Toll-like receptors 2 and 4 contribute to sepsis-induced depletion of spleen dendritic cells.
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Balancing Innate Immunity and Inflammatory State via Modulation of Neutrophil Function: A Novel Strategy to Fight SepsisThe significance and regulatory mechanisms of innate immune cells in the development of sepsisMurine Cytomegalovirus Disrupts Splenic Dendritic Cell Subsets via Type I Interferon-Dependent and -Independent Mechanisms.A historical perspective on sepsis.Critical role of cRel subunit of NF-κB in sepsis survival.Alterations of dendritic cells in sepsis: featured role in immunoparalysis.Blockade of prostaglandin E2 signaling through EP1 and EP3 receptors attenuates Flt3L-dependent dendritic cell development from hematopoietic progenitor cells.Suppression of Dendritic Cell-Derived IL-12 by Endogenous Glucocorticoids Is Protective in LPS-Induced Sepsis.Bench-to-bedside review: Immunoglobulin therapy for sepsis - biological plausibility from a critical care perspectiveToll-like Receptor 4 Signaling on Dendritic Cells Suppresses Polymorphonuclear Leukocyte CXCR2 Expression and Trafficking via Interleukin 10 During Intra-abdominal Sepsis.Perturbed mononuclear phagocyte system in severely burned and septic patients.The Protective Effect of Alpha 7 Nicotinic Acetylcholine Receptor Activation on Critical Illness and Its MechanismSepsis-induced expansion of granulocytic myeloid-derived suppressor cells promotes tumour growth through Toll-like receptor 4.Role of cellular events in the pathophysiology of sepsis.Innate immune system favors emergency monopoiesis at the expense of DC-differentiation to control systemic bacterial infection in mice.Cathepsin S Is Involved in Th17 Differentiation Through the Upregulation of IL-6 by Activating PAR-2 after Systemic Exposure to Lipopolysaccharide from Porphyromonas gingivalis.Dendritic Cells in Sepsis: Pathological Alterations and Therapeutic Implications.CD86 polymorphism affects pneumonia-induced sepsis by decreasing gene expression in monocytes.Type I interferons drive inflammasome-independent emergency monocytopoiesis during endotoxemia.Autophagy: A Potential Therapeutic Target for Reversing Sepsis-Induced Immunosuppression.Understanding the Cellular Origin of the Mononuclear Phagocyte System Sheds Light on the Myeloid Postulate of Immune Paralysis in Sepsis.
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Toll-like receptors 2 and 4 contribute to sepsis-induced depletion of spleen dendritic cells.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 05 October 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Toll-like receptors 2 and 4 co ...... ion of spleen dendritic cells.
@en
Toll-like receptors 2 and 4 co ...... ion of spleen dendritic cells.
@nl
type
label
Toll-like receptors 2 and 4 co ...... ion of spleen dendritic cells.
@en
Toll-like receptors 2 and 4 co ...... ion of spleen dendritic cells.
@nl
prefLabel
Toll-like receptors 2 and 4 co ...... ion of spleen dendritic cells.
@en
Toll-like receptors 2 and 4 co ...... ion of spleen dendritic cells.
@nl
P2093
P2860
P50
P356
P1476
Toll-like receptors 2 and 4 co ...... ion of spleen dendritic cells.
@en
P2093
Benjamin Zuber
Bertrand Sauneuf
Christophe Rousseau
Emilie Courtine
Fatah Ouaaz
Frédéric Pène
Gonzalo Sirgo
Michel Chignard
Viviane Balloy
P2860
P304
P356
10.1128/IAI.00238-09
P407
P577
2009-10-05T00:00:00Z