Involvement of antibody-dependent cell-mediated cytotoxicity in inflammatory demyelination in a mouse model of neuromyelitis optica.
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Update on biomarkers in neuromyelitis opticaTreatment of neuromyelitis optica: state-of-the-art and emerging therapiesSeronegative Neuromyelitis Optica Spectrum--the challenges on disease definition and pathogenesis.Neuronal Surface Autoantibodies in Neuropsychiatric Disorders: Are There Implications for Depression?Longitudinally extensive NMO spinal cord pathology produced by passive transfer of NMO-IgG in mice lacking complement inhibitor CD59.The treatment of neuromyelitis optica.Early loss of oligodendrocytes in human and experimental neuromyelitis optica lesions.Neuromyelitis optica pathology in rats following intraperitoneal injection of NMO-IgG and intracerebral needle injury.Human immunoglobulin G reduces the pathogenicity of aquaporin-4 autoantibodies in neuromyelitis opticaCD19 as a molecular target in CNS autoimmunity.Inhibitor(s) of the classical complement pathway in mouse serum limit the utility of mice as experimental models of neuromyelitis optica.Pathogenic aquaporin-4 reactive T cells are sufficient to induce mouse model of neuromyelitis optica.Experimental Neuromyelitis Optica Induces a Type I Interferon Signature in the Spinal Cord.Highly encephalitogenic aquaporin 4-specific T cells and NMO-IgG jointly orchestrate lesion location and tissue damage in the CNS.Experimental mouse model of optic neuritis with inflammatory demyelination produced by passive transfer of neuromyelitis optica-immunoglobulin G.Unique neuromyelitis optica pathology produced in naïve rats by intracerebral administration of NMO-IgGMarked central nervous system pathology in CD59 knockout rats following passive transfer of Neuromyelitis optica immunoglobulin G.Deletional tolerance prevents AQP4-directed autoimmunity in mice.T cell-activation in neuromyelitis optica lesions plays a role in their formation.Müller cells and retinal axons can be primary targets in experimental neuromyelitis optica spectrum disorder.Efficacy of Polyvalent Human Immunoglobulins in an Animal Model of Neuromyelitis Optica Evoked by Intrathecal Anti-Aquaporin 4 Antibodies.Neuromyelitis Optica: Deciphering a Complex Immune-Mediated Astrocytopathy.Bystander mechanism for complement-initiated early oligodendrocyte injury in neuromyelitis optica.Immunopathogenesis in Myasthenia Gravis and Neuromyelitis Optica.Determining the Spatial Relationship of Membrane-Bound Aquaporin-4 Autoantibodies by STED Nanoscopy.Mechanisms for lesion localization in neuromyelitis optica spectrum disorders.Combination Treatment of C16 Peptide and Angiopoietin-1 Alleviates Neuromyelitis Optica in an Experimental Model.Aquaporin-4 Autoantibodies From Neuromyelitis Optica Spectrum Disorder Patients Induce Complement-Independent Immunopathologies in Mice.
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Involvement of antibody-dependent cell-mediated cytotoxicity in inflammatory demyelination in a mouse model of neuromyelitis optica.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 31 August 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Involvement of antibody-depend ...... model of neuromyelitis optica.
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Involvement of antibody-depend ...... model of neuromyelitis optica.
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type
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Involvement of antibody-depend ...... model of neuromyelitis optica.
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Involvement of antibody-depend ...... model of neuromyelitis optica.
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Involvement of antibody-depend ...... model of neuromyelitis optica.
@en
Involvement of antibody-depend ...... model of neuromyelitis optica.
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P2860
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Involvement of antibody-depend ...... model of neuromyelitis optica
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A S Verkman
Alanna M Ritchie
Jeffrey L Bennett
Nithi Asavapanumas
Scott Wemlinger
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P2888
P304
P356
10.1007/S00401-013-1172-Z
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P577
2013-08-31T00:00:00Z