-73A36E65-E7E0-4D36-BDE8-4D80046A0985Q39707888-53C5474C-3626-4F63-B8B0-0E2CE75B6C52Q39779184-BB91BC2E-EA81-47F4-85D3-4ADFB36E300DQ40023085-BC06D6F8-A8FF-45B0-AE9D-52C8D8CC6B0AQ40285668-72C0B983-0FA5-4361-AEBD-A66BA4568686Q40872239-5A372430-3B9F-4637-8BE0-107B851FCDB3Q40873605-85E230AC-6FEE-44FE-8070-56ED8140A066Q42078862-B06E2FDA-A3D7-4271-AA84-5B80D73E9FA8
P2860
Response to adversity: molecular control of gene activation following genotoxic stress.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on September 1991
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Response to adversity: molecular control of gene activation following genotoxic stress.
@en
Response to adversity: molecular control of gene activation following genotoxic stress.
@nl
type
label
Response to adversity: molecular control of gene activation following genotoxic stress.
@en
Response to adversity: molecular control of gene activation following genotoxic stress.
@nl
prefLabel
Response to adversity: molecular control of gene activation following genotoxic stress.
@en
Response to adversity: molecular control of gene activation following genotoxic stress.
@nl
P1433
P1476
Response to adversity: molecular control of gene activation following genotoxic stress.
@en
P2093
Fornace AJ Jr
Holbrook NJ
P304
P577
1991-09-01T00:00:00Z