about
CD1d- and MR1-Restricted T Cells in SepsisRodent Models of Invasive Aspergillosis due to Aspergillus fumigatus: Still a Long Path toward Standardization.Evaluation of Toll-like, chemokine, and integrin receptors on monocytes and neutrophils from peripheral blood of septic patients and their correlation with clinical outcomes.Sepsis-induced immunosuppression: from cellular dysfunctions to immunotherapy.Sepsis induced by cecal ligation and puncture.The post sepsis-induced expansion and enhanced function of regulatory T cells create an environment to potentiate tumor growth.Dysregulated cytokine expression by CD4+ T cells from post-septic mice modulates both Th1 and Th2-mediated granulomatous lung inflammationHuman monocytes differentiate into dendritic cells subsets that induce anergic and regulatory T cells in sepsis.Impaired CD4+ T-cell proliferation and effector function correlates with repressive histone methylation events in a mouse model of severe sepsis.Cecal ligation and puncture.Epigenetic regulation of immune cell functions during post-septic immunosuppression.Severe sepsis exacerbates cell-mediated immunity in the lung due to an altered dendritic cell cytokine profile.Murine IL-17+ Vγ4 T lymphocytes accumulate in the lungs and play a protective role during severe sepsisCCR4 Controls the Suppressive Effects of Regulatory T Cells on Early and Late Events during Severe Sepsis.Polymicrobial Sepsis Increases Susceptibility to Chronic Viral Infection and Exacerbates CD8+ T Cell Exhaustion.Mechanical Ventilation Induces an Inflammatory Response in Preinjured Lungs in Late Phase of Sepsis.Reversal of long-term sepsis-induced immunosuppression by dendritic cells.Activation of Hepatic STAT3 Maintains Pulmonary Defense during EndotoxemiaSTAT3-mediated IL-17 production by postseptic T cells exacerbates viral immunopathology of the lung.ROS-induced ATF3 causes susceptibility to secondary infections during sepsis-associated immunosuppression.Sepsis-induced long-term immune paralysis--results of a descriptive, explorative study.Late mortality after sepsis: propensity matched cohort studyMolecular and cellular aspects of sepsis-induced immunosuppression.Gammadelta T cells mitigate the organ injury and mortality of sepsis.Toll-like receptors 2 and 4 contribute to sepsis-induced depletion of spleen dendritic cells.Chronic Toxoplasma gondii Infection Exacerbates Secondary Polymicrobial Sepsis.ICU-acquired immunosuppression and the risk for secondary fungal infections.Aspergillosis in the 'nonimmunocompromised' host.Genetic predisposition to respiratory infection and sepsis.Insights and limits of translational research in critical care medicine.ADAMTS-13 regulates neutrophil recruitment in a mouse model of invasive pulmonary aspergillosis.IL-33 contributes to sepsis-induced long-term immunosuppression by expanding the regulatory T cell population.CD4-expressing cells are early mediators of the innate immune system during sepsis.[Invasive pulmonary aspergillosis. Occurrence in a non-neutropenic female patient with abdominal sepsis].Sepsis leads to a reduced antigen-specific primary antibody response.
P2860
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P2860
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh
2003年學術文章
@zh-hant
name
Septic mice are susceptible to pulmonary aspergillosis.
@ast
Septic mice are susceptible to pulmonary aspergillosis.
@en
type
label
Septic mice are susceptible to pulmonary aspergillosis.
@ast
Septic mice are susceptible to pulmonary aspergillosis.
@en
prefLabel
Septic mice are susceptible to pulmonary aspergillosis.
@ast
Septic mice are susceptible to pulmonary aspergillosis.
@en
P2093
P2860
P1476
Septic mice are susceptible to pulmonary aspergillosis.
@en
P2093
Claudia F Benjamim
Cory M Hogaboam
Nicholas W Lukacs
Steven L Kunkel
P2860
P304
P356
10.1016/S0002-9440(10)63615-2
P407
P577
2003-12-01T00:00:00Z